The lower limit of reactivity as a potential individualised cerebral perfusion pressure target in traumatic brain injury: a CENTER-TBI high-resolution sub-study analysis.

Cerebral autoregulation Individualised cerebral perfusion pressure Lower limit of reactivity Traumatic brain injury

Journal

Critical care (London, England)
ISSN: 1466-609X
Titre abrégé: Crit Care
Pays: England
ID NLM: 9801902

Informations de publication

Date de publication:
20 05 2023
Historique:
received: 23 01 2023
accepted: 11 05 2023
medline: 22 5 2023
pubmed: 21 5 2023
entrez: 20 5 2023
Statut: epublish

Résumé

A previous retrospective single-centre study suggested that the percentage of time spent with cerebral perfusion pressure (CPP) below the individual lower limit of reactivity (LLR) is associated with mortality in traumatic brain injury (TBI) patients. We aim to validate this in a large multicentre cohort. Recordings from 171 TBI patients from the high-resolution cohort of the CENTER-TBI study were processed with ICM+ software. We derived LLR as a time trend of CPP at a level for which the pressure reactivity index (PRx) indicates impaired cerebrovascular reactivity with low CPP. The relationship with mortality was assessed with Mann-U test (first 7-day period), Kruskal-Wallis (daily analysis for 7 days), univariate and multivariate logistic regression models. AUCs (CI 95%) were calculated and compared using DeLong's test. Average LLR over the first 7 days was above 60 mmHg in 48% of patients. %time with CPP < LLR could predict mortality (AUC 0.73, p =  < 0.001). This association becomes significant starting from the third day post injury. The relationship was maintained when correcting for IMPACT covariates or for high ICP. Using a multicentre cohort, we confirmed that CPP below LLR was associated with mortality during the first seven days post injury.

Sections du résumé

BACKGROUND
A previous retrospective single-centre study suggested that the percentage of time spent with cerebral perfusion pressure (CPP) below the individual lower limit of reactivity (LLR) is associated with mortality in traumatic brain injury (TBI) patients. We aim to validate this in a large multicentre cohort.
METHODS
Recordings from 171 TBI patients from the high-resolution cohort of the CENTER-TBI study were processed with ICM+ software. We derived LLR as a time trend of CPP at a level for which the pressure reactivity index (PRx) indicates impaired cerebrovascular reactivity with low CPP. The relationship with mortality was assessed with Mann-U test (first 7-day period), Kruskal-Wallis (daily analysis for 7 days), univariate and multivariate logistic regression models. AUCs (CI 95%) were calculated and compared using DeLong's test.
RESULTS
Average LLR over the first 7 days was above 60 mmHg in 48% of patients. %time with CPP < LLR could predict mortality (AUC 0.73, p =  < 0.001). This association becomes significant starting from the third day post injury. The relationship was maintained when correcting for IMPACT covariates or for high ICP.
CONCLUSIONS
Using a multicentre cohort, we confirmed that CPP below LLR was associated with mortality during the first seven days post injury.

Identifiants

pubmed: 37210526
doi: 10.1186/s13054-023-04485-8
pii: 10.1186/s13054-023-04485-8
pmc: PMC10199598
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

194

Subventions

Organisme : Medical Research Council
ID : MR N013433-1
Pays : United Kingdom

Investigateurs

Audny Anke (A)
Ronny Beer (R)
Bo-Michael Bellander (BM)
Andras Buki (A)
Manuel Cabeleira (M)
Marco Carbonara (M)
Arturo Chieregato (A)
Giuseppe Citerio (G)
Hans Clusmann (H)
Endre Czeiter (E)
Bart Depreitere (B)
Shirin Frisvold (S)
Raimund Helbok (R)
Stefan Jankowski (S)
Daniel Kondziella (D)
Lars-Owe Koskinen (LO)
Ana Kowark (A)
Geert Meyfroidt (G)
Kirsten Moeller (K)
David Nelson (D)
Anna Piippo-Karjalainen (A)
Andreea Radoi (A)
Arminas Ragauskas (A)
Rahul Raj (R)
Jonathan Rhodes (J)
Saulius Rocka (S)
Rolf Rossaint (R)
Juan Sahuquillo (J)
Oliver Sakowitz (O)
Nina Sundström (N)
Riikka Takala (R)
Tomas Tamosuitis (T)
Olli Tenovuo (O)
Andreas Unterberg (A)
Peter Vajkoczy (P)
Alessia Vargiolu (A)
Rimantas Vilcinis (R)
Stefan Wolf (S)
Alexander Younsi (A)

Commentaires et corrections

Type : CommentIn

Informations de copyright

© 2023. The Author(s).

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Auteurs

Erta Beqiri (E)

Brain Physics Laboratory, Division of Neurosurgery, Department of Clinical Neurosciences, University of Cambridge, Cambridge, UK. vb391@cam.ac.uk.

Frederick A Zeiler (FA)

Department of Surgery, Rady Faculty of Health Sciences, University of Manitoba, Winnipeg, MB, Canada.
Section of Neurosurgery, Department of Surgery, Rady Faculty of Health Sciences, University of Manitoba, Winnipeg, MB, Canada.
Department of Human Anatomy and Cell Science, Rady Faculty of Health Sciences, University of Manitoba, Winnipeg, Canada.
Biomedical Engineering, Price Faculty of Engineering, University of Manitoba, Winnipeg, Canada.
Division of Anaesthesia, Department of Medicine, University of Cambridge, Cambridge, UK.
Department of Clinical Neuroscience, Karolinska Intitutet, Stockholm, Sweden.

Ari Ercole (A)

Division of Anaesthesia, Department of Medicine, University of Cambridge, Cambridge, UK.

Michal M Placek (MM)

Brain Physics Laboratory, Division of Neurosurgery, Department of Clinical Neurosciences, University of Cambridge, Cambridge, UK.

Jeanette Tas (J)

School for Mental Health and Neuroscience (MHeNS), University Maastricht, Maastricht, The Netherlands.
Department of Intensive Care Medicine, Maastricht University Medical Center+, Maastricht, The Netherlands.

Joseph Donnelly (J)

Brain Physics Laboratory, Division of Neurosurgery, Department of Clinical Neurosciences, University of Cambridge, Cambridge, UK.
Department of Surgery, Rady Faculty of Health Sciences, University of Manitoba, Winnipeg, MB, Canada.
Section of Neurosurgery, Department of Surgery, Rady Faculty of Health Sciences, University of Manitoba, Winnipeg, MB, Canada.
Department of Human Anatomy and Cell Science, Rady Faculty of Health Sciences, University of Manitoba, Winnipeg, Canada.
Biomedical Engineering, Price Faculty of Engineering, University of Manitoba, Winnipeg, Canada.
Division of Anaesthesia, Department of Medicine, University of Cambridge, Cambridge, UK.
Department of Clinical Neuroscience, Karolinska Intitutet, Stockholm, Sweden.
School for Mental Health and Neuroscience (MHeNS), University Maastricht, Maastricht, The Netherlands.
Department of Intensive Care Medicine, Maastricht University Medical Center+, Maastricht, The Netherlands.
Department of Medicine, University of Auckland, Auckland, New Zealand.

Marcel J H Aries (MJH)

School for Mental Health and Neuroscience (MHeNS), University Maastricht, Maastricht, The Netherlands.
Department of Intensive Care Medicine, Maastricht University Medical Center+, Maastricht, The Netherlands.

Peter J Hutchinson (PJ)

Division of Neurosurgery, Department of Clinical Neurosciences, Addenbrooke's Hospital and University of Cambridge, Cambridge, CB2 0QQ, UK.

David Menon (D)

Division of Anaesthesia, Department of Medicine, University of Cambridge, Cambridge, UK.

Nino Stocchetti (N)

Neuroscience Intensive Care Unit, Department of Anaesthesia and Critical Care, Fondazione IRCCS Ca' Granda-Ospedale Maggiore Policlinico, Milan, Italy.
Department of Pathophysiology and Transplants, University of Milan, Milan, Italy.

Marek Czosnyka (M)

Brain Physics Laboratory, Division of Neurosurgery, Department of Clinical Neurosciences, University of Cambridge, Cambridge, UK.

Peter Smielewski (P)

Brain Physics Laboratory, Division of Neurosurgery, Department of Clinical Neurosciences, University of Cambridge, Cambridge, UK.

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