Coordination-driven FBXW7 DNAzyme-Fe nanoassembly enables a binary switch of breast cancer cell cycle checkpoint responses for enhanced ferroptosis-radiotherapy.


Journal

Acta biomaterialia
ISSN: 1878-7568
Titre abrégé: Acta Biomater
Pays: England
ID NLM: 101233144

Informations de publication

Date de publication:
01 10 2023
Historique:
received: 02 03 2023
revised: 22 06 2023
accepted: 24 07 2023
medline: 18 9 2023
pubmed: 30 7 2023
entrez: 29 7 2023
Statut: ppublish

Résumé

Radiotherapy is a mainstream modality for breast cancer treatment that employs ionizing radiation (IR) to damage tumor cell DNA and elevate ROS stress, which demonstrates multiple clinically-favorable advantages including localized treatment and low invasiveness. However, breast cancer cells may activate the p53-mediated cell cycle regulation in response to radiotherapy to repair IR-induced cellular damage and facilitate post-treatment survival. F-Box and WD Repeat Domain Containing 7 (FBXW7) is a promoter of p53 degradation and critical nexus of cell proliferation and survival events. Herein, we engineered a cooperative radio-ferroptosis-stimulatory nanomedicine through coordination-driven self-assembly between ferroptosis-inducing Fe

Identifiants

pubmed: 37516418
pii: S1742-7061(23)00432-4
doi: 10.1016/j.actbio.2023.07.042
pii:
doi:

Substances chimiques

F-Box-WD Repeat-Containing Protein 7 0
DNA, Catalytic 0
Cell Cycle Proteins 0
F-Box Proteins 0
Tumor Suppressor Protein p53 0
Dopamine VTD58H1Z2X
Hyaluronic Acid 9004-61-9
Reactive Oxygen Species 0
Ubiquitin-Protein Ligases EC 2.3.2.27
FBXW7 protein, human 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

434-450

Informations de copyright

Copyright © 2023 Acta Materialia Inc. Published by Elsevier Ltd. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper.

Auteurs

Jiawu Yu (J)

School of Life Science, Chongqing University, Chongqing 400044, China.

Yuchen Zhang (Y)

School of Life Science, Chongqing University, Chongqing 400044, China.

Liqi Li (L)

Department of General Surgery, Xinqiao Hospital, Army Medical University, Chongqing 400037, China.

Yang Xiang (Y)

Laboratory of Radiation Biology, Department of Blood Transfusion, Laboratory Medicine Centre, The Second Affiliated Hospital, Army Medical University, Chongqing, China.

Xuemei Yao (X)

School of Life Science, Chongqing University, Chongqing 400044, China.

Youbo Zhao (Y)

School of Life Science, Chongqing University, Chongqing 400044, China.

Kaiyong Cai (K)

Key Laboratory of Biorheological Science and Technology, Ministry of Education, College of Bioengineering, Chongqing University, Chongqing 400044, China.

Menghuan Li (M)

School of Life Science, Chongqing University, Chongqing 400044, China. Electronic address: menghuanli@cqu.edu.cn.

Zhongjun Li (Z)

Laboratory of Radiation Biology, Department of Blood Transfusion, Laboratory Medicine Centre, The Second Affiliated Hospital, Army Medical University, Chongqing, China. Electronic address: johnneyusc@gmail.com.

Zhong Luo (Z)

School of Life Science, Chongqing University, Chongqing 400044, China. Electronic address: luozhong918@cqu.edu.cn.

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Classifications MeSH