Clinical effects of Emblica officinalis fruit consumption on cardiovascular disease risk factors: a systematic review and meta-analysis.

Amla Atherosclerosis Cardiovascular disease Cholesterol Chronic disease Emblica officinalis Inflammation Lipids Meta-analysis Phyllanthus emblica

Journal

BMC complementary medicine and therapies
ISSN: 2662-7671
Titre abrégé: BMC Complement Med Ther
Pays: England
ID NLM: 101761232

Informations de publication

Date de publication:
09 Jun 2023
Historique:
received: 09 09 2021
accepted: 13 05 2023
medline: 12 6 2023
pubmed: 10 6 2023
entrez: 9 6 2023
Statut: epublish

Résumé

Emblica officinalis (EO) fruit consumption has been found to have a beneficial effect on cardiovascular disease (CVD) physiological risk factors in preliminary clinical intervention trials; however, questions remain regarding the overall effectiveness of EO on CVD risk. The purpose of this systematic review and meta-analysis is to: 1) systematically describe the clinical research examining EO; and 2) quantitatively assess the effects of EO on CVD physiological risk factors. The Pubmed, Embase, Web of Science, and Google Scholar electronic platforms were searched for relevant randomized controlled trials (RCTs) published until April 7, 2021. Studies were included if they involved adults (age ≥ 18 years) ingesting a form of EO fruit; included blood lipids, blood pressure, and/or inflammatory biomarkers as outcomes; had clearly defined intervention and control treatments with pre- and post-intervention data; were peer-reviewed; and were written in English. Studies were excluded if they compared EO with another risk reduction intervention without a usual care control group. RCTs were assessed for methodological quality using the Cochrane risk-of-bias version 2 (ROB2) tool, qualitatively described, and quantitatively evaluated using random and fixed effect meta-analysis models. A total of nine RCTs (n = 535 participants) were included for review. Included studies followed parallel-group (n = 6) and crossover (n = 3) designs, with EO dosage ranging from 500 mg/day to 1500 mg/day, and treatment duration ranging from 14 to 84 days. Meta-analyses revealed EO to have a significant composite effect at lowering low-density lipoprotein cholesterol (LDL-C; Mean difference (MD) = -15.08 mg/dL [95% Confidence interval (CI) = -25.43 to -4.73], I Due to statistical and clinical heterogeneity in the limited number of clinical trials to date, the promising effects of EO on physiologic CVD risk factors in this review should be interpreted with caution. Further research is needed to determine if EO offers an efficacious option for primary or secondary prevention of CVD as either monotherapy or adjunct to evidence-based dietary patterns and/or standard pharmacotherapy.

Sections du résumé

BACKGROUND BACKGROUND
Emblica officinalis (EO) fruit consumption has been found to have a beneficial effect on cardiovascular disease (CVD) physiological risk factors in preliminary clinical intervention trials; however, questions remain regarding the overall effectiveness of EO on CVD risk. The purpose of this systematic review and meta-analysis is to: 1) systematically describe the clinical research examining EO; and 2) quantitatively assess the effects of EO on CVD physiological risk factors.
METHODS METHODS
The Pubmed, Embase, Web of Science, and Google Scholar electronic platforms were searched for relevant randomized controlled trials (RCTs) published until April 7, 2021. Studies were included if they involved adults (age ≥ 18 years) ingesting a form of EO fruit; included blood lipids, blood pressure, and/or inflammatory biomarkers as outcomes; had clearly defined intervention and control treatments with pre- and post-intervention data; were peer-reviewed; and were written in English. Studies were excluded if they compared EO with another risk reduction intervention without a usual care control group. RCTs were assessed for methodological quality using the Cochrane risk-of-bias version 2 (ROB2) tool, qualitatively described, and quantitatively evaluated using random and fixed effect meta-analysis models.
RESULTS RESULTS
A total of nine RCTs (n = 535 participants) were included for review. Included studies followed parallel-group (n = 6) and crossover (n = 3) designs, with EO dosage ranging from 500 mg/day to 1500 mg/day, and treatment duration ranging from 14 to 84 days. Meta-analyses revealed EO to have a significant composite effect at lowering low-density lipoprotein cholesterol (LDL-C; Mean difference (MD) = -15.08 mg/dL [95% Confidence interval (CI) = -25.43 to -4.73], I
CONCLUSIONS CONCLUSIONS
Due to statistical and clinical heterogeneity in the limited number of clinical trials to date, the promising effects of EO on physiologic CVD risk factors in this review should be interpreted with caution. Further research is needed to determine if EO offers an efficacious option for primary or secondary prevention of CVD as either monotherapy or adjunct to evidence-based dietary patterns and/or standard pharmacotherapy.

Identifiants

pubmed: 37296402
doi: 10.1186/s12906-023-03997-8
pii: 10.1186/s12906-023-03997-8
pmc: PMC10251691
doi:

Substances chimiques

Cholesterol 97C5T2UQ7J
Cholesterol, LDL 0

Types de publication

Meta-Analysis Systematic Review Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

190

Informations de copyright

© 2023. The Author(s).

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Auteurs

Paul D S Brown (PDS)

Southern Medical Program, Faculty of Medicine, University of British Columbia Okanagan Campus, 1088 Discovery Avenue, Kelowna, BC, V1V 1V7, Canada.

Nicole Ketter (N)

Department of Biology, Irving K. Barber Faculty of Science, University of British Columbia Okanagan Campus, 3187 University Way, ASC 413, Kelowna, BC, V1V 1V7, Canada.
Centre for Chronic Disease Prevention and Management, University of British Columbia Okanagan Campus, 1088 Discovery Avenue, Kelowna, BC, V1V 1V7, Canada.

Mathew Vis-Dunbar (M)

Southern Medical Program, Faculty of Medicine, University of British Columbia Okanagan Campus, 1088 Discovery Avenue, Kelowna, BC, V1V 1V7, Canada.

Brodie M Sakakibara (BM)

Southern Medical Program, Faculty of Medicine, University of British Columbia Okanagan Campus, 1088 Discovery Avenue, Kelowna, BC, V1V 1V7, Canada. brodie.sakakibara@ubc.ca.
Centre for Chronic Disease Prevention and Management, University of British Columbia Okanagan Campus, 1088 Discovery Avenue, Kelowna, BC, V1V 1V7, Canada. brodie.sakakibara@ubc.ca.
Department of Occupational Science and Occupational Therapy, University of British Columbia, T325 - 2211 , Wesbrook Mall, Vancouver, BC, V6T 2B5, Canada. brodie.sakakibara@ubc.ca.

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