Clinical and Hemodynamic Responses to Imatinib in Pulmonary Veno-Occlusive Disease/Pulmonary Capillary Hemangiomatosis: A Retrospective Pilot Study of Five Cases and Review of the Literature.


Journal

American journal of cardiovascular drugs : drugs, devices, and other interventions
ISSN: 1179-187X
Titre abrégé: Am J Cardiovasc Drugs
Pays: New Zealand
ID NLM: 100967755

Informations de publication

Date de publication:
May 2023
Historique:
accepted: 27 02 2023
medline: 8 5 2023
pubmed: 31 3 2023
entrez: 30 3 2023
Statut: ppublish

Résumé

Pulmonary veno-occlusive disease (PVOD) and pulmonary capillary hemangiomatosis (PCH) are rare types of pulmonary arterial hypertension with dismal prognoses; there is no established medical treatment for these conditions. Possible efficacy of imatinib against these conditions has been reported in 15 cases; however, how and in whom imatinib is effective remain unknown. We retrospectively evaluated clinical data from consecutive patients with PVOD/PCH treated with imatinib at our institution. The diagnosis of PVOD/PCH was established using the following criteria: pre-capillary pulmonary hypertension; diffusion capacity of the lung for carbon monoxide < 60%; and two or more high-resolution computed tomography findings of interlobular septal thickening, centrilobular opacities, and mediastinal lymphadenopathy. The dose of pulmonary vasodilators remained unchanged during the assessment of imatinib. The medical records of five patients with PVOD/PCH were reviewed. The patients were aged 67 ± 13 years, their diffusion capacity of the lung for carbon monoxide was 29 ± 8%, and their mean pulmonary artery pressure was 40 ± 7 mmHg. Imatinib was administered at 50-100 mg/day; consequently, the World Health Organization functional class improved in one patient. In addition, imatinib improved the arterial oxygen partial pressure in this and another patient (these two also experienced a decreased mean pulmonary artery pressure and pulmonary vascular resistance after imatinib usage). This study indicated that imatinib improves the clinical condition, including pulmonary hemodynamics, of some patients with PVOD/PCH. In addition, patients with a certain high-resolution computed tomography pattern or PCH-dominant vasculopathy may respond favorably to imatinib.

Sections du résumé

BACKGROUND BACKGROUND
Pulmonary veno-occlusive disease (PVOD) and pulmonary capillary hemangiomatosis (PCH) are rare types of pulmonary arterial hypertension with dismal prognoses; there is no established medical treatment for these conditions. Possible efficacy of imatinib against these conditions has been reported in 15 cases; however, how and in whom imatinib is effective remain unknown.
METHODS METHODS
We retrospectively evaluated clinical data from consecutive patients with PVOD/PCH treated with imatinib at our institution. The diagnosis of PVOD/PCH was established using the following criteria: pre-capillary pulmonary hypertension; diffusion capacity of the lung for carbon monoxide < 60%; and two or more high-resolution computed tomography findings of interlobular septal thickening, centrilobular opacities, and mediastinal lymphadenopathy. The dose of pulmonary vasodilators remained unchanged during the assessment of imatinib.
RESULTS RESULTS
The medical records of five patients with PVOD/PCH were reviewed. The patients were aged 67 ± 13 years, their diffusion capacity of the lung for carbon monoxide was 29 ± 8%, and their mean pulmonary artery pressure was 40 ± 7 mmHg. Imatinib was administered at 50-100 mg/day; consequently, the World Health Organization functional class improved in one patient. In addition, imatinib improved the arterial oxygen partial pressure in this and another patient (these two also experienced a decreased mean pulmonary artery pressure and pulmonary vascular resistance after imatinib usage).
CONCLUSIONS CONCLUSIONS
This study indicated that imatinib improves the clinical condition, including pulmonary hemodynamics, of some patients with PVOD/PCH. In addition, patients with a certain high-resolution computed tomography pattern or PCH-dominant vasculopathy may respond favorably to imatinib.

Identifiants

pubmed: 36995544
doi: 10.1007/s40256-023-00577-6
pii: 10.1007/s40256-023-00577-6
doi:

Substances chimiques

Imatinib Mesylate 8A1O1M485B
Carbon Monoxide 7U1EE4V452

Types de publication

Review Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

329-338

Informations de copyright

© 2023. The Author(s), under exclusive licence to Springer Nature Switzerland AG.

Références

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Auteurs

Junichi Nakamura (J)

Department of Respiratory Medicine, Faculty of Medicine, Hokkaido University, Sapporo, Japan.

Ichizo Tsujino (I)

Department of Respiratory Medicine, Faculty of Medicine, Hokkaido University, Sapporo, Japan. itsujino@med.hokudai.ac.jp.
Division of Respiratory and Cardiovascular Innovative Research, Faculty of Medicine, Hokkaido University, N15, W7, Kita-ku, Sapporo, Hokkaido, 060-8638, Japan. itsujino@med.hokudai.ac.jp.

Hideki Shima (H)

Department of Respiratory Medicine, Faculty of Medicine, Hokkaido University, Sapporo, Japan.

Toshitaka Nakaya (T)

Department of Respiratory Medicine, Faculty of Medicine, Hokkaido University, Sapporo, Japan.

Ayako Sugimoto (A)

Department of Respiratory Medicine, Faculty of Medicine, Hokkaido University, Sapporo, Japan.

Takahiro Sato (T)

Department of Respiratory Medicine, Faculty of Medicine, Hokkaido University, Sapporo, Japan.
Division of Respiratory and Cardiovascular Innovative Research, Faculty of Medicine, Hokkaido University, N15, W7, Kita-ku, Sapporo, Hokkaido, 060-8638, Japan.

Taku Watanabe (T)

Department of Respiratory Medicine, Faculty of Medicine, Hokkaido University, Sapporo, Japan.

Hiroshi Ohira (H)

Department of Respiratory Medicine, Faculty of Medicine, Hokkaido University, Sapporo, Japan.

Masaru Suzuki (M)

Department of Respiratory Medicine, Faculty of Medicine, Hokkaido University, Sapporo, Japan.

Satonori Tsuneta (S)

Department of Diagnostic and Interventional Radiology, Hokkaido University Hospital, Sapporo, Japan.

Ryo Hisada (R)

Department of Rheumatology, Endocrinology and Nephrology, Faculty of Medicine and Graduate School of Medicine, Hokkaido University, Sapporo, Japan.

Masaru Kato (M)

Department of Rheumatology, Endocrinology and Nephrology, Faculty of Medicine and Graduate School of Medicine, Hokkaido University, Sapporo, Japan.

Satoshi Konno (S)

Department of Respiratory Medicine, Faculty of Medicine, Hokkaido University, Sapporo, Japan.
Division of Respiratory and Cardiovascular Innovative Research, Faculty of Medicine, Hokkaido University, N15, W7, Kita-ku, Sapporo, Hokkaido, 060-8638, Japan.

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Classifications MeSH