A case of herpes simplex virus induced peripheral neuropathy and encephalitis with positive GM3 and CASPR2 antibody.


Journal

BMC neurology
ISSN: 1471-2377
Titre abrégé: BMC Neurol
Pays: England
ID NLM: 100968555

Informations de publication

Date de publication:
20 May 2023
Historique:
received: 10 11 2022
accepted: 04 05 2023
medline: 22 5 2023
pubmed: 21 5 2023
entrez: 20 5 2023
Statut: epublish

Résumé

We reported on a case involving an older patient with HSV-1 encephalitis who simultaneously experienced the onset of peripheral nerve symptoms associated with the presence of anti-GM3 immunoglobulin G (IgG). A 77-year-old male was admitted to hospital with high fever, weakness of both lower limbs, and an unstable gait. A CSF test revealed a strikingly increased protein level (1,002 mg/L, normative values: 150-450 mg/L) and MRI revealed hyper-signal lesions in the right temporal lobe, right hippocampus, right insula, and right cingulate gyrus. The CSF was positive for HSV PCR (HSV-1,17870). In addition, the serum samples were positive for CASPR2 antibodies (antibody titer: 1/10) and anti-GM3 immunoglobulin G (IgG) (+). The patient was diagnosed with HSV-1-induced peripheral nerve symptoms that were associated with encephalitis and the presence of anti-GM3 IgG and anti-CASPR2 antibodies. The patient had received included intravenous immunoglobulin, intravenous acyclovir, and corticosteroids therapy. At the one-year follow-up examination, he had regained the necessary skills associated with daily life. Herpes simplex virus infection often induces encephalitis, and reaction to the virus may trigger an autoimmune response. Early diagnosis and treatment can avoid the progression of the disease to include autoimmune encephalitis.

Sections du résumé

BACKGROUND BACKGROUND
We reported on a case involving an older patient with HSV-1 encephalitis who simultaneously experienced the onset of peripheral nerve symptoms associated with the presence of anti-GM3 immunoglobulin G (IgG).
CASE PRESENTATION METHODS
A 77-year-old male was admitted to hospital with high fever, weakness of both lower limbs, and an unstable gait. A CSF test revealed a strikingly increased protein level (1,002 mg/L, normative values: 150-450 mg/L) and MRI revealed hyper-signal lesions in the right temporal lobe, right hippocampus, right insula, and right cingulate gyrus. The CSF was positive for HSV PCR (HSV-1,17870). In addition, the serum samples were positive for CASPR2 antibodies (antibody titer: 1/10) and anti-GM3 immunoglobulin G (IgG) (+). The patient was diagnosed with HSV-1-induced peripheral nerve symptoms that were associated with encephalitis and the presence of anti-GM3 IgG and anti-CASPR2 antibodies. The patient had received included intravenous immunoglobulin, intravenous acyclovir, and corticosteroids therapy. At the one-year follow-up examination, he had regained the necessary skills associated with daily life.
CONCLUSIONS CONCLUSIONS
Herpes simplex virus infection often induces encephalitis, and reaction to the virus may trigger an autoimmune response. Early diagnosis and treatment can avoid the progression of the disease to include autoimmune encephalitis.

Identifiants

pubmed: 37210504
doi: 10.1186/s12883-023-03238-y
pii: 10.1186/s12883-023-03238-y
pmc: PMC10199470
doi:

Substances chimiques

Acyclovir X4HES1O11F
Immunoglobulin G 0

Types de publication

Case Reports Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

199

Informations de copyright

© 2023. The Author(s).

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Auteurs

Hongji Lu (H)

The Neurological Intensive Care Unit of Guangdong Provincial Hospital of Chinese Medicine, Guangzhou, 510120, China.
The Second Clinical School, Guangzhou University of Chinese Medicine, Guangzhou, 510120, China.

Yingdi Liao (Y)

The Rehabilitation Department, Kunming Municipal Hospital of Traditional Chinese Medicine, Kunming, 650000, China.

Changlin Zhang (C)

The Second Clinical School, Guangzhou University of Chinese Medicine, Guangzhou, 510120, China.
Department of Stroke Center, The Second Affiliated Hospital of Guangzhou University of Chinese Medicine, Guangzhou, 510120, China.

Wanxin Wen (W)

The Neurological Intensive Care Unit of Guangdong Provincial Hospital of Chinese Medicine, Guangzhou, 510120, China.
The Second Clinical School, Guangzhou University of Chinese Medicine, Guangzhou, 510120, China.

Yaming Du (Y)

The Second Clinical School, Guangzhou University of Chinese Medicine, Guangzhou, 510120, China.

Min Zhao (M)

The Second Clinical School, Guangzhou University of Chinese Medicine, Guangzhou, 510120, China.
The Encephallopathy Department.1 of Guangdong Provincial Hospital of Chinese Medicine, Guangzhou, 510120, China.

Lixin Wang (L)

The Neurological Intensive Care Unit of Guangdong Provincial Hospital of Chinese Medicine, Guangzhou, 510120, China. plawlx@gzucm.edu.cn.
The Second Clinical School, Guangzhou University of Chinese Medicine, Guangzhou, 510120, China. plawlx@gzucm.edu.cn.

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