Brexpiprazole suppresses cell proliferation and de novo lipogenesis through AMPK/SREBP1 pathway in colorectal cancer.


Journal

Environmental toxicology
ISSN: 1522-7278
Titre abrégé: Environ Toxicol
Pays: United States
ID NLM: 100885357

Informations de publication

Date de publication:
Oct 2023
Historique:
revised: 18 05 2023
received: 29 01 2023
accepted: 11 06 2023
medline: 19 9 2023
pubmed: 22 6 2023
entrez: 22 6 2023
Statut: ppublish

Résumé

In the present study, we investigated the role of brexpiprazole on cell proliferation and lipogenesis in colorectal cancer (CRC) and its molecular mechanism. The effect of brexpiprazole on CRC cell proliferation was determined by CCK-8, EdU assay, cell clone formation. The flow cytometry was evaluated cell cycle. Differential expression genes (DEGs) were identified by RNA-seq assay after treating HCT116 cells with or without 20 μM brexpiprazole for 24 h. Then, the top 120 DEGs were analyzed by GO and KEGG enrichment analysis. After that, Oil red O staining and the levels of total cholestenone and triglyceride were measured to assess lipogenesis capacity in CRC cells. The related molecules of cell proliferation, lipogenic and AMPK/SREBP1 signal pathways were measured by q-PCR, western blot and immunohistochemical staining. Brexpiprazole remarkably suppressed cell proliferation, lipogenesis, and induced cell cycle arrest in CRC. The underlying mechanisms probably involved the suppression of SREBP1 and the stimulation of AMPK. Brexpiprazole inhibited cell proliferation and de novo lipogenesis through AMPK/SREBP1 pathway in CRC.

Identifiants

pubmed: 37347510
doi: 10.1002/tox.23871
doi:

Substances chimiques

AMP-Activated Protein Kinases EC 2.7.11.31
brexpiprazole 2J3YBM1K8C

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

2352-2360

Subventions

Organisme : Provincial key cultivation project of Science and Technology Development Plan of North Sichuan Medical College
ID : CYB22-ZDA11
Organisme : Talent Project of First Affiliated Hospital of Kunming Medical University
ID : 2022535Q02
Organisme : Strategic Cooperation Research Project of Nanchong
ID : 22SXQT0332

Informations de copyright

© 2023 Wiley Periodicals LLC.

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Auteurs

Ting Li (T)

Institute of Basic Medical and Forensic Medicine, North Sichuan Medical College, Nanchong, China.

Xiaojie Liu (X)

Institute of Basic Medical and Forensic Medicine, North Sichuan Medical College, Nanchong, China.

Xiaoyi Long (X)

Institute of Basic Medical and Forensic Medicine, North Sichuan Medical College, Nanchong, China.

Yangyou Li (Y)

Animal Experimental Center, North Sichuan Medical College, Nanchong, China.

Jin Xiang (J)

School of Clinical Medicine, North Sichuan Medical College, Nanchong, China.

Yuanxia Lv (Y)

School of Pharmacy, North Sichuan Medical College, Nanchong, China.

Xiaoyang Zhao (X)

Institute of Basic Medical and Forensic Medicine, North Sichuan Medical College, Nanchong, China.

Shaoqing Shi (S)

Scientific Research Laboratory Center, First Affiliated Hospital of Kunming Medical University, Kunming, China.

Wei Chen (W)

Institute of Basic Medical and Forensic Medicine, North Sichuan Medical College, Nanchong, China.

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