Prognostic value of neutrophil-to-lymphocyte ratio in patients with metastatic castration-resistant prostate cancer receiving prostate-specific membrane antigen targeted radionuclide therapy.


Journal

The Prostate
ISSN: 1097-0045
Titre abrégé: Prostate
Pays: United States
ID NLM: 8101368

Informations de publication

Date de publication:
10 2023
Historique:
revised: 23 05 2023
received: 19 03 2023
accepted: 29 06 2023
medline: 28 8 2023
pubmed: 10 7 2023
entrez: 10 7 2023
Statut: ppublish

Résumé

Neutrophil count:lymphocyte count ratio (NLR) may be a prognostic factor for men with advanced prostate cancer. We hypothesized that it is associated with prostate-specific antigen (PSA) response and survival in men treated with prostate-specific membrane antigen (PSMA)-targeted radionuclide therapy (TRT). Data of 180 men with metastatic castration-resistant prostate cancer (mCRPC) who were treated in sequential prospective radionuclide clinical trials from 2002 to 2021 (utilizing 177Lu-J591, 90Y-J591, 177Lu-PSMA-617, or 225Ac-J591) were retrospectively analyzed. We used a logistic regression to determine the association between NLR and ≥50% PSA decline (PSA50) and a Cox proportional hazards model to investigate the association between NLR and overall survival (OS). A total of 94 subjects (52.2%) received 177Lu-J591, 51 (28.3%) 177Lu-PSMA-617, 28 (15.6%) 225Ac-J591, and 7 (3.9%) 90Y-J591. The median NLR of 3.75 was used as cut-off (low vs. high NLR; n = 90, respectively). On univariate analysis, NLR was not associated with PSA50 (HR 1.08; 95% confidence interval [CI] 0.99-1.17, p = 0.067). However, it was associated with worse OS (hazard ratio [HR] 1.06, 95% CI 1.02-1.09, p = 0.002), also after controlling for circulating tumor cell count and cancer and leukemia group B risk group (HR 1.05; 95% CI 1.003-1.11, p = 0.036). Men with high NLR were at a higher hazard of death from all causes (HR 1.43, 95% CI 1.05-1.94, p = 0.024). NLR provides prognostic information in the setting of patients with mCRPC receiving treatment with PSMA-TRT.

Sections du résumé

BACKGROUND
Neutrophil count:lymphocyte count ratio (NLR) may be a prognostic factor for men with advanced prostate cancer. We hypothesized that it is associated with prostate-specific antigen (PSA) response and survival in men treated with prostate-specific membrane antigen (PSMA)-targeted radionuclide therapy (TRT).
METHODS
Data of 180 men with metastatic castration-resistant prostate cancer (mCRPC) who were treated in sequential prospective radionuclide clinical trials from 2002 to 2021 (utilizing 177Lu-J591, 90Y-J591, 177Lu-PSMA-617, or 225Ac-J591) were retrospectively analyzed. We used a logistic regression to determine the association between NLR and ≥50% PSA decline (PSA50) and a Cox proportional hazards model to investigate the association between NLR and overall survival (OS).
RESULTS
A total of 94 subjects (52.2%) received 177Lu-J591, 51 (28.3%) 177Lu-PSMA-617, 28 (15.6%) 225Ac-J591, and 7 (3.9%) 90Y-J591. The median NLR of 3.75 was used as cut-off (low vs. high NLR; n = 90, respectively). On univariate analysis, NLR was not associated with PSA50 (HR 1.08; 95% confidence interval [CI] 0.99-1.17, p = 0.067). However, it was associated with worse OS (hazard ratio [HR] 1.06, 95% CI 1.02-1.09, p = 0.002), also after controlling for circulating tumor cell count and cancer and leukemia group B risk group (HR 1.05; 95% CI 1.003-1.11, p = 0.036). Men with high NLR were at a higher hazard of death from all causes (HR 1.43, 95% CI 1.05-1.94, p = 0.024).
CONCLUSIONS
NLR provides prognostic information in the setting of patients with mCRPC receiving treatment with PSMA-TRT.

Identifiants

pubmed: 37424145
doi: 10.1002/pros.24597
doi:

Substances chimiques

Actinium-225 0
Actinium NIK1K0956U
Yttrium-90 1K8M7UR6O1
Yttrium Radioisotopes 0
Prostate-Specific Antigen EC 3.4.21.77
Lutetium-177 BRH40Y9V1Q

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1351-1357

Informations de copyright

© 2023 Wiley Periodicals LLC.

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Auteurs

Judith Stangl-Kremser (J)

Department of Urology, Weill Cornell Medicine, New York, New York, USA.

Michael Sun (M)

Department of Medicine, Division of Hematology and Medical Oncology, Weill Cornell Medicine, New York, New York, USA.

Benedict Ho (B)

Department of Medicine, Division of Hematology and Medical Oncology, Weill Cornell Medicine, New York, New York, USA.

Joseph Thomas (J)

Department of Medicine, Division of Hematology and Medical Oncology, Weill Cornell Medicine, New York, New York, USA.

Jones T Nauseef (JT)

Department of Medicine, Division of Hematology and Medical Oncology, Weill Cornell Medicine, New York, New York, USA.
Meyer Cancer Center, Weill Cornell Medicine, New York, New York, USA.

Joseph R Osborne (JR)

Meyer Cancer Center, Weill Cornell Medicine, New York, New York, USA.
Department of Radiology, Division of Molecular Imaging and Therapeutics, Weill Cornell Medicine, New York, New York, USA.

Ana Molina (A)

Department of Medicine, Division of Hematology and Medical Oncology, Weill Cornell Medicine, New York, New York, USA.

Cora N Sternberg (CN)

Department of Medicine, Division of Hematology and Medical Oncology, Weill Cornell Medicine, New York, New York, USA.
Meyer Cancer Center, Weill Cornell Medicine, New York, New York, USA.
Englander Institute for Precision Medicine, Weill Cornell Medicine, New York, New York, USA.

David M Nanus (DM)

Department of Medicine, Division of Hematology and Medical Oncology, Weill Cornell Medicine, New York, New York, USA.
Meyer Cancer Center, Weill Cornell Medicine, New York, New York, USA.

Neil H Bander (NH)

Department of Urology, Weill Cornell Medicine, New York, New York, USA.
Meyer Cancer Center, Weill Cornell Medicine, New York, New York, USA.

Scott Tagawa (S)

Department of Medicine, Division of Hematology and Medical Oncology, Weill Cornell Medicine, New York, New York, USA.
Meyer Cancer Center, Weill Cornell Medicine, New York, New York, USA.

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