Natural Targeting Potent ROS-Eliminating Tungsten-Based Polyoxometalate Nanodots for Efficient Treatment of Pulmonary Hypertension.

cell proliferation phenotypic switches pulmonary artery smooth muscle cells pulmonary hypertension reactive oxygen species tungsten-based polyoxometalate nanodots

Journal

Advanced healthcare materials
ISSN: 2192-2659
Titre abrégé: Adv Healthc Mater
Pays: Germany
ID NLM: 101581613

Informations de publication

Date de publication:
09 2023
Historique:
revised: 27 04 2023
received: 24 01 2023
medline: 5 12 2023
pubmed: 17 5 2023
entrez: 17 5 2023
Statut: ppublish

Résumé

Pulmonary hypertension (PH) is a disease of pulmonary artery stenosis and blockage caused by abnormal pulmonary artery smooth muscle cells (PASMCs), with high morbidity and mortality. High levels of reactive oxygen species (ROS) in pulmonary arteries play a crucial role in inducing phenotypic switch and abnormal proliferation of PASMCs. However, antioxidants are rarely approved for the treatment of PH because of a lack of targeting and low bioavailability. In this study, the presence of an enhanced permeability and retention effect (EPR)-like effect in the pulmonary arteries of PH is revealed by tissue transmission electron microscopy (TEM). Subsequently, for the first time, tungsten-based polyoxometalate nanodots (WNDs) are developed with potent elimination of multiple ROS for efficient treatment of PH thanks to the high proportion of reduced W

Identifiants

pubmed: 37196347
doi: 10.1002/adhm.202300252
doi:

Substances chimiques

Reactive Oxygen Species 0
polyoxometalate 0
Tungsten V9306CXO6G

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e2300252

Subventions

Organisme : National Natural Science Foundation of China
ID : 82241025
Organisme : National Natural Science Foundation of China
ID : 82173817
Organisme : National Natural Science Foundation of China
ID : 81872872
Organisme : National Natural Science Foundation of China
ID : 81974508
Organisme : National Natural Science Foundation of China
ID : 21974134
Organisme : Hunan Science Fund for Distinguished Young Scholar of China
ID : 2021JJ10067
Organisme : Innovation-Driven Project of Central South University
ID : 202045005
Organisme : Hunan Provincial Natural Science Foundation of China
ID : 2021JJ31066
Organisme : Key Research Project of Ningxia Hui Autonomous Region in 2021 of China
ID : 2021BEG01001
Organisme : Key Program of Ningxia Hui Autonomous Region Natural Science Foundation of China
ID : 2022JJ21059

Commentaires et corrections

Type : ErratumIn

Informations de copyright

© 2023 Wiley-VCH GmbH.

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Auteurs

Hong Liu (H)

Xiangya School of Pharmaceutical Sciences, Central South University, Changsha, 410078, China.

Shuya Wang (S)

Xiangya School of Pharmaceutical Sciences, Central South University, Changsha, 410078, China.

Qiaohui Chen (Q)

Xiangya School of Pharmaceutical Sciences, Central South University, Changsha, 410078, China.

Xiaoyue Ge (X)

Xiangya School of Pharmaceutical Sciences, Central South University, Changsha, 410078, China.

Huang Ning (H)

Xiangya School of Pharmaceutical Sciences, Central South University, Changsha, 410078, China.

Yanzi Guo (Y)

Xiangya School of Pharmaceutical Sciences, Central South University, Changsha, 410078, China.

Di Wang (D)

Xiangya School of Pharmaceutical Sciences, Central South University, Changsha, 410078, China.

Kelong Ai (K)

Xiangya School of Pharmaceutical Sciences, Central South University, Changsha, 410078, China.
Hunan Provincial Key Laboratory of Cardiovascular Research, Changsha, 410078, China.

Changping Hu (C)

Xiangya School of Pharmaceutical Sciences, Central South University, Changsha, 410078, China.
Hunan Provincial Key Laboratory of Cardiovascular Research, Changsha, 410078, China.
Department of Pharmacy, Changzhi Medical College, Changzhi, 046000, China.

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