The association between CYB5A gene rs1790834 polymorphism and clinical improvement after 6 months of leflunomide treatment in women with rheumatoid arthritis.


Journal

Clinical rheumatology
ISSN: 1434-9949
Titre abrégé: Clin Rheumatol
Pays: Germany
ID NLM: 8211469

Informations de publication

Date de publication:
Sep 2023
Historique:
received: 11 04 2023
accepted: 26 05 2023
revised: 15 05 2023
medline: 10 8 2023
pubmed: 8 6 2023
entrez: 8 6 2023
Statut: ppublish

Résumé

Rheumatoid arthritis (RA) is a disease affecting around 1% of the population in developed countries and can be treated with leflunomide. The higher prevalence of RA among women and numerous previous studies suggested the crucial role of sex hormones. Cytochrome CYB5A regulates the synthesis of androgens. Therefore, the aim of this study was to determine the association between common CYB5A gene polymorphism and the response to leflunomide in women with RA. This study included 111 patients. All of them received oral leflunomide monotherapy at a dose of 20 mg daily. Women were genotyped for the presence of CYB5A rs1790834 polymorphism and evaluated monthly for 6 months following the initiation of treatment. After 6 months of therapy, patients with the GG genotype had higher DAS28 values and less improvement in DAS28 compared to patients with the GA and AA genotypes (p = 0.04). No statistically significant differences were found in relation to other disease activity parameters. The results of the current study suggest a possible association of the CYB5A rs1790834 polymorphism with some disease activity parameters in RA patients treated with leflunomide during the initial therapy period. However, confirmation of the effect of this polymorphism on the efficacy of leflunomide treatment requires further studies. Key Points • Leflunomide is the synthetic disease-modifying anti-rheumatic drug used in the therapy of rheumatoid arthritis. • CYB5A rs1790834 gene polymorphism may influence the clinical improvement after 6 months of leflunomide treatment in women with rheumatoid arthritis.

Identifiants

pubmed: 37289314
doi: 10.1007/s10067-023-06653-1
pii: 10.1007/s10067-023-06653-1
doi:

Substances chimiques

Leflunomide G162GK9U4W
Isoxazoles 0
Antirheumatic Agents 0
CYB5A protein, human 0
Cytochromes b5 9035-39-6

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

2477-2483

Informations de copyright

© 2023. The Author(s), under exclusive licence to International League of Associations for Rheumatology (ILAR).

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Auteurs

Filip Machaj (F)

Department of Physiology, Pomeranian Medical University, 70-111, Szczecin, Poland.
Department of Medical Biology, Medical University of Warsaw, 00-575, Warsaw, Poland.

Jakub Rosik (J)

Department of Physiology, Pomeranian Medical University, 70-111, Szczecin, Poland.

Bartosz Szostak (B)

Department of Physiology, Pomeranian Medical University, 70-111, Szczecin, Poland.

Damian Malinowski (D)

Department of Pharmacokinetics and Therapeutic Drug Monitoring, Pomeranian Medical University, 70-111, Szczecin, Poland.

Krzysztof Safranow (K)

Department of Biochemistry and Medical Chemistry, Pomeranian Medical University, 70-111, Szczecin, Poland.

Gabriela Olędzka (G)

Department of Medical Biology, Medical University of Warsaw, 00-575, Warsaw, Poland.

Emilia Wiechec (E)

Division of Cell Biology, Department of Biomedical and Clinical Sciences, Linköping University, 581 85, Linköping, Sweden.
Faculty of Medicine, Academy of Silesia, 40-555, Katowice, Poland.

Andrzej Pawlik (A)

Department of Physiology, Pomeranian Medical University, 70-111, Szczecin, Poland. pawand@poczta.onet.pl.

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