Microtubule binding of the human augmin complex is directly controlled by importins and Ran-GTP.


Journal

Journal of cell science
ISSN: 1477-9137
Titre abrégé: J Cell Sci
Pays: England
ID NLM: 0052457

Informations de publication

Date de publication:
15 06 2023
Historique:
received: 19 02 2023
accepted: 18 05 2023
medline: 27 6 2023
pubmed: 26 6 2023
entrez: 26 6 2023
Statut: ppublish

Résumé

Mitotic spindle assembly during cell division is a highly regulated process. Ran-GTP produced around chromosomes controls the activity of a multitude of spindle assembly factors by releasing them from inhibitory interaction with importins. A major consequence of Ran-GTP regulation is the local stimulation of branched microtubule nucleation around chromosomes, which is mediated by the augmin complex (composed of the eight subunits HAUS1-HAUS8), a process that is crucially important for correct spindle assembly. However, augmin is not known to be a direct target of the Ran-GTP pathway, raising the question of how its activity is controlled. Here, we present the in vitro reconstitution of Ran-GTP-regulated microtubule binding of the human augmin complex. We demonstrate that importins directly bind to augmin, which prevents augmin from binding to microtubules. Ran-GTP relieves this inhibition. Therefore, the augmin complex is a direct target of the Ran-GTP pathway, suggesting that branching microtubule nucleation is directly regulated by the Ran-GTP gradient around chromosomes in dividing cells.

Identifiants

pubmed: 37357828
pii: 319485
doi: 10.1242/jcs.261096
pmc: PMC10323247
pii:
doi:

Substances chimiques

Karyopherins 0
Guanosine Triphosphate 86-01-1
Microtubule-Associated Proteins 0
ran GTP-Binding Protein EC 3.6.5.2
augmin protein, human 0
Cell Cycle Proteins 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : NIGMS NIH HHS
ID : P41 GM103311
Pays : United States

Informations de copyright

© 2023. Published by The Company of Biologists Ltd.

Déclaration de conflit d'intérêts

Competing interests The authors declare no competing or financial interests.

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Auteurs

Kseniya Ustinova (K)

Centre for Genomic Regulation (CRG), Department of Quantitative Cell Biology, The Barcelona Institute of Science and Technology (BIST), Carrer del Dr. Aiguader 88, 08003 Barcelona, Spain.

Felix Ruhnow (F)

Centre for Genomic Regulation (CRG), Department of Quantitative Cell Biology, The Barcelona Institute of Science and Technology (BIST), Carrer del Dr. Aiguader 88, 08003 Barcelona, Spain.

Maria Gili (M)

Centre for Genomic Regulation (CRG), Department of Quantitative Cell Biology, The Barcelona Institute of Science and Technology (BIST), Carrer del Dr. Aiguader 88, 08003 Barcelona, Spain.

Thomas Surrey (T)

Centre for Genomic Regulation (CRG), Department of Quantitative Cell Biology, The Barcelona Institute of Science and Technology (BIST), Carrer del Dr. Aiguader 88, 08003 Barcelona, Spain.
Universitat Pompeu Fabra (UPF), Barcelona, Spain.
Catalan Institution for Research and Advanced Studies (ICREA), Passeig Lluis Companys 23, Barcelona 08010, Spain.

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Classifications MeSH