First-in-human study of E7130 (a tumor microenvironment-ameliorating microtubule inhibitor) in patients with advanced solid tumors: Primary results of the dose-escalation part.


Journal

Cancer
ISSN: 1097-0142
Titre abrégé: Cancer
Pays: United States
ID NLM: 0374236

Informations de publication

Date de publication:
01 08 2023
Historique:
revised: 23 02 2023
received: 23 08 2022
accepted: 27 02 2023
medline: 6 7 2023
pubmed: 21 4 2023
entrez: 20 04 2023
Statut: ppublish

Résumé

E7130 is a novel anticancer agent created from a total synthetic study of norhalichondrin B. The authors report the E7130 dose-escalation part of a first-in-human study of patients with advanced solid tumors (NCT03444701). Japanese patients ≥20 years of age were enrolled. E7130 was administered intravenously in two cycles: day 1 of a 21-day cycle (Q3W) or days 1 and 15 of a 28-day cycle (Q2W). Doses were escalated from 270 to 550 μg/m Forty-four patients were enrolled: 15 in the E7130 Q3W group and 29 in the Q2W group. Treatment-emergent adverse events (TEAEs) occurred in all patients; the most common TEAE overall was leukopenia (78.6%). Grade 3-4 TEAEs occurred in 93.3% of patients in the Q3W group and 86.2% of patients in the Q2W group. None had a TEAE resulting in study drug discontinuation, and no treatment-related deaths were reported. Per the DLT evaluation, the MTDs were determined as 480 μg/m E7130 480 μg/m

Sections du résumé

BACKGROUND
E7130 is a novel anticancer agent created from a total synthetic study of norhalichondrin B. The authors report the E7130 dose-escalation part of a first-in-human study of patients with advanced solid tumors (NCT03444701).
METHODS
Japanese patients ≥20 years of age were enrolled. E7130 was administered intravenously in two cycles: day 1 of a 21-day cycle (Q3W) or days 1 and 15 of a 28-day cycle (Q2W). Doses were escalated from 270 to 550 μg/m
RESULTS
Forty-four patients were enrolled: 15 in the E7130 Q3W group and 29 in the Q2W group. Treatment-emergent adverse events (TEAEs) occurred in all patients; the most common TEAE overall was leukopenia (78.6%). Grade 3-4 TEAEs occurred in 93.3% of patients in the Q3W group and 86.2% of patients in the Q2W group. None had a TEAE resulting in study drug discontinuation, and no treatment-related deaths were reported. Per the DLT evaluation, the MTDs were determined as 480 μg/m
CONCLUSIONS
E7130 480 μg/m

Identifiants

pubmed: 37080942
doi: 10.1002/cncr.34788
doi:

Substances chimiques

Vascular Endothelial Growth Factor A 0
Antineoplastic Agents 0
Biomarkers 0

Banques de données

ClinicalTrials.gov
['NCT03444701']

Types de publication

Clinical Trial, Phase I Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2348-2359

Informations de copyright

© 2023 The Authors. Cancer published by Wiley Periodicals LLC on behalf of American Cancer Society.

Références

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Auteurs

Toshihiko Doi (T)

Department of Experimental Therapeutics, National Cancer Center Hospital East, Chiba, Japan.

Nobuaki Matsubara (N)

Department of Medical Oncology, National Cancer Center Hospital East, Chiba, Japan.

Yoichi Naito (Y)

Department of Experimental Therapeutics, National Cancer Center Hospital East, Chiba, Japan.
Department of Medical Oncology, National Cancer Center Hospital East, Chiba, Japan.
Department of General Internal Medicine, National Cancer Center Hospital East, Chiba, Japan.

Yasutoshi Kuboki (Y)

Department of Experimental Therapeutics, National Cancer Center Hospital East, Chiba, Japan.

Kenichi Harano (K)

Department of Experimental Therapeutics, National Cancer Center Hospital East, Chiba, Japan.
Department of Medical Oncology, National Cancer Center Hospital East, Chiba, Japan.

Makiko Ono (M)

Department of Medical Oncology, The Cancer Institute Hospital of Japanese Foundation for Cancer Research, Tokyo, Japan.

Tetsuya Urasaki (T)

Department of Medical Oncology, The Cancer Institute Hospital of Japanese Foundation for Cancer Research, Tokyo, Japan.

Akihiro Ohmoto (A)

Department of Medical Oncology, The Cancer Institute Hospital of Japanese Foundation for Cancer Research, Tokyo, Japan.

Tsubasa Kawanai (T)

Japan and Asia Clinical Development Department, Oncology Business Group, Eisai Co., Ltd., Tokyo, Japan.

Takashi Hisai (T)

Oncology Department, Medical Headquarters, Eisai Co., Ltd., Tokyo, Japan.

Hiroki Ikezawa (H)

Clinical Data Science Department, Medicine Development Center, Eisai Co., Ltd., Tokyo, Japan.

Sari Shiba (S)

Clinical Pharmacology Science Department, Medicine Development Center, Eisai Co., Ltd., Tokyo, Japan.

Ken Ito (K)

Oncology Tsukuba Research Development, Discovery, Medicine Creation, Eisai Co., Ltd., Ibaraki, Japan.

Taro Semba (T)

Halichondrin Operation, Discovery, Medicine Creation, Oncology Business Group, Eisai Co., Ltd., Ibaraki, Japan.

Osamu Asano (O)

Halichondrin Operation, Discovery, Medicine Creation, Oncology Business Group, Eisai Co., Ltd., Ibaraki, Japan.

Shunji Takahashi (S)

Department of Medical Oncology, The Cancer Institute Hospital of Japanese Foundation for Cancer Research, Tokyo, Japan.

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