Limited concordance of heparin/platelet factor 4 antibody assays for the diagnosis of heparin-induced thrombocytopenia: an analysis of the TORADI-HIT study.

contributing factors correlation diagnostic odds ratio heparin-induced platelet activation assay heparin-induced thrombocytopenia heparin/PF4 antibody assays

Journal

Journal of thrombosis and haemostasis : JTH
ISSN: 1538-7836
Titre abrégé: J Thromb Haemost
Pays: England
ID NLM: 101170508

Informations de publication

Date de publication:
09 2023
Historique:
received: 28 01 2023
revised: 26 04 2023
accepted: 17 05 2023
medline: 21 8 2023
pubmed: 30 5 2023
entrez: 29 5 2023
Statut: ppublish

Résumé

Anecdotal reports suggest that the correlation between heparin/platelet factor 4 (PF4) antibody assays for the diagnosis of heparin-induced thrombocytopenia (HIT) is limited. To investigate the correlation between widely used assays and examine possible factors contributing to variability. This is a large, prospective cohort study with 10 participating tertiary care hospitals including 1393 patients with suspected HIT in clinical practice. HIT was defined by a positive heparin-induced platelet activation (HIPA) assay (washed platelet reference standard test). Three different immunoassays were used to measure heparin/PF4 antibodies: chemiluminescent immunoassay, enzyme-linked immunosorbent assay, and particle gel immunoassay. Various factors that could influence the assays were examined: sex (male or female), age (<65 years or ≥65 years), unfractionated heparin exposure, presence of thrombosis, cardiovascular surgery, and intensive care unit. Spearman's correlation coefficients were calculated. Z-scores and diagnostic odds ratios were determined in the aforementioned subgroups of patients. Among 1393 patients, 119 were classified as HIT-positive (prevalence, 8.5%). The median 4Ts score was 5 (IQR, 4-6) in patients with HIT compared with 3 (IQR, 2-4) in patients without HIT. Correlations (r The correlation between widely used heparin/PF4 antibody assays was weak, and key factors could not explain this variability. Standardization of immunoassays is requested to improve comparability.

Sections du résumé

BACKGROUND
Anecdotal reports suggest that the correlation between heparin/platelet factor 4 (PF4) antibody assays for the diagnosis of heparin-induced thrombocytopenia (HIT) is limited.
OBJECTIVES
To investigate the correlation between widely used assays and examine possible factors contributing to variability.
METHODS
This is a large, prospective cohort study with 10 participating tertiary care hospitals including 1393 patients with suspected HIT in clinical practice. HIT was defined by a positive heparin-induced platelet activation (HIPA) assay (washed platelet reference standard test). Three different immunoassays were used to measure heparin/PF4 antibodies: chemiluminescent immunoassay, enzyme-linked immunosorbent assay, and particle gel immunoassay. Various factors that could influence the assays were examined: sex (male or female), age (<65 years or ≥65 years), unfractionated heparin exposure, presence of thrombosis, cardiovascular surgery, and intensive care unit. Spearman's correlation coefficients were calculated. Z-scores and diagnostic odds ratios were determined in the aforementioned subgroups of patients.
RESULTS
Among 1393 patients, 119 were classified as HIT-positive (prevalence, 8.5%). The median 4Ts score was 5 (IQR, 4-6) in patients with HIT compared with 3 (IQR, 2-4) in patients without HIT. Correlations (r
CONCLUSION
The correlation between widely used heparin/PF4 antibody assays was weak, and key factors could not explain this variability. Standardization of immunoassays is requested to improve comparability.

Identifiants

pubmed: 37247669
pii: S1538-7836(23)00430-0
doi: 10.1016/j.jtha.2023.05.016
pii:
doi:

Substances chimiques

Heparin 9005-49-6
Platelet Factor 4 37270-94-3
Antibodies 0
Anticoagulants 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2559-2568

Informations de copyright

Copyright © 2023 International Society on Thrombosis and Haemostasis. Published by Elsevier Inc. All rights reserved.

Auteurs

Angelika Hammerer-Lercher (A)

Department of Clinical Chemistry, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland; Institute of Laboratory Medicine, Kantonsspital Aarau, Aarau, Switzerland.

Henning Nilius (H)

Department of Clinical Chemistry, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland; Graduate School for Health Sciences, University of Bern, Bern, Switzerland.

Jan-Dirk Studt (JD)

Division of Medical Oncology and Hematology, University and University Hospital Zurich, Zurich, Switzerland.

Dimitrios A Tsakiris (DA)

Diagnostic Haematology, Basel University Hospital, Basel, Switzerland.

Andreas Greinacher (A)

Institut für Transfusionsmedizin, Universitätsmedizin Greifswald, Greifswald, Germany.

Adriana Mendez (A)

Institute of Laboratory Medicine, Kantonsspital Aarau, Aarau, Switzerland.

Adrian Schmidt (A)

Clinic of Medical Oncology and Hematology, Municipal Hospital Zurich Triemli, Zurich, Switzerland.

Walter A Wuillemin (WA)

Division of Hematology and Central Hematology Laboratory, Cantonal Hospital of Lucerne, University of Bern, Bern, Switzerland.

Bernhard Gerber (B)

Clinic of Hematology, Oncology Institute of Southern Switzerland, Bellinzona, Switzerland.

Johanna A Kremer Hovinga (JA)

Department of Hematology and Central Hematology Laboratory, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland.

Prakash Vishnu (P)

Division of Hematology, CHI Franciscan Medical Group, Seattle, USA.

Lukas Graf (L)

Cantonal Hospital of St. Gallen, St. Gallen, Switzerland.

Tamam Bakchoul (T)

Centre for Clinical Transfusion Medicine, University Hospital of Tübingen, Tübingen, Germany.

Michael Nagler (M)

Department of Clinical Chemistry, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland. Electronic address: michael.nagler@insel.ch.

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Classifications MeSH