Mechanisms of Atrial Fibrillation: How Our Knowledge Affects Clinical Practice.
atrial fibrillation
fibrosis
inflammation oxidative stress
triggers
Journal
Life (Basel, Switzerland)
ISSN: 2075-1729
Titre abrégé: Life (Basel)
Pays: Switzerland
ID NLM: 101580444
Informations de publication
Date de publication:
25 May 2023
25 May 2023
Historique:
received:
15
04
2023
revised:
17
05
2023
accepted:
19
05
2023
medline:
28
6
2023
pubmed:
28
6
2023
entrez:
28
6
2023
Statut:
epublish
Résumé
Atrial fibrillation (AF) is a very common arrhythmia that mainly affects older individuals. The mechanism of atrial fibrillation is complex and is related to the pathogenesis of trigger activation and the perpetuation of arrhythmia. The pulmonary veins in the left atrium arei confirm that onfirm the most common triggers due to their distinct anatomical and electrophysiological properties. As a result, their electrical isolation by ablation is the cornerstone of invasive AF treatment. Multiple factors and comorbidities affect the atrial tissue and lead to myocardial stretch. Several neurohormonal and structural changes occur, leading to inflammation and oxidative stress and, consequently, a fibrotic substrate created by myofibroblasts, which encourages AF perpetuation. Several mechanisms are implemented into daily clinical practice in both interventions in and the medical treatment of atrial fibrillation.
Identifiants
pubmed: 37374043
pii: life13061260
doi: 10.3390/life13061260
pmc: PMC10303005
pii:
doi:
Types de publication
Journal Article
Review
Langues
eng
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