Polyvinyl Alcohol (PVA)-Based Nanoniosome for Enhanced in vitro Delivery and Anticancer Activity of Thymol.

apoptosis cancerous cell lines herbal anticancer drug niosome pH-dependent release polyvinyl alcohol thymol

Journal

International journal of nanomedicine
ISSN: 1178-2013
Titre abrégé: Int J Nanomedicine
Pays: New Zealand
ID NLM: 101263847

Informations de publication

Date de publication:
2023
Historique:
received: 29 01 2023
accepted: 21 06 2023
medline: 5 7 2023
pubmed: 3 7 2023
entrez: 3 7 2023
Statut: epublish

Résumé

There is an unmet need to develop potent therapeutics against cancer with minimal side effects and systemic toxicity. Thymol (TH) is an herbal medicine with anti-cancer properties that has been investigated scientifically. This study shows that TH induces apoptosis in cancerous cell lines such as MCF-7, AGS, and HepG2. Furthermore, this study reveals that TH can be encapsulated in a Polyvinyl alcohol (PVA)-coated niosome (Nio-TH/PVA) to enhance its stability and enable its controlled release as a model drug in the cancerous region. TH-loaded niosome (Nio-TH) was fabricated and optimized using Box-Behnken method and the size, polydispersity index (PDI) and entrapment efficiency (EE) were characterized by employing DLS, TEM and SEM, respectively. Additionally, in vitro drug release and kinetic studies were performed. Cytotoxicity, antiproliferative activity, and the mechanism were assessed by MTT assay, quantitative real-time PCR, flow cytometry, cell cycle, caspase activity evaluation, reactive oxygen species investigation, and cell migration assays. This study demonstrated the exceptional stability of Nio-TH/PVA at 4 °C for two months and its pH-dependent release profile. It also showed its high toxicity on cancerous cell lines and high compatibility with HFF cells. It revealed the modulation of Caspase-3/Caspase-9, MMP-2/MMP-9 and Cyclin D/ Cyclin E genes by Nio-TH/PVA on the studied cell lines. It confirmed the induction of apoptosis by Nio-TH/PVA in flow cytometry, caspase activity, ROS level, and DAPI staining assays. It also verified the inhibition of metastasis by Nio-TH/PVA in migration assays. Overall, the results of this study revealed that Nio-TH/PVA may effectively transport hydrophobic drugs to cancer cells with a controlled-release profile to induce apoptosis while exhibiting no detectable side effects due to their biocompatibility with normal cells.

Identifiants

pubmed: 37396433
doi: 10.2147/IJN.S401725
pii: 401725
pmc: PMC10314792
doi:

Substances chimiques

Polyvinyl Alcohol 9002-89-5
Thymol 3J50XA376E
Liposomes 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

3459-3488

Informations de copyright

© 2023 Abdihaji et al.

Déclaration de conflit d'intérêts

The authors declare that they have no conflicts of interest in this work.

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Auteurs

Mohammadreza Abdihaji (M)

Department of Biology, The Center for Genomics and Bioinformatics, Indiana University, Bloomington, IN, USA.

Masoumeh Mirzaei Chegeni (M)

Department of Biology, Science and Research Branch, Islamic Azad University, Tehran, Iran.

Alireza Hadizadeh (A)

Research Center for Advanced Technologies in Cardiovascular Medicine, Cardiovascular Diseases Research Institute, Tehran University of Medical Sciences, Tehran, Iran.

Negar Farrokhzad (N)

Faculty of Biology, Medicine and Health, University of Manchester, Manchester, UK.

Zahra Kheradmand (Z)

Department of Agriculture, Islamic Azad University Maragheh Branch, Maragheh, Iran.

Parastoo Fakhrfatemi (P)

Faculty of Pharmacy, Shiraz University of Medical Sciences, Shiraz, Iran.

Fardad Faress (F)

Department of Business, Data Analysis, The University of Texas Rio Grande Valley (UTRGV), Edinburg, TX, USA.

Kasra Moeinabadi-Bidgoli (K)

Basic and Molecular Epidemiology of Gastroenterology Disorders Research Center, Shahid Beheshti University of Medical Sciences, Tehran, Iran.

Hassan Noorbazargan (H)

Department of Biotechnology, School of Advanced Technologies in Medicine, Shahid Beheshti University of Medical Sciences, Tehran, Iran.

Ebrahim Mostafavi (E)

Stanford Cardiovascular Institute, Stanford University School of Medicine, Stanford, CA, USA.
Department of Medicine, Stanford University School of Medicine, Stanford, CA, USA.

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