Can Fetuin-A Deficiency Predict the Severity of COVID-19 Disease in Kidney Transplant Recipients?


Journal

Transplantation proceedings
ISSN: 1873-2623
Titre abrégé: Transplant Proc
Pays: United States
ID NLM: 0243532

Informations de publication

Date de publication:
Jun 2023
Historique:
received: 14 12 2022
accepted: 05 01 2023
medline: 3 7 2023
pubmed: 6 5 2023
entrez: 5 5 2023
Statut: ppublish

Résumé

This study aims to investigate whether fetuin A deficiency predicts the prognosis of COVID-19 disease in kidney transplant recipients (KTRs). The study was conducted on 35 hospitalized KTRs with COVID-19 pneumonia between November 2020 and June 2021. Serums were collected for fetuin-A measurement at admission and after six months of follow-up. The demographic and laboratory data of the patients were recorded and analyzed with the appropriate statistical method. A total of 35 KTRs, 23 of which (65.7%) were men, were included in the study. The mean age of the patients was 51.6 ± 14.0 years. Seventeen (48.6%) patients had severe disease criteria and required intensive care (ICU) support. Biopsy-proven acute rejection developed in 6 (17.1%) patients in the follow-up. At admission, the median fetuin-A value was 173.5 mcg/mL (143.5-199.25) in the moderate disease group and 126.0 mcg/mL (89.4-165.5) in the severe patient group (p = 0.005). While the Median fetuin-A value at the time of diagnosis was 173.5 mcg/mL (143.5-199.25), and in the 6th month was 208 mcg/mL [184-229] (p<0.001). By ROC analysis, the effect of serum fetuin-A level in predicting the severity of COVID-19 disease was significant (AUC: 0.771, p = 0.006, 95% CI: 0.615-0.927). When serum fetuin-A cut-off value was taken as 138 mcg/mL to determine disease severity, it was shown to have 83.3% sensitivity and 64.7% specificity. Serum fetuin-A level can predict disease severity in kidney transplant recipients in the presence of active COVID-19 disease.

Sections du résumé

BACKGROUND BACKGROUND
This study aims to investigate whether fetuin A deficiency predicts the prognosis of COVID-19 disease in kidney transplant recipients (KTRs).
METHOD METHODS
The study was conducted on 35 hospitalized KTRs with COVID-19 pneumonia between November 2020 and June 2021. Serums were collected for fetuin-A measurement at admission and after six months of follow-up. The demographic and laboratory data of the patients were recorded and analyzed with the appropriate statistical method.
RESULTS RESULTS
A total of 35 KTRs, 23 of which (65.7%) were men, were included in the study. The mean age of the patients was 51.6 ± 14.0 years. Seventeen (48.6%) patients had severe disease criteria and required intensive care (ICU) support. Biopsy-proven acute rejection developed in 6 (17.1%) patients in the follow-up. At admission, the median fetuin-A value was 173.5 mcg/mL (143.5-199.25) in the moderate disease group and 126.0 mcg/mL (89.4-165.5) in the severe patient group (p = 0.005). While the Median fetuin-A value at the time of diagnosis was 173.5 mcg/mL (143.5-199.25), and in the 6th month was 208 mcg/mL [184-229] (p<0.001). By ROC analysis, the effect of serum fetuin-A level in predicting the severity of COVID-19 disease was significant (AUC: 0.771, p = 0.006, 95% CI: 0.615-0.927). When serum fetuin-A cut-off value was taken as 138 mcg/mL to determine disease severity, it was shown to have 83.3% sensitivity and 64.7% specificity.
CONCLUSIONS CONCLUSIONS
Serum fetuin-A level can predict disease severity in kidney transplant recipients in the presence of active COVID-19 disease.

Identifiants

pubmed: 37147199
pii: S0041-1345(23)00167-7
doi: 10.1016/j.transproceed.2023.01.040
pmc: PMC10028346
pii:
doi:

Substances chimiques

alpha-2-HS-Glycoprotein 0
AHSG protein, human 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1156-1159

Informations de copyright

Copyright © 2023 Elsevier Inc. All rights reserved.

Auteurs

Musa Pinar (M)

Division of Nephrology, Sakarya University Faculty of Medicine, Sakarya, Turkey.

Hamad Dheir (H)

Division of Nephrology, Sakarya University Faculty of Medicine, Sakarya, Turkey.

Aysel Tocoglu (A)

Department of Internal Medicine, Sakarya University Faculty of Medicine, Sakarya, Turkey.

Hande Toptan (H)

Department of Microbiology, Sakarya University Faculty of Medicine, Sakarya, Turkey.

Taner Demirci (T)

Department of Endocrinology, Sakarya University Faculty of Medicine, Sakarya, Turkey.

Selcuk Yaylaci (S)

Department of Internal Medicine, Sakarya University Faculty of Medicine, Sakarya, Turkey.

Gozde Cakirsoy Cakar (GC)

Department of Pathology, Sakarya University Faculty of Medicine, Sakarya, Turkey.

Necattin Firat (N)

Department of General Surgery, Sakarya University Faculty of Medicine, Sakarya, Turkey.

Emrah Akin (E)

Department of General Surgery, Sakarya University Faculty of Medicine, Sakarya, Turkey.

Mahmud İslam (M)

Division of Nephrology, Sakarya University Faculty of Medicine, Sakarya, Turkey. Electronic address: drisleem@gmail.com.

Oguz Karabay (O)

Department of Infectious Diseases and Clinical Microbiology, Sakarya University Faculty of Medicine, Sakarya, Turkey.

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