Disparities among Black and White patients in plaque burden and composition and long-term impact.


Journal

Cardiovascular revascularization medicine : including molecular interventions
ISSN: 1878-0938
Titre abrégé: Cardiovasc Revasc Med
Pays: United States
ID NLM: 101238551

Informations de publication

Date de publication:
10 2023
Historique:
received: 27 04 2023
accepted: 27 04 2023
medline: 6 11 2023
pubmed: 5 6 2023
entrez: 4 6 2023
Statut: ppublish

Résumé

Black patients presenting to the catheterization laboratory have more risk factors and worse long-term outcomes. This sub-analysis of the Lipid Rich Plaque (LRP) study quantifies the plaque burden and composition of Black vs White patients and associated outcomes. Patients with a singular, self-reported race presenting for cardiac catheterization were enrolled if near-infrared spectroscopy/intravascular ultrasound (NIRS-IVUS) imaging of non-stented, non-culprit (NC) vessels was performed. Lipidic content was quantified at the 4-mm region with maximum Lipid Core Burden Index (maxLCBI Among 1346 patients with a singular, self-reported race, 182 were Black. Black vs White patients were more likely to be female, had higher rates of traditional risk factors, and were more likely to present acutely. Both patients and segments were more likely to have maxLCBI Compared to White patients, Black patients had more lipid-rich plaques with greater plaque burden. Both high lipidic burden and Black race were independently predictive of NC-MACE within 2 years. https://clinicaltrials.gov/ct2/show/NCT02033694, NCT02033694.

Sections du résumé

BACKGROUND
Black patients presenting to the catheterization laboratory have more risk factors and worse long-term outcomes. This sub-analysis of the Lipid Rich Plaque (LRP) study quantifies the plaque burden and composition of Black vs White patients and associated outcomes.
METHODS
Patients with a singular, self-reported race presenting for cardiac catheterization were enrolled if near-infrared spectroscopy/intravascular ultrasound (NIRS-IVUS) imaging of non-stented, non-culprit (NC) vessels was performed. Lipidic content was quantified at the 4-mm region with maximum Lipid Core Burden Index (maxLCBI
RESULTS
Among 1346 patients with a singular, self-reported race, 182 were Black. Black vs White patients were more likely to be female, had higher rates of traditional risk factors, and were more likely to present acutely. Both patients and segments were more likely to have maxLCBI
CONCLUSIONS
Compared to White patients, Black patients had more lipid-rich plaques with greater plaque burden. Both high lipidic burden and Black race were independently predictive of NC-MACE within 2 years.
CLINICAL TRIAL REGISTRATION
https://clinicaltrials.gov/ct2/show/NCT02033694, NCT02033694.

Identifiants

pubmed: 37271594
pii: S1553-8389(23)00165-3
doi: 10.1016/j.carrev.2023.04.023
pii:
doi:

Substances chimiques

Lipids 0

Banques de données

ClinicalTrials.gov
['NCT02033694']

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

28-32

Informations de copyright

Copyright © 2023 Elsevier Inc. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of competing interest The authors declare the following financial interests/personal relationships which may be considered as potential competing interests: Carlo Di Mario is the recipient of research grants (through the Department of Clinical & Experimental medicine of the University of Florence) from AMGEN, Behring, Chiesi, Daiichi-Sankyo, Edwards, Medtronic, and Shockwave. Ziad Ali reports grants from NIH/NHLBI, Abbott Vascular and Cardiovascular Systems Inc., personal fees from Amgen, AstraZeneca and Boston Scientific, and equity from Shockwave Medical. Hector Garcia-Garcia reports the following institutional grant support: Biotronik, Boston Scientific, Medtronic, Abbott, Neovasc, Shockwave, Philips, and CorFlow. Gary Mintz reports honoraria from Boston Scientific and Philips. Ron Waksman reports serving on the advisory boards of Abbott Vascular, Boston Scientific, Medtronic, Philips IGT, and Pi-Cardia Ltd.; being a consultant for Abbott Vascular, Biotronik, Boston Scientific, Cordis, Medtronic, Philips IGT, Pi-Cardia Ltd., Swiss Interventional Systems/SIS Medical AG, Transmural Systems Inc., and Venous MedTech; receiving institutional grant support from Amgen, Biotronik, Boston Scientific, Chiesi, Medtronic, and Philips IGT; and being an investor in MedAlliance and Transmural Systems Inc. All other authors report no conflicts of interest.

Auteurs

Rebecca Torguson (R)

The Zena and Michael A. Wiener Cardiovascular Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.

Gary S Mintz (GS)

Section of Interventional Cardiology, MedStar Washington Hospital Center, Washington, DC, USA.

Carlo Di Mario (C)

Structural Interventional Cardiology, Department of Clinical & Experimental Medicine, Careggi University Hospital, Florence, Italy.

Tim Ten Cate (T)

Department of Cardiology, Radboud University Medical Center, Nijmegen, Netherlands.

Ziad A Ali (ZA)

DeMatteis Cardiovascular Institute, St. Francis Hospital & Heart Center, Roslyn, NY, USA; Clinical Trials Center, Cardiovascular Research Foundation, New York, NY, USA.

Varinder Singh (V)

Department of Cardiology, Northwell Health, New York, NY, USA.

William Skinner (W)

Department of Cardiology, Central Baptist Hospital, Lexington, KY, USA.

Cheng Zhang (C)

Section of Interventional Cardiology, MedStar Washington Hospital Center, Washington, DC, USA.

Corey Shea (C)

Section of Interventional Cardiology, MedStar Washington Hospital Center, Washington, DC, USA.

Kayode O Kuku (KO)

Section of Interventional Cardiology, MedStar Washington Hospital Center, Washington, DC, USA.

Hector M Garcia-Garcia (HM)

Section of Interventional Cardiology, MedStar Washington Hospital Center, Washington, DC, USA.

Ron Waksman (R)

Section of Interventional Cardiology, MedStar Washington Hospital Center, Washington, DC, USA. Electronic address: ron.waksman@medstar.net.

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