Nrf2 Activation Does Not Protect from Aldosterone-Induced Kidney Damage in Mice.

Keap1 knockdown aldosterone hypertension kidney injury oxidative stress

Journal

Antioxidants (Basel, Switzerland)
ISSN: 2076-3921
Titre abrégé: Antioxidants (Basel)
Pays: Switzerland
ID NLM: 101668981

Informations de publication

Date de publication:
22 Mar 2023
Historique:
received: 03 01 2023
revised: 10 02 2023
accepted: 21 03 2023
medline: 30 3 2023
entrez: 29 3 2023
pubmed: 30 3 2023
Statut: epublish

Résumé

Nuclear factor erythroid 2-related factor 2 (Nrf2) is downregulated in chronic kidney disease (CKD). Activation of Nrf2 might be a therapeutic option in CKD. Here we investigate the effect of Nrf2 activation on aldosterone (Aldo)-induced renal injury. Wild-type (WT) mice, transgenic Keap1 hypomorphic (Nrf2ꜛ, genotype results in upregulation of Nrf2 expression) mice and WT mice treated with the Nrf2 activator sulforaphane (Sulf) received Aldo for 4 weeks. In Aldo-treated mice, kidneys were significantly heavier and pathologically altered, reflected by increased urinary albumin levels and tissue damage. In Nrf2ꜛ-Aldo mice the tubule damage marker NGAL was significantly decreased. Increased oxidative damage markers (8-OHdG, 15-isoprostane F

Identifiants

pubmed: 36979025
pii: antiox12030777
doi: 10.3390/antiox12030777
pmc: PMC10044832
pii:
doi:

Types de publication

Journal Article

Langues

eng

Subventions

Organisme : Deutsche Forschungsgemeinschaft
ID : Schu 2367/5-4

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Auteurs

Ronja Brinks (R)

Institute of Toxicology, Medical Faculty, University of Düsseldorf, 40225 Düsseldorf, Germany.

Christoph Jan Wruck (CJ)

Department of Anatomy and Cell Biology, Uniklinik RWTH Aachen, 52074 Aachen, Germany.

Jutta Schmitz (J)

Institute of Toxicology, Medical Faculty, University of Düsseldorf, 40225 Düsseldorf, Germany.

Nicole Schupp (N)

Institute of Toxicology, Medical Faculty, University of Düsseldorf, 40225 Düsseldorf, Germany.

Classifications MeSH