The potential role of human islet amyloid polypeptide in type 2 diabetes mellitus and Alzheimer's diseases.
Alzheimer’s disease
Human islet amyloid polypeptide insulin resistance
Type 2 diabetes mellitus
Journal
Diabetology & metabolic syndrome
ISSN: 1758-5996
Titre abrégé: Diabetol Metab Syndr
Pays: England
ID NLM: 101488958
Informations de publication
Date de publication:
13 May 2023
13 May 2023
Historique:
received:
01
03
2023
accepted:
08
05
2023
medline:
13
5
2023
pubmed:
13
5
2023
entrez:
12
5
2023
Statut:
epublish
Résumé
Human Islet amyloid polypeptide (hIAPP) from pancreatic β cells in the islet of Langerhans has different physiological functions including inhibiting the release of insulin and glucagon. Type 2 diabetes mellitus (T2DM) is an endocrine disorder due to relative insulin insufficiency and insulin resistance (IR) is associated with increased circulating hIAPP. Remarkably, hIAPP has structural similarity with amyloid beta (Aβ) and can engage in the pathogenesis of T2DM and Alzheimer's disease (AD). Therefore, the present review aimed to elucidate how hIAPP acts as a link between T2DM and AD. IR, aging and low β cell mass increase expression of hIAPP which binds cell membrane leading to the aberrant release of Ca
Identifiants
pubmed: 37173803
doi: 10.1186/s13098-023-01082-1
pii: 10.1186/s13098-023-01082-1
pmc: PMC10182652
doi:
Types de publication
Journal Article
Review
Langues
eng
Pagination
101Informations de copyright
© 2023. The Author(s).
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