Endosome Traffic Modulates Pro-Inflammatory Signal Transduction in CD4
CD4+ T cells
IL-17
IL-2
JAK/STAT
autoimmunity
endosome traffic
glucose
glutamine
interferon
kynurenine
lysosome
mTOR
metabolism
systemic lupus erythematosus
tryptophan
Journal
International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791
Informations de publication
Date de publication:
28 Jun 2023
28 Jun 2023
Historique:
received:
05
05
2023
revised:
10
06
2023
accepted:
26
06
2023
medline:
17
7
2023
pubmed:
14
7
2023
entrez:
14
7
2023
Statut:
epublish
Résumé
Endocytic recycling regulates the cell surface receptor composition of the plasma membrane. The surface expression levels of the T cell receptor (TCR), in concert with signal transducing co-receptors, regulate T cell responses, such as proliferation, differentiation, and cytokine production. Altered TCR expression contributes to pro-inflammatory skewing, which is a hallmark of autoimmune diseases, such as systemic lupus erythematosus (SLE), defined by a reduced function of regulatory T cells (Tregs) and the expansion of CD4
Identifiants
pubmed: 37445926
pii: ijms241310749
doi: 10.3390/ijms241310749
pmc: PMC10341602
pii:
doi:
Substances chimiques
Cytokines
0
Receptors, Antigen, T-Cell
0
Types de publication
Journal Article
Review
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : NIAMS NIH HHS
ID : AR076092
Pays : United States
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