Ketone bodies: A double-edged sword for mammalian life span.


Journal

Aging cell
ISSN: 1474-9726
Titre abrégé: Aging Cell
Pays: England
ID NLM: 101130839

Informations de publication

Date de publication:
06 2023
Historique:
received: 30 01 2023
accepted: 24 03 2023
medline: 15 6 2023
pubmed: 16 4 2023
entrez: 15 4 2023
Statut: ppublish

Résumé

Accumulating evidence suggests health benefits of ketone bodies, and especially for longevity. However, the precise role of endogenous ketogenesis in mammalian life span, and the safety and efficacy of the long-term exogenous supplementation of ketone bodies remain unclear. In the present study, we show that a deficiency in endogenous ketogenesis, induced by whole-body Hmgcs2 deletion, shortens life span in mice, and that this is prevented by daily ketone body supplementation using a diet containing 1,3-butanediol, a precursor of β-hydroxybutyrate. Furthermore, feeding the 1,3-butanediol-containing diet from early in life increases midlife mortality in normal mice, but in aged mice it extends life span and prevents the high mortality associated with atherosclerosis in ApoE-deficient mice. By contrast, an ad libitum low-carbohydrate ketogenic diet markedly increases mortality. In conclusion, endogenous ketogenesis affects mammalian survival, and ketone body supplementation may represent a double-edged sword with respect to survival, depending on the method of administration and health status.

Identifiants

pubmed: 37060184
doi: 10.1111/acel.13833
pmc: PMC10265173
doi:

Substances chimiques

Ketone Bodies 0
1,3-butylene glycol 3XUS85K0RA
Butylene Glycols 0
3-Hydroxybutyric Acid TZP1275679

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e13833

Informations de copyright

© 2023 The Authors. Aging Cell published by Anatomical Society and John Wiley & Sons Ltd.

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Auteurs

Issei Tomita (I)

Department of Medicine, Shiga University of Medical Science, Tsukinowa-cho, Seta, Otsu, Shiga, 520-2192, Japan.

Hiroaki Tsuruta (H)

Department of Medicine, Shiga University of Medical Science, Tsukinowa-cho, Seta, Otsu, Shiga, 520-2192, Japan.

Mako Yasuda-Yamahara (M)

Department of Medicine, Shiga University of Medical Science, Tsukinowa-cho, Seta, Otsu, Shiga, 520-2192, Japan.

Kosuke Yamahara (K)

Department of Medicine, Shiga University of Medical Science, Tsukinowa-cho, Seta, Otsu, Shiga, 520-2192, Japan.

Shogo Kuwagata (S)

Department of Medicine, Shiga University of Medical Science, Tsukinowa-cho, Seta, Otsu, Shiga, 520-2192, Japan.

Yuki Tanaka-Sasaki (Y)

Department of Medicine, Shiga University of Medical Science, Tsukinowa-cho, Seta, Otsu, Shiga, 520-2192, Japan.

Masami Chin-Kanasaki (M)

Department of Medicine, Shiga University of Medical Science, Tsukinowa-cho, Seta, Otsu, Shiga, 520-2192, Japan.

Yukihiro Fujita (Y)

Department of Medicine, Shiga University of Medical Science, Tsukinowa-cho, Seta, Otsu, Shiga, 520-2192, Japan.

Eiichiro Nishi (E)

Department of Pharmacology, Shiga University of Medical Science, Tsukinowa-cho, Seta, Tsukinowa-cho, Seta, Otsu, Shiga, 520-2192, Japan.

Hideki Katagiri (H)

Department of Metabolism and Diabetes, Tohoku University Graduate School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi, 980-8574, Japan.

Hiroshi Maegawa (H)

Department of Medicine, Shiga University of Medical Science, Tsukinowa-cho, Seta, Otsu, Shiga, 520-2192, Japan.

Shinji Kume (S)

Department of Medicine, Shiga University of Medical Science, Tsukinowa-cho, Seta, Otsu, Shiga, 520-2192, Japan.

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Classifications MeSH