A spatially resolved timeline of the human maternal-fetal interface.


Journal

Nature
ISSN: 1476-4687
Titre abrégé: Nature
Pays: England
ID NLM: 0410462

Informations de publication

Date de publication:
Jul 2023
Historique:
received: 03 09 2021
accepted: 08 06 2023
medline: 21 7 2023
pubmed: 20 7 2023
entrez: 19 7 2023
Statut: ppublish

Résumé

Beginning in the first trimester, fetally derived extravillous trophoblasts (EVTs) invade the uterus and remodel its spiral arteries, transforming them into large, dilated blood vessels. Several mechanisms have been proposed to explain how EVTs coordinate with the maternal decidua to promote a tissue microenvironment conducive to spiral artery remodelling (SAR)

Identifiants

pubmed: 37468587
doi: 10.1038/s41586-023-06298-9
pii: 10.1038/s41586-023-06298-9
pmc: PMC10356615
doi:

Substances chimiques

MRC1 protein, human 0
CD163 antigen 0
LGALS9 protein, human 0
IDO1 protein, human 0
HAVCR2 protein, human 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

595-605

Subventions

Organisme : NIAID NIH HHS
ID : K08 AI137209
Pays : United States
Organisme : NCI NIH HHS
ID : P30 CA008748
Pays : United States

Informations de copyright

© 2023. The Author(s).

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Auteurs

Shirley Greenbaum (S)

Department of Pathology, Stanford University, Stanford, CA, USA. greenbaumsh@gmail.com.
Department of Obstetrics and Gynecology, Hadassah-Hebrew University Medical Center, Jerusalem, Israel. greenbaumsh@gmail.com.

Inna Averbukh (I)

Department of Pathology, Stanford University, Stanford, CA, USA.

Erin Soon (E)

Department of Pathology, Stanford University, Stanford, CA, USA.
Immunology Program, Stanford University, Stanford, CA, USA.

Gabrielle Rizzuto (G)

Department of Pathology, University of Californica San Francisco, San Francisco, CA, USA.

Alex Baranski (A)

Department of Pathology, Stanford University, Stanford, CA, USA.

Noah F Greenwald (NF)

Department of Pathology, Stanford University, Stanford, CA, USA.
Cancer Biology Program, Stanford University, Stanford, CA, USA.

Adam Kagel (A)

Department of Pathology, Stanford University, Stanford, CA, USA.

Marc Bosse (M)

Department of Pathology, Stanford University, Stanford, CA, USA.

Eleni G Jaswa (EG)

Department of Obstetrics Gynecology and Reproductive Sciences, University of California San Francisco, San Francisco, CA, USA.

Zumana Khair (Z)

Department of Pathology, Stanford University, Stanford, CA, USA.

Shirley Kwok (S)

Department of Pathology, Stanford University, Stanford, CA, USA.

Shiri Warshawsky (S)

Department of Pathology, Stanford University, Stanford, CA, USA.

Hadeesha Piyadasa (H)

Department of Pathology, Stanford University, Stanford, CA, USA.

Mako Goldston (M)

Department of Pathology, Stanford University, Stanford, CA, USA.

Angie Spence (A)

Department of Pathology, Stanford University, Stanford, CA, USA.

Geneva Miller (G)

Division of Biology and Bioengineering, California Institute of Technology, Pasadena, CA, USA.

Morgan Schwartz (M)

Division of Biology and Bioengineering, California Institute of Technology, Pasadena, CA, USA.

Will Graf (W)

Division of Biology and Bioengineering, California Institute of Technology, Pasadena, CA, USA.

David Van Valen (D)

Division of Biology and Bioengineering, California Institute of Technology, Pasadena, CA, USA.

Virginia D Winn (VD)

Department of Obstetrics and Gynecology, Stanford University, Stanford, CA, USA.

Travis Hollmann (T)

Department of Pathology, Memorial Sloan Kettering Cancer Center, New York, NY, USA.

Leeat Keren (L)

Department of Molecular Cell Biology, Weizmann Institute of Science, Rehovot, Israel.

Matt van de Rijn (M)

Department of Pathology, Stanford University, Stanford, CA, USA.

Michael Angelo (M)

Department of Pathology, Stanford University, Stanford, CA, USA. mangelo0@stanford.edu.

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