Transdiagnostic behavioral and genetic contributors to repetitive negative thinking: A machine learning approach.

Machine learning ensemble method Neuroticism Polygenic risk score Principal component analysis Repetitive negative thinking

Journal

Journal of psychiatric research
ISSN: 1879-1379
Titre abrégé: J Psychiatr Res
Pays: England
ID NLM: 0376331

Informations de publication

Date de publication:
06 2023
Historique:
received: 11 11 2022
revised: 25 04 2023
accepted: 03 05 2023
medline: 29 5 2023
pubmed: 14 5 2023
entrez: 13 5 2023
Statut: ppublish

Résumé

Repetitive negative thinking (RNT) is a symptom that can negatively impact the treatment and course of common psychiatric disorders such as depression and anxiety. We aimed to characterize behavioral and genetic correlates of RNT to infer potential contributors to its genesis and maintenance. We applied a machine learning (ML) ensemble method to define the contribution of fear, interoceptive, reward, and cognitive variables to RNT, along with polygenic risk scores (PRS) for neuroticism, obsessive compulsive disorder (OCD), worry, insomnia, and headaches. We used the PRS and 20 principal components of the behavioral and cognitive variables to predict intensity of RNT. We employed the Tulsa-1000 study, a large database of deeply phenotyped individuals recruited between 2015 and 2018. PRS for neuroticism was the main predictor of RNT intensity (R This study is an exploratory approach that must be validated with a second, independent cohort. Furthermore, this is an association study, limiting causal inference. RNT is highly determined by genetic risk for neuroticism, a behavioral construct that confers risk to a variety of internalizing disorders, and by emotional processing and learning features, including interoceptive aversiveness. These results suggest that targeting emotional and interoceptive processing areas, which involve central autonomic network structures, could be useful in the modulation of RNT intensity.

Sections du résumé

BACKGROUND
Repetitive negative thinking (RNT) is a symptom that can negatively impact the treatment and course of common psychiatric disorders such as depression and anxiety. We aimed to characterize behavioral and genetic correlates of RNT to infer potential contributors to its genesis and maintenance.
METHODS
We applied a machine learning (ML) ensemble method to define the contribution of fear, interoceptive, reward, and cognitive variables to RNT, along with polygenic risk scores (PRS) for neuroticism, obsessive compulsive disorder (OCD), worry, insomnia, and headaches. We used the PRS and 20 principal components of the behavioral and cognitive variables to predict intensity of RNT. We employed the Tulsa-1000 study, a large database of deeply phenotyped individuals recruited between 2015 and 2018.
RESULTS
PRS for neuroticism was the main predictor of RNT intensity (R
LIMITATIONS
This study is an exploratory approach that must be validated with a second, independent cohort. Furthermore, this is an association study, limiting causal inference.
CONCLUSIONS
RNT is highly determined by genetic risk for neuroticism, a behavioral construct that confers risk to a variety of internalizing disorders, and by emotional processing and learning features, including interoceptive aversiveness. These results suggest that targeting emotional and interoceptive processing areas, which involve central autonomic network structures, could be useful in the modulation of RNT intensity.

Identifiants

pubmed: 37178517
pii: S0022-3956(23)00225-X
doi: 10.1016/j.jpsychires.2023.05.039
pii:
doi:

Banques de données

ClinicalTrials.gov
['NCT02450240']

Types de publication

Journal Article Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

207-213

Informations de copyright

Copyright © 2023 Elsevier Ltd. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of competing interest The authors have no competing interests to report.

Auteurs

Katherine L Forthman (KL)

Laureate Institute for Brain Research, 6655 South Yale Avenue, Tulsa, OK, 74136, USA.

Rayus Kuplicki (R)

Laureate Institute for Brain Research, 6655 South Yale Avenue, Tulsa, OK, 74136, USA.

Hung-Wen Yeh (HW)

Laureate Institute for Brain Research, 6655 South Yale Avenue, Tulsa, OK, 74136, USA; Health Services & Outcomes Research, Children's Mercy Research Institute, 2401 Gilham Road, Kansas City, MO, 64108, USA; School of Medicine, University of Missouri-Kansas City, 2411 Holmes St, Kansas City, MO, 64108, USA.

Sahib S Khalsa (SS)

Laureate Institute for Brain Research, 6655 South Yale Avenue, Tulsa, OK, 74136, USA; Oxley College of Health Sciences, University of Tulsa, 1215 South Boulder Ave W, Tulsa, OK, 74119, USA.

Martin P Paulus (MP)

Laureate Institute for Brain Research, 6655 South Yale Avenue, Tulsa, OK, 74136, USA; Oxley College of Health Sciences, University of Tulsa, 1215 South Boulder Ave W, Tulsa, OK, 74119, USA.

Salvador M Guinjoan (SM)

Laureate Institute for Brain Research, 6655 South Yale Avenue, Tulsa, OK, 74136, USA; Department of Psychiatry, Oklahoma University Health Sciences Center, The University of Oklahoma-Tulsa, Schusterman Center, 4502 E. 41st Street, Tulsa, OK, 74135, USA. Electronic address: sguinjoan@laureateinstitute.org.

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