The additive value of CA19.9 monitoring in a pancreatic cyst surveillance program.

CA19.9 GICA biomarkers early detection follow-up pancreatic cancer pancreatic cysts pancreatic lesions surgical intervention surveillance

Journal

United European gastroenterology journal
ISSN: 2050-6414
Titre abrégé: United European Gastroenterol J
Pays: England
ID NLM: 101606807

Informations de publication

Date de publication:
09 2023
Historique:
received: 25 02 2023
accepted: 05 05 2023
medline: 12 9 2023
pubmed: 12 7 2023
entrez: 12 7 2023
Statut: ppublish

Résumé

Surveillance of pancreatic cysts focuses on the detection of (mostly morphologic) features warranting surgery. European guidelines consider elevated CA19.9 as a relative indication for surgery. We aimed to evaluate the role of CA19.9 monitoring for early detection and management in a cyst surveillance population. The PACYFIC-registry is a prospective collaboration that investigates the yield of pancreatic cyst surveillance performed at the discretion of the treating physician. We included participants for whom at least one serum CA19.9 value was determined by a minimum follow-up of 12 months. Of 1865 PACYFIC participants, 685 met the inclusion criteria for this study (mean age 67 years, SD 10; 61% female). During a median follow-up of 25 months (IQR 24, 1966 visits), 29 participants developed high-grade dysplasia (HGD) or pancreatic cancer. At baseline, CA19.9 ranged from 1 to 591 kU/L (median 10 kU/L [IQR 14]), and was elevated (≥37 kU/L) in 64 participants (9%). During 191 of 1966 visits (10%), an elevated CA19.9 was detected, and these visits more often led to an intensified follow-up (42%) than those without an elevated CA19.9 (27%; p < 0.001). An elevated CA19.9 was the sole reason for surgery in five participants with benign disease (10%). The baseline CA19.9 value was (as continuous or dichotomous variable at the 37 kU/L threshold) not independently associated with HGD or pancreatic cancer development, whilst a CA19.9 of ≥ 133 kU/L was (HR 3.8, 95% CI 1.1-13, p = 0.03). In this pancreatic cyst surveillance cohort, CA19.9 monitoring caused substantial harm by shortening surveillance intervals (and performance of unnecessary surgery). The current CA19.9 cutoff was not predictive of HGD and pancreatic cancer, whereas a higher cutoff may decrease false-positive values. The role of CA19.9 monitoring should be critically appraised prior to implementation in surveillance programs and guidelines.

Sections du résumé

BACKGROUND
Surveillance of pancreatic cysts focuses on the detection of (mostly morphologic) features warranting surgery. European guidelines consider elevated CA19.9 as a relative indication for surgery. We aimed to evaluate the role of CA19.9 monitoring for early detection and management in a cyst surveillance population.
METHODS
The PACYFIC-registry is a prospective collaboration that investigates the yield of pancreatic cyst surveillance performed at the discretion of the treating physician. We included participants for whom at least one serum CA19.9 value was determined by a minimum follow-up of 12 months.
RESULTS
Of 1865 PACYFIC participants, 685 met the inclusion criteria for this study (mean age 67 years, SD 10; 61% female). During a median follow-up of 25 months (IQR 24, 1966 visits), 29 participants developed high-grade dysplasia (HGD) or pancreatic cancer. At baseline, CA19.9 ranged from 1 to 591 kU/L (median 10 kU/L [IQR 14]), and was elevated (≥37 kU/L) in 64 participants (9%). During 191 of 1966 visits (10%), an elevated CA19.9 was detected, and these visits more often led to an intensified follow-up (42%) than those without an elevated CA19.9 (27%; p < 0.001). An elevated CA19.9 was the sole reason for surgery in five participants with benign disease (10%). The baseline CA19.9 value was (as continuous or dichotomous variable at the 37 kU/L threshold) not independently associated with HGD or pancreatic cancer development, whilst a CA19.9 of ≥ 133 kU/L was (HR 3.8, 95% CI 1.1-13, p = 0.03).
CONCLUSIONS
In this pancreatic cyst surveillance cohort, CA19.9 monitoring caused substantial harm by shortening surveillance intervals (and performance of unnecessary surgery). The current CA19.9 cutoff was not predictive of HGD and pancreatic cancer, whereas a higher cutoff may decrease false-positive values. The role of CA19.9 monitoring should be critically appraised prior to implementation in surveillance programs and guidelines.

Identifiants

pubmed: 37435855
doi: 10.1002/ueg2.12422
pmc: PMC10493362
doi:

Substances chimiques

CA-19-9 Antigen 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

601-611

Informations de copyright

© 2023 The Authors. United European Gastroenterology Journal published by Wiley Periodicals LLC on behalf of United European Gastroenterology.

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Auteurs

Iris J M Levink (IJM)

Department of Gastroenterology & Hepatology, Erasmus University Medical Center, Rotterdam, The Netherlands.

Sanne C Jaarsma (SC)

Department of Gastroenterology & Hepatology, Erasmus University Medical Center, Rotterdam, The Netherlands.

Brechtje D M Koopmann (BDM)

Department of Gastroenterology & Hepatology, Erasmus University Medical Center, Rotterdam, The Netherlands.
Department of Public Health, Erasmus University Medical Center, Rotterdam, The Netherlands.

Priscilla A van Riet (PA)

Department of Gastroenterology & Hepatology, Erasmus University Medical Center, Rotterdam, The Netherlands.

Kasper A Overbeek (KA)

Department of Gastroenterology & Hepatology, Erasmus University Medical Center, Rotterdam, The Netherlands.

Jihane Meziani (J)

Department of Gastroenterology & Hepatology, Erasmus University Medical Center, Rotterdam, The Netherlands.

Marloes L J A Sprij (MLJA)

Department of Gastroenterology & Hepatology, Erasmus University Medical Center, Rotterdam, The Netherlands.

Riccardo Casadei (R)

Department of Surgery, Bologna University, Bologna, Italy.

Carlo Ingaldi (C)

Department of Surgery, Bologna University, Bologna, Italy.

Marcin Polkowski (M)

Department of Gastroenterology, Hepatology, and Clinical Oncology, Center of Postgraduate Medical Education, Warsaw, Poland.
Department of Oncological Gastroenterology, The Maria Sklodowska-Curie National Research Institute of Oncology, Warsaw, Poland.

Megan M L Engels (MML)

Department of Gastroenterology & Hepatology, Mayo Clinic, Jacksonville, Florida, USA.
Department of Gastroenterology & Hepatology, Leiden University Medical Center, Leiden, The Netherlands.

Laurens A van der Waaij (LA)

Department of Gastroenterology & Hepatology, Martini Hospital, Groningen, The Netherlands.

Silvia Carrara (S)

Department of Gastroenterology, IRCCS Humanitas Research Hospital, Milan, Italy.

Elizabeth Pando (E)

Department of Surgery, Vall d'Hebron Institute of Research, Barcelona, Spain.

Marlies Vornhülz (M)

Department of Gastroenterology & Hepatology, Ludwig-Maximilians-University of Munich, Munich, Germany.

Pieter Honkoop (P)

Department of Gastroenterology & Hepatology, Albert Schweitzer Hospital, Dordrecht, The Netherlands.

Erik J Schoon (EJ)

Department of Gastroenterology & Hepatology, Catharina Hospital, Eindhoven, The Netherlands.

Johanna Laukkarinen (J)

Department of Surgery, Tampere University Hospital, Tampere, Finland.

Jilling F Bergmann (JF)

Department of Gastroenterology & Hepatology, Haga Ziekenhuis, The Hague, The Netherlands.

Gemma Rossi (G)

Pancreato-Biliary Endoscopy and Endosonography Division, Pancreas Translational and Clinical Research Center, San Raffaele Scientific Institute IRCCS, Vita Salute San Raffaele University, Milan, Italy.

Frederike G I van Vilsteren (FGI)

Department of Gastroenterology & Hepatology, University Medical Center Groningen, Groningen, The Netherlands.

Anne-Marie van Berkel (AM)

Department of Gastroenterology & Hepatology, Noordwest Hospital, Alkmaar, The Netherlands.

Trevor Tabone (T)

Department of Gastroenterology & Hepatology, Mater dei Hospital, Msida, Malta.

Matthijs P Schwartz (MP)

Department of Gastroenterology & Hepatology, Meander Medical Center, Amersfoort, The Netherlands.

Adriaan C I T L Tan (ACITL)

Department of Gastroenterology & Hepatology, Canisius Wilhelmina Hospital, Nijmegen, The Netherlands.

Jeanin E van Hooft (JE)

Department of Gastroenterology & Hepatology, Leiden University Medical Center, Leiden, The Netherlands.
Department of Gastroenterology & Hepatology, Amsterdam UMC, Amsterdam, The Netherlands.

Rutger Quispel (R)

Department of Gastroenterology & Hepatology, Reinier de Graaf, Delft, The Netherlands.

Ellert van Soest (E)

Department of Gastroenterology & Hepatology, Spaarne Gasthuis, Haarlem, The Netherlands.

Laszlo Czacko (L)

Department of Gastroenterology & Hepatology, University of Szeged, Szeged, Hungary.

Marco J Bruno (MJ)

Department of Gastroenterology & Hepatology, Erasmus University Medical Center, Rotterdam, The Netherlands.

Djuna L Cahen (DL)

Department of Gastroenterology & Hepatology, Erasmus University Medical Center, Rotterdam, The Netherlands.
Department of Gastroenterology & Hepatology, Amstelland, Amstelveen, The Netherlands.

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