Comparable Response Following Exposure to Biodiesel and Diesel Exhaust Particles in Advanced Multicellular Human Lung Models.
COX-2
DNA damage
MQ-ALI
PBEC-ALI
PGE2
biodiesel
lung
oxidative stress
particles
petro-diesel
phagocytosis
Journal
Toxics
ISSN: 2305-6304
Titre abrégé: Toxics
Pays: Switzerland
ID NLM: 101639637
Informations de publication
Date de publication:
14 Jun 2023
14 Jun 2023
Historique:
received:
12
03
2023
revised:
31
05
2023
accepted:
06
06
2023
medline:
27
6
2023
pubmed:
27
6
2023
entrez:
27
6
2023
Statut:
epublish
Résumé
Biodiesel is considered to be a sustainable alternative for fossil fuels such as petroleum-based diesel. However, we still lack knowledge about the impact of biodiesel emissions on humans, as airways and lungs are the primary target organs of inhaled toxicants. This study investigated the effect of exhaust particles from well-characterized rapeseed methyl ester (RME) biodiesel exhaust particles (BDEP) and petro-diesel exhaust particles (DEP) on primary bronchial epithelial cells (PBEC) and macrophages (MQ). The advanced multicellular physiologically relevant bronchial mucosa models were developed using human primary bronchial epithelial cells (PBEC) cultured at air-liquid interface (ALI) in the presence or absence of THP-1 cell-derived macrophages (MQ). The experimental set-up used for BDEP and DEP exposures (18 µg/cm
Identifiants
pubmed: 37368632
pii: toxics11060532
doi: 10.3390/toxics11060532
pmc: PMC10304096
pii:
doi:
Types de publication
Journal Article
Langues
eng
Subventions
Organisme : The Swedish Research Council
ID : (LP: 2018-03233)
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