Sex Differences in Coronary Atherosclerotic Phenotype and Healing Pattern on Optical Coherence Tomography Imaging.


Journal

Circulation. Cardiovascular imaging
ISSN: 1942-0080
Titre abrégé: Circ Cardiovasc Imaging
Pays: United States
ID NLM: 101479935

Informations de publication

Date de publication:
08 2023
Historique:
medline: 17 8 2023
pubmed: 28 7 2023
entrez: 28 7 2023
Statut: ppublish

Résumé

Layered plaque, a signature of previous plaque disruption, is a known predictor of rapid plaque progression. Layered plaque can be identified in vivo by optical coherence tomography. Studies have reported differences in plaque burden between women and men, but sex differences in the pattern of layered plaque are unknown. Preintervention optical coherence tomography images of 533 patients with chronic coronary syndromes were analyzed. Detailed plaque characteristics of layered and nonlayered plaques of the target lesion were compared between men and women. The prevalence of layered plaque was similar between men (N=418) and women (N=115; 55% versus 54%; In men, layered plaques exhibit more features of vascular inflammation and vulnerability as well as evidence of previous plaque rupture, compared with nonlayered plaques, whereas in women, no difference was observed between layered and nonlayered plaques. Vascular inflammation (plaque rupture) may be the predominant mechanism of layered plaque in men, whereas a less inflammatory mechanism may play a key role in women. URL: http://www. gov; Unique Identifier: NCT01110538, NCT04523194.

Sections du résumé

BACKGROUND
Layered plaque, a signature of previous plaque disruption, is a known predictor of rapid plaque progression. Layered plaque can be identified in vivo by optical coherence tomography. Studies have reported differences in plaque burden between women and men, but sex differences in the pattern of layered plaque are unknown.
METHODS
Preintervention optical coherence tomography images of 533 patients with chronic coronary syndromes were analyzed. Detailed plaque characteristics of layered and nonlayered plaques of the target lesion were compared between men and women.
RESULTS
The prevalence of layered plaque was similar between men (N=418) and women (N=115; 55% versus 54%;
CONCLUSIONS
In men, layered plaques exhibit more features of vascular inflammation and vulnerability as well as evidence of previous plaque rupture, compared with nonlayered plaques, whereas in women, no difference was observed between layered and nonlayered plaques. Vascular inflammation (plaque rupture) may be the predominant mechanism of layered plaque in men, whereas a less inflammatory mechanism may play a key role in women.
REGISTRATION
URL: http://www.
CLINICALTRIALS
gov; Unique Identifier: NCT01110538, NCT04523194.

Identifiants

pubmed: 37503629
doi: 10.1161/CIRCIMAGING.123.015227
doi:

Banques de données

ClinicalTrials.gov
['NCT01110538', 'NCT04523194']

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e015227

Commentaires et corrections

Type : CommentIn

Auteurs

Lena Marie Seegers (LM)

Cardiology Division, Massachusetts General Hospital, Harvard Medical School, Boston (L.M.S., D.D.Y., M.A., A.N., H.Y., D.K., K.S., T.N., I.M.).

Doreen DeFaria Yeh (D)

Cardiology Division, Massachusetts General Hospital, Harvard Medical School, Boston (L.M.S., D.D.Y., M.A., A.N., H.Y., D.K., K.S., T.N., I.M.).

Taishi Yonetsu (T)

Department of Interventional Cardiology, Tokyo Medical and Dental University, Japan (T.Y.).

Tomoyo Sugiyama (T)

Department of Cardiovascular Medicine, Tsuchiura Kyodo General Hospital, Ibaraki, Japan (T.S., T.K.).

Yoshiyasu Minami (Y)

Department of Cardiovascular Medicine, Kitasato University School of Medicine, Sagamihara, Kanagawa, Japan (Y.M.).

Tsunenari Soeda (T)

Department of Cardiovascular Medicine, Nara Prefecture General Medical Center, Japan (T.S.).

Makoto Araki (M)

Cardiology Division, Massachusetts General Hospital, Harvard Medical School, Boston (L.M.S., D.D.Y., M.A., A.N., H.Y., D.K., K.S., T.N., I.M.).

Akihiro Nakajima (A)

Cardiology Division, Massachusetts General Hospital, Harvard Medical School, Boston (L.M.S., D.D.Y., M.A., A.N., H.Y., D.K., K.S., T.N., I.M.).

Haruhito Yuki (H)

Cardiology Division, Massachusetts General Hospital, Harvard Medical School, Boston (L.M.S., D.D.Y., M.A., A.N., H.Y., D.K., K.S., T.N., I.M.).

Daisuke Kinoshita (D)

Cardiology Division, Massachusetts General Hospital, Harvard Medical School, Boston (L.M.S., D.D.Y., M.A., A.N., H.Y., D.K., K.S., T.N., I.M.).

Keishi Suzuki (K)

Cardiology Division, Massachusetts General Hospital, Harvard Medical School, Boston (L.M.S., D.D.Y., M.A., A.N., H.Y., D.K., K.S., T.N., I.M.).

Takayuki Niida (T)

Cardiology Division, Massachusetts General Hospital, Harvard Medical School, Boston (L.M.S., D.D.Y., M.A., A.N., H.Y., D.K., K.S., T.N., I.M.).

Hang Lee (H)

Biostatistics Center, Massachusetts General Hospital, Harvard Medical School, Boston (H.L.).

Iris McNulty (I)

Cardiology Division, Massachusetts General Hospital, Harvard Medical School, Boston (L.M.S., D.D.Y., M.A., A.N., H.Y., D.K., K.S., T.N., I.M.).

Sunao Nakamura (S)

Interventional Cardiology Unit, New Tokyo Hospital, Chiba, Japan (S.N.).

Tsunekazu Kakuta (T)

Department of Cardiovascular Medicine, Tsuchiura Kyodo General Hospital, Ibaraki, Japan (T.S., T.K.).

Valentin Fuster (V)

Zena and Michael A. Wiener Cardiovascular Institute, Icahn School of Medicine at Mount Sinai, New York (V.F.).

Ik-Kyung Jang (IK)

Division of Cardiology, Kyung Hee University Hospital, Seoul, Korea (I.-K.J.).

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