LGG-1/GABARAP lipidation is not required for autophagy and development in


Journal

eLife
ISSN: 2050-084X
Titre abrégé: Elife
Pays: England
ID NLM: 101579614

Informations de publication

Date de publication:
03 07 2023
Historique:
received: 22 12 2022
accepted: 30 06 2023
medline: 14 7 2023
pubmed: 3 7 2023
entrez: 3 7 2023
Statut: epublish

Résumé

The ubiquitin-like proteins Atg8/LC3/GABARAP are required for multiple steps of autophagy, such as initiation, cargo recognition and engulfment, vesicle closure and degradation. Most of LC3/GABARAP functions are considered dependent on their post-translational modifications and their association with the autophagosome membrane through a conjugation to a lipid, the phosphatidyl-ethanolamine. Contrarily to mammals,

Identifiants

pubmed: 37395461
doi: 10.7554/eLife.85748
pii: 85748
pmc: PMC10338037
doi:
pii:

Substances chimiques

Microtubule-Associated Proteins 0
GABARAP protein, human 0
Apoptosis Regulatory Proteins 0
LGG-2 protein, C elegans 0
Caenorhabditis elegans Proteins 0
LGG-1 protein, C elegans 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : European Research Council
ID : ERC-CoG-616499
Pays : International

Informations de copyright

© 2023, Leboutet et al.

Déclaration de conflit d'intérêts

RL, CL, LM, MP, GQ, MR, MC, TH, EC, CL, RL No competing interests declared

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Auteurs

Romane Leboutet (R)

Institute for Integrative Biology of the Cell (I2BC), Université Paris-Saclay, CEA, CNRS, Gif-sur-Yvette, France.
INSERM U1280, Gif-sur-Yvette, France.

Céline Largeau (C)

Institute for Integrative Biology of the Cell (I2BC), Université Paris-Saclay, CEA, CNRS, Gif-sur-Yvette, France.
INSERM U1280, Gif-sur-Yvette, France.

Leonie Müller (L)

Institute for Genetics and Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases (CECAD), University of Cologne, Cologne, Germany.

Magali Prigent (M)

Institute for Integrative Biology of the Cell (I2BC), Université Paris-Saclay, CEA, CNRS, Gif-sur-Yvette, France.
INSERM U1280, Gif-sur-Yvette, France.

Grégoire Quinet (G)

Laboratoire de Chimie de Coordination (LCC), CNRS, Toulouse, France.

Manuel S Rodriguez (MS)

Laboratoire de Chimie de Coordination (LCC), CNRS, Toulouse, France.

Marie-Hélène Cuif (MH)

Institute for Integrative Biology of the Cell (I2BC), Université Paris-Saclay, CEA, CNRS, Gif-sur-Yvette, France.
INSERM U1280, Gif-sur-Yvette, France.

Thorsten Hoppe (T)

Institute for Genetics and Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases (CECAD), University of Cologne, Cologne, Germany.
Center for Molecular Medicine Cologne (CMMC), Faculty of Medicine and University Hospital of Cologne, Cologne, Germany.

Emmanuel Culetto (E)

Institute for Integrative Biology of the Cell (I2BC), Université Paris-Saclay, CEA, CNRS, Gif-sur-Yvette, France.
INSERM U1280, Gif-sur-Yvette, France.

Christophe Lefebvre (C)

Institute for Integrative Biology of the Cell (I2BC), Université Paris-Saclay, CEA, CNRS, Gif-sur-Yvette, France.
INSERM U1280, Gif-sur-Yvette, France.

Renaud Legouis (R)

Institute for Integrative Biology of the Cell (I2BC), Université Paris-Saclay, CEA, CNRS, Gif-sur-Yvette, France.
INSERM U1280, Gif-sur-Yvette, France.

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