T2-Fluid-attenuated inversion recovery (FLAIR) pseudoprogression in patients with anaplastic oligodendrogliomas treated with procarbazine, lomustine and vincristine (PCV) chemotherapy alone.


Journal

European journal of neurology
ISSN: 1468-1331
Titre abrégé: Eur J Neurol
Pays: England
ID NLM: 9506311

Informations de publication

Date de publication:
09 2023
Historique:
revised: 10 05 2023
received: 27 02 2023
accepted: 15 05 2023
medline: 8 8 2023
pubmed: 19 5 2023
entrez: 19 5 2023
Statut: ppublish

Résumé

Pseudoprogression in gliomas has been extensively described after radiotherapy with or without chemotherapy, but not after chemotherapy alone. Here we describe the occurrence of pseudoprogression in patients with anaplastic oligodendrogliomas treated with postoperative procarbazine, lomustine and vincristine (PCV) chemotherapy alone. We retrospectively reviewed the medical and radiological files of patients with 1p/19q codeleted, IDH-mutant anaplastic oligodendrogliomas treated with PCV chemotherapy alone who presented magnetic resonance imaging (MRI) modifications suggestive of tumour progression and in whom the final diagnosis was a pseudoprogression. We identified six patients. All patients underwent a surgical resection and were treated with PCV chemotherapy without radiotherapy. After a median of 11 months following the initiation of chemotherapy (range: 3-49 months), the patients developed asymptomatic white matter MRI modifications around the surgical cavity leading to the suspicion of a tumour progression. These modifications appeared as hyperintense on T2-fluid-attenuated inversion recovery (FLAIR) sequence, hypointense on T1 sequence, and lacked mass effect (0/6), contrast enhancement (0/6), restriction on diffusion-weighted imaging (0/4), relative cerebral blood volume (rCBV) increase on perfusion MRI (0/4), and hypermetabolism on Anaplastic oligodendroglioma patients treated with postoperative PCV chemotherapy alone occasionally develop T2/FLAIR hyperintensities around the surgical cavity that can wrongly suggest tumour progression. Multimodal imaging and close follow-up should be considered in this situation.

Sections du résumé

BACKGROUND
Pseudoprogression in gliomas has been extensively described after radiotherapy with or without chemotherapy, but not after chemotherapy alone. Here we describe the occurrence of pseudoprogression in patients with anaplastic oligodendrogliomas treated with postoperative procarbazine, lomustine and vincristine (PCV) chemotherapy alone.
METHODS
We retrospectively reviewed the medical and radiological files of patients with 1p/19q codeleted, IDH-mutant anaplastic oligodendrogliomas treated with PCV chemotherapy alone who presented magnetic resonance imaging (MRI) modifications suggestive of tumour progression and in whom the final diagnosis was a pseudoprogression.
RESULTS
We identified six patients. All patients underwent a surgical resection and were treated with PCV chemotherapy without radiotherapy. After a median of 11 months following the initiation of chemotherapy (range: 3-49 months), the patients developed asymptomatic white matter MRI modifications around the surgical cavity leading to the suspicion of a tumour progression. These modifications appeared as hyperintense on T2-fluid-attenuated inversion recovery (FLAIR) sequence, hypointense on T1 sequence, and lacked mass effect (0/6), contrast enhancement (0/6), restriction on diffusion-weighted imaging (0/4), relative cerebral blood volume (rCBV) increase on perfusion MRI (0/4), and hypermetabolism on
CONCLUSIONS
Anaplastic oligodendroglioma patients treated with postoperative PCV chemotherapy alone occasionally develop T2/FLAIR hyperintensities around the surgical cavity that can wrongly suggest tumour progression. Multimodal imaging and close follow-up should be considered in this situation.

Identifiants

pubmed: 37204066
doi: 10.1111/ene.15873
doi:

Substances chimiques

Lomustine 7BRF0Z81KG
Vincristine 5J49Q6B70F
Procarbazine 35S93Y190K

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2879-2883

Informations de copyright

© 2023 The Authors. European Journal of Neurology published by John Wiley & Sons Ltd on behalf of European Academy of Neurology.

Références

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Auteurs

Inés Esparragosa Vazquez (I)

Neuro-Oncology Department, Hospices Civils of Lyon, Lyon, France.

Mané Ndiaye (M)

Neuro-Oncology Department, Centre Georges François Leclerc, Dijon, France.

Anna Luisa Di Stefano (AL)

Neuro-Oncology Department, Hôpital Foch, Suresnes, France.

Nadia Younan (N)

Neuro-Oncology Department, Hôpital Foch, Suresnes, France.

Delphine Larrieu-Ciron (D)

Neuro-Oncology Department, Institut Universitaire du Cancer de Toulouse, Toulouse, France.

Antoine Seyve (A)

Neuro-Oncology Department, Hospices Civils of Lyon, Lyon, France.

Thiébaud Picart (T)

Neuro-Surgery Department, Hospices Civils of Lyon, Lyon, France.

David Meyronet (D)

Neuropathology Department, Hospices Civils of Lyon, Lyon, France.
Cancer Research Center of Lyon, INSERM U1052, CNRS UMR 5286, Cancer Cell Plasticity Department, Transcriptome Diversity in Stem Cells Laboratory, Lyon, France.

Claire Boutet (C)

Radiology Department, University Hospital of St-Etienne, St-Etienne, France.

François Vassal (F)

Neuro-Surgery Department, University Hospital of St-Etienne, St-Etienne, France.

Catherine Carpentier (C)

Department of Neurology 2-Mazarin, APHP, University Hospital Pitié Salpêtrière-Charles Foix, Paris, France.

Dominique Figarella-Branger (D)

Department of Pathology and Neuropathology, La Timone Hospital, Aix Marseille University, Marseille, France.

Caroline Dehais (C)

Department of Neurology 2-Mazarin, APHP, University Hospital Pitié Salpêtrière-Charles Foix, Paris, France.

Fabien Forest (F)

Department of Pathology, University Hospital of Saint Etienne, Saint Etienne, France.

Romain Rivoirard (R)

Oncology Department, Hôpital Privé de la Loire, Saint Etienne, France.

François Ducray (F)

Neuro-Oncology Department, Hospices Civils of Lyon, Lyon, France.
Cancer Research Center of Lyon, INSERM U1052, CNRS UMR 5286, Cancer Cell Plasticity Department, Transcriptome Diversity in Stem Cells Laboratory, Lyon, France.

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Classifications MeSH