Pembrolizumab alone and pembrolizumab plus chemotherapy in previously treated, extrapulmonary poorly differentiated neuroendocrine carcinomas.


Journal

British journal of cancer
ISSN: 1532-1827
Titre abrégé: Br J Cancer
Pays: England
ID NLM: 0370635

Informations de publication

Date de publication:
08 2023
Historique:
received: 14 11 2022
accepted: 25 04 2023
revised: 04 04 2023
pmc-release: 19 05 2024
medline: 14 7 2023
pubmed: 20 5 2023
entrez: 19 5 2023
Statut: ppublish

Résumé

To date, single-agent immune checkpoint inhibitor (CPI) therapy has proven to be ineffective against biomarker-unselected extrapulmonary poorly differentiated neuroendocrine carcinomas (EP-PDNECs). The efficacy of CPI in combination with chemotherapy remains under investigation. Patients with advanced, progressive EP-PDNECs were enrolled in a two-part study of pembrolizumab-based therapy. In Part A, patients received pembrolizumab alone. In Part B, patients received pembrolizumab plus chemotherapy. objective response rate (ORR). Secondary endpoints: safety, progression-free survival (PFS) and overall survival (OS). Tumours were profiled for programmed death-ligand 1 expression, microsatellite-high/mismatch repair deficient status, mutational burden (TMB), genomic correlates. Tumour growth rate was evaluated. Part A (N = 14): ORR (pembrolizumab alone) 7% (95% CI, 0.2-33.9%), median PFS 1.8 months (95% CI, 1.7-21.4), median OS 7.8 months (95% CI, 3.1-not reached); 14% of patients (N = 2) had grade 3/4 treatment-related adverse events (TRAEs). Part B (N = 22): ORR (pembrolizumab plus chemotherapy) 5% (95% CI, 0-22.8%), median PFS 2.0 months (95% CI, 1.9-3.4), median OS 4.8 months (95% CI, 4.1-8.2); 45% of patients (N = 10) had grade 3/4 TRAEs. The two patients with objective response had high-TMB tumours. Treatment with pembrolizumab alone and pembrolizumab plus chemotherapy was ineffective in advanced, progressive EP-PDNECs. ClinicalTrials.gov NCT03136055.

Sections du résumé

BACKGROUND
To date, single-agent immune checkpoint inhibitor (CPI) therapy has proven to be ineffective against biomarker-unselected extrapulmonary poorly differentiated neuroendocrine carcinomas (EP-PDNECs). The efficacy of CPI in combination with chemotherapy remains under investigation.
METHODS
Patients with advanced, progressive EP-PDNECs were enrolled in a two-part study of pembrolizumab-based therapy. In Part A, patients received pembrolizumab alone. In Part B, patients received pembrolizumab plus chemotherapy.
PRIMARY ENDPOINT
objective response rate (ORR). Secondary endpoints: safety, progression-free survival (PFS) and overall survival (OS). Tumours were profiled for programmed death-ligand 1 expression, microsatellite-high/mismatch repair deficient status, mutational burden (TMB), genomic correlates. Tumour growth rate was evaluated.
RESULTS
Part A (N = 14): ORR (pembrolizumab alone) 7% (95% CI, 0.2-33.9%), median PFS 1.8 months (95% CI, 1.7-21.4), median OS 7.8 months (95% CI, 3.1-not reached); 14% of patients (N = 2) had grade 3/4 treatment-related adverse events (TRAEs). Part B (N = 22): ORR (pembrolizumab plus chemotherapy) 5% (95% CI, 0-22.8%), median PFS 2.0 months (95% CI, 1.9-3.4), median OS 4.8 months (95% CI, 4.1-8.2); 45% of patients (N = 10) had grade 3/4 TRAEs. The two patients with objective response had high-TMB tumours.
DISCUSSION
Treatment with pembrolizumab alone and pembrolizumab plus chemotherapy was ineffective in advanced, progressive EP-PDNECs.
CLINICAL TRIAL REGISTRATION
ClinicalTrials.gov NCT03136055.

Identifiants

pubmed: 37208512
doi: 10.1038/s41416-023-02298-8
pii: 10.1038/s41416-023-02298-8
pmc: PMC10338510
doi:

Substances chimiques

Antibodies, Monoclonal, Humanized 0
pembrolizumab DPT0O3T46P

Banques de données

ClinicalTrials.gov
['NCT03136055']

Types de publication

Clinical Trial Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

291-300

Subventions

Organisme : NCI NIH HHS
ID : P30 CA006516
Pays : United States
Organisme : NCI NIH HHS
ID : P30 CA008748
Pays : United States
Organisme : NCI NIH HHS
ID : P30 CA082103
Pays : United States

Informations de copyright

© 2023. The Author(s), under exclusive licence to Springer Nature Limited.

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Auteurs

Nitya Raj (N)

Memorial Sloan Kettering (MSK) Cancer Center, New York, NY, USA. rajn@mskcc.org.

Jennifer A Chan (JA)

Dana-Farber Cancer Institute, Boston, MA, USA.

Stephanie J Wang (SJ)

University of California San Francisco (UCSF), San Francisco, CA, USA.

Rahul R Aggarwal (RR)

University of California San Francisco (UCSF), San Francisco, CA, USA.

Susan Calabrese (S)

University of California San Francisco (UCSF), San Francisco, CA, USA.

April DeMore (A)

Memorial Sloan Kettering (MSK) Cancer Center, New York, NY, USA.

Lawrence Fong (L)

University of California San Francisco (UCSF), San Francisco, CA, USA.

Jennifer Grabowsky (J)

University of California San Francisco (UCSF), San Francisco, CA, USA.

Thomas A Hope (TA)

University of California San Francisco (UCSF), San Francisco, CA, USA.

Kanti Pallav Kolli (KP)

University of California San Francisco (UCSF), San Francisco, CA, USA.

Claire K Mulvey (CK)

University of California San Francisco (UCSF), San Francisco, CA, USA.

Pamela N Munster (PN)

University of California San Francisco (UCSF), San Francisco, CA, USA.

Kimberly Perez (K)

Dana-Farber Cancer Institute, Boston, MA, USA.

Sippy Punn (S)

Memorial Sloan Kettering (MSK) Cancer Center, New York, NY, USA.

Diane Reidy-Lagunes (D)

Memorial Sloan Kettering (MSK) Cancer Center, New York, NY, USA.

Sofia Von Fedak (S)

Dana-Farber Cancer Institute, Boston, MA, USA.

Li Zhang (L)

University of California San Francisco (UCSF), San Francisco, CA, USA.

Emily K Bergsland (EK)

University of California San Francisco (UCSF), San Francisco, CA, USA. emily.bergsland@ucsf.edu.

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