Accessory renal arteries - a source of hypertension: A case report.
Accessory artery
Case report
Renal artery stenosis
Renovascular disease
Risk factor
Secondary hypertension
Young patients
Journal
World journal of clinical cases
ISSN: 2307-8960
Titre abrégé: World J Clin Cases
Pays: United States
ID NLM: 101618806
Informations de publication
Date de publication:
06 Mar 2023
06 Mar 2023
Historique:
received:
13
09
2022
revised:
11
11
2022
accepted:
07
02
2023
entrez:
17
3
2023
pubmed:
18
3
2023
medline:
18
3
2023
Statut:
ppublish
Résumé
Secondary hypertension is a relatively rare condition most commonly caused by renovascular disease due to atherosclerotic vascular disease or fibromuscular dysplasia. Although accessory renal arteries are frequent, to date, only six cases of secondary hypertension determined by their existence have been reported. We describe a case of a 39-year-old female who came to the emergency department with an urgent hypertensive crisis and hypertensive encephalopathy. Despite normal renal arteries, the computed tomography angiography revealed an inferior polar artery with 50% stenosis of its diameter. Conservative treatment with amlodipine, indapamide and perindopril was adopted, leading to blood pressure control within one month. To the best of our knowledge, there are controversies regarding accessory renal arteries as a potential etiology for secondary hypertension, but the seven similar cases already described, along with the current case, could reinforce the necessity of more studies concerning this subject.
Sections du résumé
BACKGROUND
BACKGROUND
Secondary hypertension is a relatively rare condition most commonly caused by renovascular disease due to atherosclerotic vascular disease or fibromuscular dysplasia. Although accessory renal arteries are frequent, to date, only six cases of secondary hypertension determined by their existence have been reported.
CASE SUMMARY
METHODS
We describe a case of a 39-year-old female who came to the emergency department with an urgent hypertensive crisis and hypertensive encephalopathy. Despite normal renal arteries, the computed tomography angiography revealed an inferior polar artery with 50% stenosis of its diameter. Conservative treatment with amlodipine, indapamide and perindopril was adopted, leading to blood pressure control within one month.
CONCLUSION
CONCLUSIONS
To the best of our knowledge, there are controversies regarding accessory renal arteries as a potential etiology for secondary hypertension, but the seven similar cases already described, along with the current case, could reinforce the necessity of more studies concerning this subject.
Identifiants
pubmed: 36926389
doi: 10.12998/wjcc.v11.i7.1506
pmc: PMC10011986
doi:
Types de publication
Case Reports
Langues
eng
Pagination
1506-1512Informations de copyright
©The Author(s) 2023. Published by Baishideng Publishing Group Inc. All rights reserved.
Déclaration de conflit d'intérêts
Conflict-of-interest statement: The authors declare that they have no conflict of interest to disclose.
Références
Hypertension. 2005 Aug;46(2):380-5
pubmed: 15967872
Am J Hypertens. 2010 Nov;23(11):1159-69
pubmed: 20864945
Case Rep Nephrol. 2020 Jul 22;2020:8879165
pubmed: 32774955
AJR Am J Roentgenol. 2004 Jun;182(6):1521-4
pubmed: 15150000
Clin Hypertens. 2018 Nov 01;24:15
pubmed: 30410790
Lancet. 1965 Feb 13;1(7381):341-4
pubmed: 14246702
J Hypertens. 2018 Oct;36(10):1953-2041
pubmed: 30234752
Int J Hypertens. 2020 Apr 08;2020:7109502
pubmed: 32328302
Hypertension. 2021 Sep;78(4):898-911
pubmed: 34455817
Br J Surg. 1963 Jan;50:368-74
pubmed: 13979763
Am Fam Physician. 2010 Dec 15;82(12):1471-8
pubmed: 21166367
J Hypertens. 2000 Oct;18(10):1437-44
pubmed: 11057431
J Vasc Surg Cases Innov Tech. 2020 Jun 26;6(3):425-429
pubmed: 33367190
Hypertens Res. 2020 Oct;43(10):1015-1027
pubmed: 32572170
Evid Based Complement Alternat Med. 2021 Dec 29;2021:9957361
pubmed: 35003315
Saudi J Kidney Dis Transpl. 2017 Mar-Apr;28(2):292-297
pubmed: 28352010
J Vasc Interv Radiol. 2006 Oct;17(10):1713-4
pubmed: 17057017
N Engl J Med. 2001 Feb 8;344(6):431-42
pubmed: 11172181