Predictors of target lesion failure after treatment of left main, bifurcation, or chronic total occlusion lesions with ultrathin-strut drug-eluting coronary stents in the ULTRA registry.


Journal

Catheterization and cardiovascular interventions : official journal of the Society for Cardiac Angiography & Interventions
ISSN: 1522-726X
Titre abrégé: Catheter Cardiovasc Interv
Pays: United States
ID NLM: 100884139

Informations de publication

Date de publication:
08 2023
Historique:
revised: 18 04 2023
received: 19 01 2023
accepted: 13 05 2023
medline: 7 8 2023
pubmed: 26 5 2023
entrez: 26 5 2023
Statut: ppublish

Résumé

Data about the long-term performance of new-generation ultrathin-strut drug-eluting stents (DES) in challenging coronary lesions, such as left main (LM), bifurcation, and chronic total occlusion (CTO) lesions are scant. The international multicenter retrospective observational ULTRA study included consecutive patients treated from September 2016 to August 2021 with ultrathin-strut (<70 µm) DES in challenging de novo lesions. Primary endpoint was target lesion failure (TLF): composite of cardiac death, target-lesion revascularization (TLR), target-vessel myocardial infarction (TVMI), or definite stent thrombosis (ST). Secondary endpoints included all-cause death, acute myocardial infarction (AMI), target vessel revascularization, and TLF components. TLF predictors were assessed with Cox multivariable analysis. Of 1801 patients (age: 66.6 ± 11.2 years; male: 1410 [78.3%]), 170 (9.4%) experienced TLF during follow-up of 3.1 ± 1.4 years. In patients with LM, CTO, and bifurcation lesions, TLF rates were 13.5%, 9.9%, and 8.9%, respectively. Overall, 160 (8.9%) patients died (74 [4.1%] from cardiac causes). AMI and TVMI rates were 6.0% and 3.2%, respectively. ST occurred in 11 (1.1%) patients while 77 (4.3%) underwent TLR. Multivariable analysis identified the following predictors of TLF: age, STEMI with cardiogenic shock, impaired left ventricular ejection fraction, diabetes, and renal dysfunction. Among the procedural variables, total stent length increased TLF risk (HR: 1.01, 95% CI: 1-1.02 per mm increase), while intracoronary imaging reduced the risk substantially (HR: 0.35, 95% CI: 0.12-0.82). Ultrathin-strut DES showed high efficacy and satisfactory safety, even in patients with challenging coronary lesions. Yet, despite using contemporary gold-standard DES, the association persisted between established patient- and procedure-related features of risk and impaired 3-year clinical outcome.

Sections du résumé

BACKGROUND
Data about the long-term performance of new-generation ultrathin-strut drug-eluting stents (DES) in challenging coronary lesions, such as left main (LM), bifurcation, and chronic total occlusion (CTO) lesions are scant.
METHODS
The international multicenter retrospective observational ULTRA study included consecutive patients treated from September 2016 to August 2021 with ultrathin-strut (<70 µm) DES in challenging de novo lesions. Primary endpoint was target lesion failure (TLF): composite of cardiac death, target-lesion revascularization (TLR), target-vessel myocardial infarction (TVMI), or definite stent thrombosis (ST). Secondary endpoints included all-cause death, acute myocardial infarction (AMI), target vessel revascularization, and TLF components. TLF predictors were assessed with Cox multivariable analysis.
RESULTS
Of 1801 patients (age: 66.6 ± 11.2 years; male: 1410 [78.3%]), 170 (9.4%) experienced TLF during follow-up of 3.1 ± 1.4 years. In patients with LM, CTO, and bifurcation lesions, TLF rates were 13.5%, 9.9%, and 8.9%, respectively. Overall, 160 (8.9%) patients died (74 [4.1%] from cardiac causes). AMI and TVMI rates were 6.0% and 3.2%, respectively. ST occurred in 11 (1.1%) patients while 77 (4.3%) underwent TLR. Multivariable analysis identified the following predictors of TLF: age, STEMI with cardiogenic shock, impaired left ventricular ejection fraction, diabetes, and renal dysfunction. Among the procedural variables, total stent length increased TLF risk (HR: 1.01, 95% CI: 1-1.02 per mm increase), while intracoronary imaging reduced the risk substantially (HR: 0.35, 95% CI: 0.12-0.82).
CONCLUSIONS
Ultrathin-strut DES showed high efficacy and satisfactory safety, even in patients with challenging coronary lesions. Yet, despite using contemporary gold-standard DES, the association persisted between established patient- and procedure-related features of risk and impaired 3-year clinical outcome.

Identifiants

pubmed: 37232278
doi: 10.1002/ccd.30696
doi:

Substances chimiques

Sirolimus W36ZG6FT64

Types de publication

Multicenter Study Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

221-232

Informations de copyright

© 2023 Wiley Periodicals LLC.

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Auteurs

Ovidio de Filippo (O)

Cardiovascular and Thoracic Department, Division of Cardiology, A. O. U. Città della Salute e della Scienza, Turin, Italy.
Department of Biomedical Sciences, University of Turin, Turin, Italy.

Francesco Bruno (F)

Cardiovascular and Thoracic Department, Division of Cardiology, A. O. U. Città della Salute e della Scienza, Turin, Italy.
Department of Biomedical Sciences, University of Turin, Turin, Italy.

Tineke H Pinxterhuis (TH)

Department of Cardiology, Thoraxcentrum Twente, Medisch Spectrum Twente, Enschede, The Netherlands.
Department of Health Technology and Services Research, Faculty BMS, Technical Medical Centre, University of Twente, Enschede, The Netherlands.

Mariusz Gąsior (M)

Faculty of Medical Sciences in Zabrze, Medical University of Silesia, Katowice, Poland.

Leor Perl (L)

Cardiology Department, Rabin Medical Center, Beilinson Hospital, Petah Tikva, Israel.

Luca Gaido (L)

Division of Cardiology, Ospedale Maria Vittoria, Torino, Italy.

Domenico Tuttolomondo (D)

Cardiology Unit, University Hospital of Parma, Parma, Italy.

Antonio Greco (A)

Division of Cardiology, Azienda Ospedaliero Universitaria Policlinico "G. Rodolico-San Marco", University of Catania, Catania, Italy.

Roberto Verardi (R)

Division of Cardiology, Ospedale Maggiore Carlo Alberto Pizzardi, Bologna, Italy.

Gianluca Lo Martire (G)

Cardiovascular and Thoracic Department, Division of Cardiology, A. O. U. Città della Salute e della Scienza, Turin, Italy.
Department of Biomedical Sciences, University of Turin, Turin, Italy.

Mario Iannaccone (M)

Division of Cardiology, Ospedale San Giovanni Bosco, Turin, Italy.

Attilio Leone (A)

Department of Advanced Biomedical Sciences, Division of Cardiology, University of Naples Federico II, Naples, Italy.

Gaetano Liccardo (G)

Department of Biomedical Sciences Pieve Emanuele, Humanitas University, Milan, Italy.

Serena Caglioni (S)

Cardiology Unit, Azienda Ospedaliero Universitaria Di Ferrara, Ferrara, Italy.

Rocio González Ferreiro (R)

Cardiology Department, University Hospital Alvaro Cunqueiro, Galicia Sur Health Research Institute (IISGS), Pontevedra, Spain.

Giulio Rodinò (G)

Department of Cardiac, Thoracic, Vascular Sciences and Public Health, University of Padua Medical School, Padova, Italy.

Giuseppe Musumeci (G)

Cardiology Department, Azienda Ospedaliera Ordine Mauriziano Umberto I, Turin, Italy.

Giuseppe Patti (G)

Department of Translational Medicine, Maggiore della Carità Hospital, University of Eastern Piedmont, Novara, Italy.

Irene Borzillo (I)

Cardiovascular and Thoracic Department, Division of Cardiology, A. O. U. Città della Salute e della Scienza, Turin, Italy.
Department of Biomedical Sciences, University of Turin, Turin, Italy.

Giuseppe Tarantini (G)

Department of Cardiac, Thoracic, Vascular Sciences and Public Health, University of Padua Medical School, Padova, Italy.

Wojciech Wańha (W)

Department of Cardiology and Structural Heart Diseases, Medical University of Silesia, Katowice, Poland.

Bruno Casella (B)

Department of Computer Science, University of Turin, Turin, Italy.

Eline H Ploumen (EH)

Department of Cardiology, Thoraxcentrum Twente, Medisch Spectrum Twente, Enschede, The Netherlands.
Department of Health Technology and Services Research, Faculty BMS, Technical Medical Centre, University of Twente, Enschede, The Netherlands.

Łukasz Pyka (Ł)

Faculty of Medical Sciences in Zabrze, Medical University of Silesia, Katowice, Poland.

Ran Kornowski (R)

Cardiology Department, Rabin Medical Center, Beilinson Hospital, Petah Tikva, Israel.

Andrea Gagnor (A)

Division of Cardiology, Ospedale Maria Vittoria, Torino, Italy.

Raffaele Piccolo (R)

Department of Advanced Biomedical Sciences, Division of Cardiology, University of Naples Federico II, Naples, Italy.

Sergio Raposeiras Roubin (SR)

Cardiology Department, University Hospital Alvaro Cunqueiro, Galicia Sur Health Research Institute (IISGS), Pontevedra, Spain.

Davide Capodanno (D)

Division of Cardiology, Azienda Ospedaliero Universitaria Policlinico "G. Rodolico-San Marco", University of Catania, Catania, Italy.

Paolo Zocca (P)

Department of Cardiology, Thoraxcentrum Twente, Medisch Spectrum Twente, Enschede, The Netherlands.
Department of Health Technology and Services Research, Faculty BMS, Technical Medical Centre, University of Twente, Enschede, The Netherlands.

Federico Conrotto (F)

Cardiovascular and Thoracic Department, Division of Cardiology, A. O. U. Città della Salute e della Scienza, Turin, Italy.
Department of Biomedical Sciences, University of Turin, Turin, Italy.

Gaetano M De Ferrari (GM)

Cardiovascular and Thoracic Department, Division of Cardiology, A. O. U. Città della Salute e della Scienza, Turin, Italy.
Department of Biomedical Sciences, University of Turin, Turin, Italy.

Clemens von Birgelen (C)

Department of Cardiology, Thoraxcentrum Twente, Medisch Spectrum Twente, Enschede, The Netherlands.
Department of Health Technology and Services Research, Faculty BMS, Technical Medical Centre, University of Twente, Enschede, The Netherlands.

Fabrizio D'Ascenzo (F)

Cardiovascular and Thoracic Department, Division of Cardiology, A. O. U. Città della Salute e della Scienza, Turin, Italy.
Department of Biomedical Sciences, University of Turin, Turin, Italy.

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