Birth weight, adult body size, and risk of colorectal cancer.


Journal

Cancer epidemiology
ISSN: 1877-783X
Titre abrégé: Cancer Epidemiol
Pays: Netherlands
ID NLM: 101508793

Informations de publication

Date de publication:
08 2023
Historique:
received: 12 04 2023
revised: 09 06 2023
accepted: 23 06 2023
medline: 4 8 2023
pubmed: 7 7 2023
entrez: 6 7 2023
Statut: ppublish

Résumé

Evidence suggests that birth weight may be associated with colorectal cancer (CRC) risk later in life. Whether the association is mediated by adult body size remains unexamined. Cox proportional hazards models (Hazard Ratio (HR) and 95 % Confidence Intervals (CI)) were used to evaluate the association between self-reported birth weight (<6 lbs, 6-<8 lbs, ≥8 lbs) and CRC risk among 70,397 postmenopausal women from the Women's Health Initiative. Further, we assessed whether this association was mediated by adult body size using multiple mediation analyses. Compared with birth weights of 6-< 8 lbs, birth weight ≥ 8 lbs was associated with higher CRC risk in postmenopausal women (HR = 1.31, 95 % CI 1.16-1.48). This association was significantly mediated by adult height (proportion mediated =11.4 %), weight (11.2 %), waist circumference (10.9 %), and body mass index at baseline (4.0 %). The joint effect of adult height and weight explained 21.6 % of this positive association. Our data support the hypothesis that the intrauterine environment and fetal development may be related to the risk of developing CRC later in life. While adult body size partially explains this association, further investigation is required to identify other factors that mediate the link between birth weight and CRC.

Sections du résumé

BACKGROUND
Evidence suggests that birth weight may be associated with colorectal cancer (CRC) risk later in life. Whether the association is mediated by adult body size remains unexamined.
METHOD
Cox proportional hazards models (Hazard Ratio (HR) and 95 % Confidence Intervals (CI)) were used to evaluate the association between self-reported birth weight (<6 lbs, 6-<8 lbs, ≥8 lbs) and CRC risk among 70,397 postmenopausal women from the Women's Health Initiative. Further, we assessed whether this association was mediated by adult body size using multiple mediation analyses.
RESULTS
Compared with birth weights of 6-< 8 lbs, birth weight ≥ 8 lbs was associated with higher CRC risk in postmenopausal women (HR = 1.31, 95 % CI 1.16-1.48). This association was significantly mediated by adult height (proportion mediated =11.4 %), weight (11.2 %), waist circumference (10.9 %), and body mass index at baseline (4.0 %). The joint effect of adult height and weight explained 21.6 % of this positive association.
CONCLUSION
Our data support the hypothesis that the intrauterine environment and fetal development may be related to the risk of developing CRC later in life. While adult body size partially explains this association, further investigation is required to identify other factors that mediate the link between birth weight and CRC.

Identifiants

pubmed: 37413805
pii: S1877-7821(23)00087-5
doi: 10.1016/j.canep.2023.102407
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

102407

Informations de copyright

Copyright © 2023 Elsevier Ltd. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest None.

Auteurs

Le Su (L)

Department of Epidemiology and Biostatistics, School of Public Health, Indiana University Bloomington, Bloomington, IN, USA. Electronic address: lesu@iu.edu.

Michael Hendryx (M)

Department of Environmental and Occupational Health, School of Public Health, Indiana University, Bloomington, IN, USA.

Ming Li (M)

Department of Epidemiology and Biostatistics, School of Public Health, Indiana University Bloomington, Bloomington, IN, USA.

Margaret S Pichardo (MS)

Department of Chronic Disease Epidemiology, Yale School of Public Health, Yale University, New Haven, CT, USA.

Su Yon Jung (SY)

Department of Translational Sciences Section, School of Nursing, University of California, Los Angeles, CA, USA.

Dorothy S Lane (DS)

Department of Family, Population and Preventive Medicine, Renaissance School of Medicine, Stony Brook University, NY, USA.

Rowan Chlebowski (R)

Department of Medical Oncology, The Lundquist Institute, Torrance, CA, USA.

Yangbo Sun (Y)

Department of Preventive Medicine, College of Medicine, University of Tennessee Health Science Center, Memphis, TN, USA.

Chao Li (C)

Division of Epidemiology, Vanderbilt University Medical Center, Vanderbilt University, Nashville, TN, USA.

Juhua Luo (J)

Department of Epidemiology and Biostatistics, School of Public Health, Indiana University Bloomington, Bloomington, IN, USA.

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