Development of a new HISCL automated CXCL9 immunoassay.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
01 04 2023
Historique:
received: 21 11 2022
accepted: 28 03 2023
medline: 4 4 2023
entrez: 3 4 2023
pubmed: 4 4 2023
Statut: epublish

Résumé

C-X-C motif chemokine ligand 9 (CXCL9), a candidate biomarker, reflects type 1 (T1) inflammation pathology. Here, we report the analytical performance and clinical characteristics of a new CXCL9 reagent for a fully automated immunoassay device. We evaluated the limits of blank, detection, and quantitation (LoQ) along with other efficacy parameters, and the ability of the assay to report patient health, COVID-19 status, and the presence of asthma and/or interstitial lung diseases (ILDs). The coefficient of variation for 5-day total precision using two instruments was 7% across two controls, serum, and plasma panels. LoQ of 2.2 pg/mL suggested the efficacy of the assay in detecting T1 inflammation in plasma or serum; no cross-reactivity or interference was observed. We identified high serum CXCL9 levels in samples from patients with acute COVID-19 infections (n = 57), chronic bird-related hypersensitivity pneumonitis (n = 61), asthma (n = 194), and ILDs (n = 84) compared to healthy individuals (< 39.0 pg/mL). Furthermore, CXCL9 levels increased with age in asthma patients, and an opposite trend was observed for T2 inflammatory factors. These results suggest the utility of the automated CXCL9 immunoassay for measuring CXCL9 in clinical samples and reflect its role in T1 inflammation.

Identifiants

pubmed: 37005469
doi: 10.1038/s41598-023-32513-8
pii: 10.1038/s41598-023-32513-8
pmc: PMC10066986
doi:

Substances chimiques

Biomarkers 0
Chemokine CXCL9 0
Chemokine CXCL10 0
CXCL9 protein, human 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

5342

Informations de copyright

© 2023. The Author(s).

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Auteurs

Takehiro Hasegawa (T)

Research and Development Division, Sysmex R&D Centre Europe GmbH, Falkenried 88, 20251, Hamburg, Germany. Hasegawa.Takehiro@sysmex-rdce.com.

Maho Yoshida (M)

Scientific Affairs, Sysmex Corporation, 1-3-2, Murotani, Nishi-Ku, Kobe, Hyogo, 651-14 2241, Japan.

Shunsuke Watanabe (S)

Central Research Laboratories, Sysmex Corporation, 4-4-4, Takatsuka-Dai, Nish Ward, Kobe, Japan.

Takami Kondo (T)

Scientific Affairs, Sysmex Corporation, 1-3-2, Murotani, Nishi-Ku, Kobe, Hyogo, 651-14 2241, Japan.

Hideo Asada (H)

Department of Dermatology, Nara Medical University School of Medicine, 840 Shijo, Kashihara, Nara, 634-8522, Japan.

Atsushi Nakagawa (A)

Kobe City Medical Centre General Hospital, 2-1-1, Minamimachi, Minatojima, Chuo Ward, Kobe, Japan.

Keisuke Tomii (K)

Kobe City Medical Centre General Hospital, 2-1-1, Minamimachi, Minatojima, Chuo Ward, Kobe, Japan.

Masami Kameda (M)

Department of Respiratory Medicine and Allergology, Sapporo Medical University School of Medicine, Sapporo, Hokkaido, Japan.

Mitsuo Otsuka (M)

Department of Respiratory Medicine and Allergology, Sapporo Medical University School of Medicine, Sapporo, Hokkaido, Japan.

Koji Kuronuma (K)

Department of Respiratory Medicine and Allergology, Sapporo Medical University School of Medicine, Sapporo, Hokkaido, Japan.

Hirofumi Chiba (H)

Department of Respiratory Medicine and Allergology, Sapporo Medical University School of Medicine, Sapporo, Hokkaido, Japan.

Shinji Katayanagi (S)

Department of Respiratory Medicine, Tokyo Medical and Dental University, 1-5-45 10 Yushima, Bunkyo-Ku, Tokyo, 113-8519, Japan.

Yasunari Miyazaki (Y)

Department of Respiratory Medicine, Tokyo Medical and Dental University, 1-5-45 10 Yushima, Bunkyo-Ku, Tokyo, 113-8519, Japan.

Akio Mori (A)

National Hospital Organization, Sagamihara National Hospital, Clinical Research Centre, Sagamihara, Japan.

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Classifications MeSH