Analysis of the fecal metagenome in long-term survivors of pancreas cancer.

Akkermansia muciniphila Faecalibacterium prausnitzii Malassezia long-term survivor metagenomics microbiome pancreatic cancer

Journal

Cancer
ISSN: 1097-0142
Titre abrégé: Cancer
Pays: United States
ID NLM: 0374236

Informations de publication

Date de publication:
01 07 2023
Historique:
revised: 06 02 2023
received: 15 11 2022
accepted: 07 02 2023
medline: 12 6 2023
pubmed: 22 3 2023
entrez: 21 3 2023
Statut: ppublish

Résumé

The 5-year overall survival of pancreas adenocarcinoma (PCa) remains less than 10%. Clinical and tumor genomic characteristics have not differentiated PCa long-term survivors (LTSs) from unselected patients. Preclinical studies using fecal transplant experiments from LTSs of PCa have revealed delayed tumor growth through unknown mechanisms involving the fecal microbiota. However, features of the fecal microbiome in patients with long-term survival are not well described. In this cross-sectional study, comprehensive shotgun metagenomics was performed on stool from PCa patients with long-term survival (n = 16). LTS was defined as >4 years from pancreatectomy and all therapy without recurrence. LTSs were compared to control patients with PCa who completed pancreatectomy and chemotherapy (n = 8). Stool was sequenced using an Illumina NextSeq500. Statistical analyses were performed in R with MicrobiomeSeq and Phyloseq for comparison of LTSs and controls. All patients underwent pancreatectomy and chemotherapy before sample donation. The median time from pancreatectomy of 6 years (4-14 years) for LTSs without evidence of disease compared to a median disease-free survival of 1.8 years from pancreatectomy in the control group. No differences were observed in overall microbial diversity for LTSs and controls using Shannon/Simpson indexes. Significant enrichment of species relative abundance was observed in LTSs for the Ruminococacceae family specifically Faecalibacterium prausnitzii species as well as Akkermansia muciniphila species. Stool from patients cured from PCa has more relative abundance of Faecalibacterium prausnitzii and Akkermansia muciniphila. Additional studies are needed to explore potential mechanisms by which the fecal microbiota may influence survival in PCa. Although pancreatic cancer treatments have improved, the number of long-term survivors has remained stagnant with a 5-year overall survival estimate of 9%. Emerging evidence suggests that microbes within the gastrointestinal tract can influence cancer response through activation of the immune system. In this study, we profiled the stool microbiome in long-term survivors of pancreas cancer and controls. Several enriched species previously associated with enhanced tumor immune response were observed including Faecalibacterium prausnitzii and Akkermansia muciniphila. These findings warrant additional study assessing mechanisms by which the fecal microbiota may enhance pancreatic cancer immune response.

Sections du résumé

BACKGROUND
The 5-year overall survival of pancreas adenocarcinoma (PCa) remains less than 10%. Clinical and tumor genomic characteristics have not differentiated PCa long-term survivors (LTSs) from unselected patients. Preclinical studies using fecal transplant experiments from LTSs of PCa have revealed delayed tumor growth through unknown mechanisms involving the fecal microbiota. However, features of the fecal microbiome in patients with long-term survival are not well described.
METHODS
In this cross-sectional study, comprehensive shotgun metagenomics was performed on stool from PCa patients with long-term survival (n = 16). LTS was defined as >4 years from pancreatectomy and all therapy without recurrence. LTSs were compared to control patients with PCa who completed pancreatectomy and chemotherapy (n = 8). Stool was sequenced using an Illumina NextSeq500. Statistical analyses were performed in R with MicrobiomeSeq and Phyloseq for comparison of LTSs and controls.
RESULTS
All patients underwent pancreatectomy and chemotherapy before sample donation. The median time from pancreatectomy of 6 years (4-14 years) for LTSs without evidence of disease compared to a median disease-free survival of 1.8 years from pancreatectomy in the control group. No differences were observed in overall microbial diversity for LTSs and controls using Shannon/Simpson indexes. Significant enrichment of species relative abundance was observed in LTSs for the Ruminococacceae family specifically Faecalibacterium prausnitzii species as well as Akkermansia muciniphila species.
CONCLUSIONS
Stool from patients cured from PCa has more relative abundance of Faecalibacterium prausnitzii and Akkermansia muciniphila. Additional studies are needed to explore potential mechanisms by which the fecal microbiota may influence survival in PCa.
PLAIN LANGUAGE SUMMARY
Although pancreatic cancer treatments have improved, the number of long-term survivors has remained stagnant with a 5-year overall survival estimate of 9%. Emerging evidence suggests that microbes within the gastrointestinal tract can influence cancer response through activation of the immune system. In this study, we profiled the stool microbiome in long-term survivors of pancreas cancer and controls. Several enriched species previously associated with enhanced tumor immune response were observed including Faecalibacterium prausnitzii and Akkermansia muciniphila. These findings warrant additional study assessing mechanisms by which the fecal microbiota may enhance pancreatic cancer immune response.

Identifiants

pubmed: 36943918
doi: 10.1002/cncr.34748
doi:

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1986-1994

Subventions

Organisme : NCI NIH HHS
ID : K08 CA237735
Pays : United States

Informations de copyright

© 2023 The Authors. Cancer published by Wiley Periodicals LLC on behalf of American Cancer Society.

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Auteurs

Jordan Kharofa (J)

Department of Radiation Oncology, University of Cincinnati College of Medicine, Cincinnati, Ohio, USA.

David Haslam (D)

Microbial Metagenomics Analysis Center at Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, USA.

Rachael Wilkinson (R)

Department of Infectious Disease, University of Cincinnati College of Medicine, Cincinnati, Ohio, USA.

Allison Weiss (A)

Department of Microbiology, University of Cincinnati College of Medicine, Cincinnati, Ohio, USA.

Sameer Patel (S)

Division of Surgical Oncology, University of Cincinnati College of Medicine, Cincinnati, Ohio, USA.

Kyle Wang (K)

Department of Radiation Oncology, University of Cincinnati College of Medicine, Cincinnati, Ohio, USA.

Hope Esslinger (H)

Department of Radiation Oncology, University of Cincinnati College of Medicine, Cincinnati, Ohio, USA.

Olugbenga Olowokure (O)

Department of Hematology/Oncology Division, University of Cincinnati College of Medicine, Cincinnati, Ohio, USA.

Davendra Sohal (D)

Department of Hematology/Oncology Division, University of Cincinnati College of Medicine, Cincinnati, Ohio, USA.

Greg Wilson (G)

Division of Surgical Oncology, University of Cincinnati College of Medicine, Cincinnati, Ohio, USA.

Syed Ahmad (S)

Division of Surgical Oncology, University of Cincinnati College of Medicine, Cincinnati, Ohio, USA.

Senu Apewokin (S)

Department of Infectious Disease, University of Cincinnati College of Medicine, Cincinnati, Ohio, USA.

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