Steroid responsive idiopathic calcitriol induced hypercalcemia: a case report and review of the literature.


Journal

BMC nephrology
ISSN: 1471-2369
Titre abrégé: BMC Nephrol
Pays: England
ID NLM: 100967793

Informations de publication

Date de publication:
06 06 2023
Historique:
received: 15 02 2023
accepted: 17 05 2023
medline: 8 6 2023
pubmed: 7 6 2023
entrez: 6 6 2023
Statut: epublish

Résumé

Idiopathic Calcitriol Induced Hypercalcemia is a rare cause of a common condition of hypercalcemia. Hypercalcemia is most commonly the result of hyperparathyroidism and together with hypercalcemia of malignancy accounts for over 95% of cases. Idiopathic Calcitriol Induced Hypercalcemia can mimic hypercalcemia secondary to granulomatous diseases like sarcoidosis, but with apparent absences of both imaging and physical exam findings consistent with the disease. We report here a 51-year-old man who presented with recurrent nephrolithiasis, hypercalcemia, and acute kidney injury. A 51-year-old man presented with severe back pain and mild hematuria. He had a history of recurrent nephrolithiasis over the course of a 15-year period. On presentation his calcium was elevated at 13.4 mg/dL, creatinine was 3.1 mg/dL (from baseline of 1.2), and his PTH was reduced at 5 pg/mL. CT abdomen and pelvis showed acute nephrolithiasis which was managed medically. Work up for the hypercalcemia included an SPEP which was normal, Vit D,1,25 (OH)2 was elevated at 80.4 pg/mL, CT chest showed no evidence of sarcoidosis. Management with 10 mg prednisone showed marked improvement in the hypercalcemia and he no longer had any symptoms of hypercalcemia. Idiopathic Calcitriol Induced Hypercalcemia is a rare cause of hypercalcemia. All reported cases benefit from more intensive long-term immunosuppression. This report helps consolidate the diagnosis of Idiopathic Calcitriol Induced Hypercalcemia and encourages researchers to better investigate its underlying pathogenesis.

Sections du résumé

BACKGROUND
Idiopathic Calcitriol Induced Hypercalcemia is a rare cause of a common condition of hypercalcemia. Hypercalcemia is most commonly the result of hyperparathyroidism and together with hypercalcemia of malignancy accounts for over 95% of cases. Idiopathic Calcitriol Induced Hypercalcemia can mimic hypercalcemia secondary to granulomatous diseases like sarcoidosis, but with apparent absences of both imaging and physical exam findings consistent with the disease. We report here a 51-year-old man who presented with recurrent nephrolithiasis, hypercalcemia, and acute kidney injury.
CASE PRESENTATION
A 51-year-old man presented with severe back pain and mild hematuria. He had a history of recurrent nephrolithiasis over the course of a 15-year period. On presentation his calcium was elevated at 13.4 mg/dL, creatinine was 3.1 mg/dL (from baseline of 1.2), and his PTH was reduced at 5 pg/mL. CT abdomen and pelvis showed acute nephrolithiasis which was managed medically. Work up for the hypercalcemia included an SPEP which was normal, Vit D,1,25 (OH)2 was elevated at 80.4 pg/mL, CT chest showed no evidence of sarcoidosis. Management with 10 mg prednisone showed marked improvement in the hypercalcemia and he no longer had any symptoms of hypercalcemia.
CONCLUSION
Idiopathic Calcitriol Induced Hypercalcemia is a rare cause of hypercalcemia. All reported cases benefit from more intensive long-term immunosuppression. This report helps consolidate the diagnosis of Idiopathic Calcitriol Induced Hypercalcemia and encourages researchers to better investigate its underlying pathogenesis.

Identifiants

pubmed: 37280532
doi: 10.1186/s12882-023-03203-4
pii: 10.1186/s12882-023-03203-4
pmc: PMC10245421
doi:

Substances chimiques

Calcitriol FXC9231JVH
Vitamin D 1406-16-2

Types de publication

Review Case Reports Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

159

Informations de copyright

© 2023. The Author(s).

Références

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Eur J Intern Med. 2008 Dec;19(8):e105-6
pubmed: 19046703
Allergy Asthma Clin Immunol. 2013 Aug 15;9(1):30
pubmed: 23947590
N Engl J Med. 2007 Nov 22;357(21):2153-65
pubmed: 18032765
Pediatr Nephrol. 2006 Feb;21(2):281-5
pubmed: 16362392
Curr Opin Pulm Med. 2000 Sep;6(5):442-7
pubmed: 10958237
N Engl J Med. 1994 Jul 7;331(1):42-5
pubmed: 8202102

Auteurs

Omar El Fadel (OE)

Sidney Kimmel Medical College, Thomas Jefferson University, Philadelphia, United States.

Anshel Kenkare (A)

Sidney Kimmel Medical College, Thomas Jefferson University, Philadelphia, United States.

Jingjing Zhang (J)

Department of Internal Medicine, Division of Nephrology, Thomas Jefferson University, Philadelphia, United States. jingjing.zhang@jefferson.edu.

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Classifications MeSH