Primary Effusion Lymphoma in an HIV-Negative Patient with Chronic Myeloid Leukemia Treated with Dasatinib.


Journal

Pathobiology : journal of immunopathology, molecular and cellular biology
ISSN: 1423-0291
Titre abrégé: Pathobiology
Pays: Switzerland
ID NLM: 9007504

Informations de publication

Date de publication:
2023
Historique:
received: 28 11 2022
accepted: 28 03 2023
medline: 2 11 2023
pubmed: 31 3 2023
entrez: 30 3 2023
Statut: ppublish

Résumé

Primary effusion lymphoma (PEL) is a malignant lymphomatous effusion, which by definition is Kaposi sarcoma herpesvirus/human herpesvirus 8-positive. PEL typically occurs in HIV-infected patients but can also occur in HIV-negative individuals, including in organ transplant recipients. Tyrosine kinase inhibitors (TKIs) are currently the standard of care for patients with chronic myeloid leukemia (CML), BCR::ABL1-positive. Although TKIs are extremely effective in treating CML, they alter T-cell function by inhibiting peripheral T-cell migration and altering T-cell trafficking and have been associated with the development of pleural effusions. We report a case of PEL in a young, relatively immunocompetent patient with no history of organ transplant receiving dasatinib for CML, BCR::ABL1-positive. We hypothesize that the loss of T-cell function secondary to TKI therapy (dasatinib) may have resulted in the unchecked cellular proliferation of Kaposi sarcoma herpesvirus (KSHV)-infected cells, leading to the emergence of a PEL. We recommend cytologic investigation and KSHV testing in patients being treated with dasatinib for CML who present with persistent or recurrent effusions.

Identifiants

pubmed: 36996787
pii: 000530429
doi: 10.1159/000530429
pmc: PMC10614567
doi:

Substances chimiques

Dasatinib RBZ1571X5H

Types de publication

Case Reports

Langues

eng

Sous-ensembles de citation

IM

Pagination

356-364

Informations de copyright

© 2023 The Author(s). Published by S. Karger AG, Basel.

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Auteurs

Ivo N SahBandar (IN)

Department of Pathology and Laboratory Medicine, Weill Cornell Medicine, New York, New York, USA, ins4002@nyp.org.

Chandler B Sy (CB)

Department of Pathology and Laboratory Medicine, Weill Cornell Medicine, New York, New York, USA.

Tayler van den Akker (T)

Department of Pathology and Laboratory Medicine, Weill Cornell Medicine, New York, New York, USA.

David Kim (D)

Department of Pathology and Laboratory Medicine, Weill Cornell Medicine, New York, New York, USA.

Julia T Geyer (JT)

Department of Pathology and Laboratory Medicine, Weill Cornell Medicine, New York, New York, USA.

Amy Chadburn (A)

Department of Pathology and Laboratory Medicine, Weill Cornell Medicine, New York, New York, USA.

Ethel Cesarman (E)

Department of Pathology and Laboratory Medicine, Weill Cornell Medicine, New York, New York, USA.

Giorgio Inghirami (G)

Department of Pathology and Laboratory Medicine, Weill Cornell Medicine, New York, New York, USA.

John N Allan (JN)

Division of Hematology and Medical Oncology, Department of Medicine, Weill Cornell Medicine, New York, New York, USA.

Momin T Siddiqui (MT)

Department of Pathology and Laboratory Medicine, Weill Cornell Medicine, New York, New York, USA.

Madhu M Ouseph (MM)

Department of Pathology and Laboratory Medicine, Weill Cornell Medicine, New York, New York, USA.

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