Pathological overview of steatohepatitic hepatocellular carcinoma in a surgical series.


Journal

Histopathology
ISSN: 1365-2559
Titre abrégé: Histopathology
Pays: England
ID NLM: 7704136

Informations de publication

Date de publication:
Oct 2023
Historique:
revised: 29 03 2023
received: 15 02 2023
accepted: 01 05 2023
medline: 13 9 2023
pubmed: 24 5 2023
entrez: 24 5 2023
Statut: ppublish

Résumé

According to the last WHO classification, steatohepatitic hepatocellular carcinoma (SH-HCC) is recognized as a distinct HCC subtype, even though a consensual definition is still lacking. The objectives of the study were to carefully describe the morphological features of SH-HCC and evaluate its impact on prognosis. We conducted a single-centre retrospective study including 297 surgically resected HCC. Pathological features including SH criteria (steatosis, ballooning, Mallory-Denk bodies, fibrosis, and inflammation) were assessed. SH-HCC was defined by the presence of at least four of the five SH criteria and the SH component represented >50% of the tumour area. According to this definition, 39 (13%) HCC cases corresponded to SH-HCC and 30 cases (10%) corresponded to HCC with an SH component (<50%). SH criteria in SH-HCC and non-SH-HCC were distributed as follows: ballooning (100% versus 11%), fibrosis (100% versus 81%), inflammation (100% versus 67%), steatosis (92% versus 8%), and Mallory-Denk bodies (74% versus 3%). Inflammation markers (c-reactive protein [CRP] and serum amyloid A [SAA]) were significantly more expressed in SH-HCC compared to non-SH-HCC (82% versus 14%, P = <0.001). Five-year recurrence-free survival (RFS) and 5-year overall survival (OS) were similar for SH-HCC and non-SH-HCC (P = 0.413 and P = 0.866, respectively). The percentage of SH component does not impact OS and RFS. We confirm in a large cohort the relatively high prevalence (13%) of SH-HCC. Ballooning is the most specific criteria for this subtype. The percentage of the SH component does not impact prognosis.

Identifiants

pubmed: 37222200
doi: 10.1111/his.14941
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

526-537

Informations de copyright

© 2023 John Wiley & Sons Ltd.

Références

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Auteurs

Loïc Trapani (L)

Université Paris Cité, Paris, France.
AP-HP.Nord, Department of Pathology, FHU MOSAIC, Beaujon Hospital, Clichy, France.

Aurélie Beaufrère (A)

Université Paris Cité, Paris, France.
AP-HP.Nord, Department of Pathology, FHU MOSAIC, Beaujon Hospital, Clichy, France.
Centre de Recherche sur l'Inflammation, INSERM UMR 1149, Paris, France.

Christian Hobeika (C)

AP-HP, Department of HPB and digestive surgery, Pitié-Salpétrière Hospital, Paris, France.

Tatiana Codjia (T)

Centre de Recherche sur l'Inflammation, INSERM UMR 1149, Paris, France.
AP-HP.Nord, Department of HPB surgery, Beaujon Hospital, Clichy, France.

Miguel Albuquerque (M)

AP-HP.Nord, Department of Pathology, FHU MOSAIC, Beaujon Hospital, Clichy, France.
Centre de Recherche sur l'Inflammation, INSERM UMR 1149, Paris, France.

Mohamed Bouattour (M)

Centre de Recherche sur l'Inflammation, INSERM UMR 1149, Paris, France.
AP-HP.Nord, Department of Hepatology, Beaujon Hospital, Clichy, France.

Mickael Lesurtel (M)

Université Paris Cité, Paris, France.
AP-HP.Nord, Department of HPB surgery, Beaujon Hospital, Clichy, France.

François Cauchy (F)

Centre de Recherche sur l'Inflammation, INSERM UMR 1149, Paris, France.

Valérie Paradis (V)

Université Paris Cité, Paris, France.
AP-HP.Nord, Department of Pathology, FHU MOSAIC, Beaujon Hospital, Clichy, France.
Centre de Recherche sur l'Inflammation, INSERM UMR 1149, Paris, France.

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