Longitudinal motor decline in dementia with Lewy bodies, Parkinson disease dementia, and Alzheimer's dementia in a community autopsy cohort.


Journal

Alzheimer's & dementia : the journal of the Alzheimer's Association
ISSN: 1552-5279
Titre abrégé: Alzheimers Dement
Pays: United States
ID NLM: 101231978

Informations de publication

Date de publication:
10 2023
Historique:
revised: 04 05 2023
received: 12 01 2023
accepted: 05 05 2023
pmc-release: 01 10 2024
medline: 12 10 2023
pubmed: 9 7 2023
entrez: 8 7 2023
Statut: ppublish

Résumé

We examined the progression of extrapyramidal symptoms and signs in autopsy-confirmed dementia with Lewy bodies (DLB), Parkinson's disease dementia (PDD), and Alzheimer's disease dementia (AD). Longitudinal data were obtained from Arizona Study of Aging and Neurodegenerative Disease, with PDD (n = 98), AD (n = 47) and DLB (n = 48) further sub-grouped as with or without parkinsonism (DLB+ and DLB-). Within-group Unified Parkinson's Disease Rating Scale (UPDRS) -II and UPDRS-III trajectories were analyzed using non-linear mixed effects models. In DLB, 65.6% had parkinsonism. Baseline UPDRS-II and III scores (off-stage) were highest (P < 0.001) for PDD (mean ± SD 14.3 ± 7.8 and 27.4 ± 16.3), followed by DLB+ (6.0 ± 8.8 and 17.2 ± 17.1), DLB- (1.1 ± 1.3 and 3.3 ± 5.5) and AD (3.2 ± 6.1 and 8.2 ± 13.6). Compared to PDD, the DLB+ group had faster UPDRS-III progression over 8-years (Cohen's-d range 0.98 to 2.79, P < 0.001), driven by gait (P < 0.001) and limb bradykinesia (P = 0.02) subscales. Motor deficits progress faster in DLB+ than PDD, providing insights about expected changes in motor function. Dementia with Lewy bodies has faster motor progression than Parkinson's disease dementia Linear and non-linear mixed modeling analysis of longitudinal data was utilized Findings have implications for clinical prognostication and trial design.

Identifiants

pubmed: 37422286
doi: 10.1002/alz.13357
pmc: PMC10592344
mid: NIHMS1919180
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

4377-4387

Subventions

Organisme : NIA NIH HHS
ID : P30 AG019610
Pays : United States
Organisme : NINDS NIH HHS
ID : U24 NS072026
Pays : United States

Informations de copyright

© 2023 the Alzheimer's Association.

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Auteurs

Parichita Choudhury (P)

Cleo Roberts Center, Banner Sun Health Research Institute, Sun City, Arizona, USA.

Nan Zhang (N)

Department of Quantitative Health Sciences, Mayo Clinic College of Medicine, Scottsdale, Arizona, USA.

Charles H Adler (CH)

Department of Neurology, Mayo Clinic College of Medicine, Scottsdale, Arizona, USA.

Kewei Chen (K)

Computational Imaging Lab, Banner Alzheimer's Institute, Phoenix, Arizona, USA.

Christine Belden (C)

Cleo Roberts Center, Banner Sun Health Research Institute, Sun City, Arizona, USA.

Erika Driver-Dunckley (E)

Department of Neurology, Mayo Clinic College of Medicine, Scottsdale, Arizona, USA.

Shyamal H Mehta (SH)

Department of Neurology, Mayo Clinic College of Medicine, Scottsdale, Arizona, USA.

David R Shprecher (DR)

Cleo Roberts Center, Banner Sun Health Research Institute, Sun City, Arizona, USA.

Geidy E Serrano (GE)

Civin Laboratory for Neuropathology, Banner Sun Health Research Institute, Sun City, Arizona, USA.

Holly A Shill (HA)

Department of Neurology, Barrow Neurological Institute, Phoenix, Arizona, USA.

Thomas G Beach (TG)

Civin Laboratory for Neuropathology, Banner Sun Health Research Institute, Sun City, Arizona, USA.

Alireza Atri (A)

Cleo Roberts Center, Banner Sun Health Research Institute, Sun City, Arizona, USA.
Center for Brain/Mind Medicine & Department of Neurology, Brigham and Women's Hospital, Boston, Massachusetts, USA.
Department of Neurology, Harvard Medical School, Boston, Massachusetts, USA.

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