Relationship between HuR and tumor drug resistance.


Journal

Clinical & translational oncology : official publication of the Federation of Spanish Oncology Societies and of the National Cancer Institute of Mexico
ISSN: 1699-3055
Titre abrégé: Clin Transl Oncol
Pays: Italy
ID NLM: 101247119

Informations de publication

Date de publication:
Jul 2023
Historique:
received: 21 08 2022
accepted: 31 01 2023
medline: 12 6 2023
pubmed: 23 3 2023
entrez: 22 3 2023
Statut: ppublish

Résumé

Human resistance protein R (HuR), also known as embryonic lethal abnormal visual-like protein (ELAVL1), is an RNA-binding protein widely expressed in vivo that affects the mRNA stability of targeted and is involved in post-transcriptional regulation. Recent studies have shown that HuR is aberrantly expressed in different human cancers and is an essential factor in poor clinical prognosis. The role of HuR in numerous tumors suggests that it could be a new target for tumor therapy and as a marker for efficacy and prognostic assessment. This review focuses on the relationship between HuR and drug resistance in different tumors and briefly describes the structure, function, and inhibitors of HuR. We summarize the mechanisms by which HuR causes tumor resistance and the molecular targets affected.

Identifiants

pubmed: 36947360
doi: 10.1007/s12094-023-03109-5
pii: 10.1007/s12094-023-03109-5
doi:

Substances chimiques

ELAV-Like Protein 1 0

Types de publication

Journal Article Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

1999-2014

Subventions

Organisme : National Natural Science Foundation of China-Zhejiang Joint Fund for the Integration of Industrialization and Informatization
ID : 81672474
Organisme : National Natural Science Foundation of China-Zhejiang Joint Fund for the Integration of Industrialization and Informatization
ID : 81874049

Informations de copyright

© 2023. The Author(s), under exclusive licence to Federación de Sociedades Españolas de Oncología (FESEO).

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Auteurs

Qiancheng Ma (Q)

College of Biotechnology and Bioengineering, Zhejiang University of Technology, Hangzhou, 310014, China.

Qiliang Lu (Q)

Qingdao Medical College, Qingdao University, Qingdao, 266000, China.

Xiangxiang Lei (X)

Hangzhou Medical College, Hangzhou, 310053, China.

Jie Zhao (J)

College of Biotechnology and Bioengineering, Zhejiang University of Technology, Hangzhou, 310014, China.

Wen Sun (W)

Zhejiang Chinese Medical University, Hangzhou, 310053, China.

Dongsheng Huang (D)

The Key Laboratory of Tumor Molecular Diagnosis, and Individualized Medicine of Zhejiang Province, Zhejiang Provincial People's Hospital, Affiliated People's Hospital, Hangzhou Medical College, Hangzhou, 310014, China. dshuang@hmc.edu.cn.

Qing Zhu (Q)

College of Biotechnology and Bioengineering, Zhejiang University of Technology, Hangzhou, 310014, China.

Qiuran Xu (Q)

The Key Laboratory of Tumor Molecular Diagnosis, and Individualized Medicine of Zhejiang Province, Zhejiang Provincial People's Hospital, Affiliated People's Hospital, Hangzhou Medical College, Hangzhou, 310014, China. windway626@sina.com.

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