Mechanistic basis for potent neutralization of Sin Nombre hantavirus by a human monoclonal antibody.


Journal

Nature microbiology
ISSN: 2058-5276
Titre abrégé: Nat Microbiol
Pays: England
ID NLM: 101674869

Informations de publication

Date de publication:
07 2023
Historique:
received: 22 07 2022
accepted: 17 05 2023
medline: 7 7 2023
pubmed: 16 6 2023
entrez: 15 6 2023
Statut: ppublish

Résumé

Rodent-borne hantaviruses are prevalent worldwide and upon spillover to human populations, cause severe disease for which no specific treatment is available. A potent antibody response is key for recovery from hantavirus infection. Here we study a highly neutralizing human monoclonal antibody, termed SNV-42, which was derived from a memory B cell isolated from an individual with previous Sin Nombre virus (SNV) infection. Crystallographic analysis demonstrates that SNV-42 targets the Gn subcomponent of the tetrameric (Gn-Gc)

Identifiants

pubmed: 37322112
doi: 10.1038/s41564-023-01413-y
pii: 10.1038/s41564-023-01413-y
pmc: PMC10322703
doi:

Substances chimiques

Antibodies, Monoclonal 0
Antibodies, Neutralizing 0
Glycoproteins 0

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1293-1303

Subventions

Organisme : NIGMS NIH HHS
ID : T32 GM008320
Pays : United States
Organisme : Medical Research Council
ID : MR/S007555/1
Pays : United Kingdom
Organisme : Medical Research Council
ID : MR/V031635/1
Pays : United Kingdom
Organisme : Wellcome Trust
Pays : United Kingdom
Organisme : Wellcome Trust
ID : 203141/Z/16/Z
Pays : United Kingdom

Informations de copyright

© 2023. The Author(s).

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Auteurs

Robert Stass (R)

Division of Structural Biology, Wellcome Centre for Human Genetics, University of Oxford, Oxford, UK.

Taylor B Engdahl (TB)

Department of Pathology, Microbiology and Immunology, Vanderbilt University Medical Center, Nashville, TN, USA.

Nathaniel S Chapman (NS)

Department of Pathology, Microbiology and Immunology, Vanderbilt University Medical Center, Nashville, TN, USA.

Rachael M Wolters (RM)

Department of Pathology, Microbiology and Immunology, Vanderbilt University Medical Center, Nashville, TN, USA.

Laura S Handal (LS)

Vanderbilt Vaccine Center, Vanderbilt University Medical Center, Nashville, TN, USA.

Summer M Diaz (SM)

Vanderbilt Vaccine Center, Vanderbilt University Medical Center, Nashville, TN, USA.

James E Crowe (JE)

Department of Pathology, Microbiology and Immunology, Vanderbilt University Medical Center, Nashville, TN, USA. james.crowe@vumc.org.
Vanderbilt Vaccine Center, Vanderbilt University Medical Center, Nashville, TN, USA. james.crowe@vumc.org.
Department of Pediatrics, Vanderbilt University Medical Center, Nashville, TN, USA. james.crowe@vumc.org.

Thomas A Bowden (TA)

Division of Structural Biology, Wellcome Centre for Human Genetics, University of Oxford, Oxford, UK. thomas.bowden@strubi.ox.ac.uk.

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