Contribution of Genetic Test to Early Diagnosis of Methylenetetrahydrofolate Reductase (MTHFR) Deficiency: The Experience of a Reference Center in Southern Italy.

genetic test hereditary metabolic diseases homocysteine methionine methylenetetrahydrofolate reductase (MTHFR) deficiency newborn screening (NBS) for inborn error of metabolism

Journal

Genes
ISSN: 2073-4425
Titre abrégé: Genes (Basel)
Pays: Switzerland
ID NLM: 101551097

Informations de publication

Date de publication:
26 04 2023
Historique:
received: 21 03 2023
revised: 21 04 2023
accepted: 24 04 2023
medline: 29 5 2023
pubmed: 27 5 2023
entrez: 27 5 2023
Statut: epublish

Résumé

the deficiency of 5,10-Methylenetetrahydrofolate reductase (MTHFR) constitutes a rare and severe metabolic disease and is included in most expanded newborn screening (NBS) programs worldwide. Patients with severe MTHFR deficiency develop neurological disorders and premature vascular disease. Timely diagnosis through NBS allows early treatment, resulting in improved outcomes. we report the diagnostic yield of genetic testing for MTHFR deficiency diagnosis, in a reference Centre of Southern Italy between 2017 and 2022. MTHFR deficiency was suspected in four newborns showing hypomethioninemia and hyperhomocysteinemia; otherwise, one patient born in pre-screening era showed clinical symptoms and laboratory signs that prompted to perform genetic testing for MTHFR deficiency. molecular analysis of the our results strongly support the need for genetic testing to quickly support the definitive diagnosis of MTHFR deficiency and start therapy. Furthermore, our study extends knowledge of the molecular epidemiology of MTHFR deficiency by identifying a novel mutation in the

Sections du résumé

BACKGROUND
the deficiency of 5,10-Methylenetetrahydrofolate reductase (MTHFR) constitutes a rare and severe metabolic disease and is included in most expanded newborn screening (NBS) programs worldwide. Patients with severe MTHFR deficiency develop neurological disorders and premature vascular disease. Timely diagnosis through NBS allows early treatment, resulting in improved outcomes.
METHODS
we report the diagnostic yield of genetic testing for MTHFR deficiency diagnosis, in a reference Centre of Southern Italy between 2017 and 2022. MTHFR deficiency was suspected in four newborns showing hypomethioninemia and hyperhomocysteinemia; otherwise, one patient born in pre-screening era showed clinical symptoms and laboratory signs that prompted to perform genetic testing for MTHFR deficiency.
RESULTS
molecular analysis of the
CONCLUSIONS
our results strongly support the need for genetic testing to quickly support the definitive diagnosis of MTHFR deficiency and start therapy. Furthermore, our study extends knowledge of the molecular epidemiology of MTHFR deficiency by identifying a novel mutation in the

Identifiants

pubmed: 37239340
pii: genes14050980
doi: 10.3390/genes14050980
pmc: PMC10218448
pii:
doi:

Substances chimiques

Methylenetetrahydrofolate Reductase (NADPH2) EC 1.5.1.20
MTHFR protein, human EC 1.5.1.20

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

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Auteurs

Ferdinando Barretta (F)

Department of Molecular Medicine and Medical Biotechnology, University of Naples Federico II, 80131 Naples, Italy.
CEINGE Advanced Biotechnologies Franco Salvatore, 80131 Naples, Italy.

Fabiana Uomo (F)

Department of Molecular Medicine and Medical Biotechnology, University of Naples Federico II, 80131 Naples, Italy.

Simona Fecarotta (S)

Metabolic Diseases Unit, Department of Translational Medical Science, Section of Pediatrics, University of Naples Federico II, 80131 Naples, Italy.

Lucia Albano (L)

CEINGE Advanced Biotechnologies Franco Salvatore, 80131 Naples, Italy.

Daniela Crisci (D)

CEINGE Advanced Biotechnologies Franco Salvatore, 80131 Naples, Italy.

Alessandra Verde (A)

Metabolic Diseases Unit, Department of Translational Medical Science, Section of Pediatrics, University of Naples Federico II, 80131 Naples, Italy.

Maria Grazia Fisco (MG)

CEINGE Advanced Biotechnologies Franco Salvatore, 80131 Naples, Italy.

Giovanna Gallo (G)

CEINGE Advanced Biotechnologies Franco Salvatore, 80131 Naples, Italy.

Daniela Dottore Stagna (D)

Department of Molecular Medicine and Medical Biotechnology, University of Naples Federico II, 80131 Naples, Italy.

Maria Rosaria Pricolo (MR)

Centro Nacional de Investigaciones Cardiovasculares (CNIC), 28029 Madrid, Spain.

Marianna Alagia (M)

Metabolic Diseases Unit, Department of Translational Medical Science, Section of Pediatrics, University of Naples Federico II, 80131 Naples, Italy.

Gaetano Terrone (G)

Metabolic Diseases Unit, Department of Translational Medical Science, Section of Pediatrics, University of Naples Federico II, 80131 Naples, Italy.

Alessandro Rossi (A)

Metabolic Diseases Unit, Department of Translational Medical Science, Section of Pediatrics, University of Naples Federico II, 80131 Naples, Italy.

Giancarlo Parenti (G)

Metabolic Diseases Unit, Department of Translational Medical Science, Section of Pediatrics, University of Naples Federico II, 80131 Naples, Italy.

Margherita Ruoppolo (M)

Department of Molecular Medicine and Medical Biotechnology, University of Naples Federico II, 80131 Naples, Italy.
CEINGE Advanced Biotechnologies Franco Salvatore, 80131 Naples, Italy.

Cristina Mazzaccara (C)

Department of Molecular Medicine and Medical Biotechnology, University of Naples Federico II, 80131 Naples, Italy.
CEINGE Advanced Biotechnologies Franco Salvatore, 80131 Naples, Italy.

Giulia Frisso (G)

Department of Molecular Medicine and Medical Biotechnology, University of Naples Federico II, 80131 Naples, Italy.
CEINGE Advanced Biotechnologies Franco Salvatore, 80131 Naples, Italy.

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