Decay of HIV RNA in Seminal Plasma and Rectal Fluid in Treatment-Naive Adults Starting Antiretroviral Therapy With Dolutegravir Plus Lamivudine or Bictegravir/Emtricitabine/Tenofovir Alafenamide.


Journal

The Journal of infectious diseases
ISSN: 1537-6613
Titre abrégé: J Infect Dis
Pays: United States
ID NLM: 0413675

Informations de publication

Date de publication:
03 10 2023
Historique:
received: 13 07 2023
accepted: 30 07 2023
medline: 5 10 2023
pubmed: 1 8 2023
entrez: 1 8 2023
Statut: ppublish

Résumé

Decay of HIV in seminal plasma (SP) and rectal fluid (RF) has not yet been described for the antiretroviral combination of dolutegravir (DTG) + lamivudine (3TC). In this randomized multicenter pilot trial, males who were antiretroviral naive were randomized (2:1) to DTG + 3TC or bictegravir/emtricitabine/tenofovir alafenamide (BIC/FTC/TAF). HIV-1 RNA was measured in blood plasma (BP), SP, and RF at baseline; days 3, 7, 14, and 28; and weeks 12 and 24. Of 25 individuals enrolled, 24 completed the study (DTG + 3TC, n = 16; BIC/FTC/TAF, n = 8). No significant differences were observed between groups for median decline in HIV-1 RNA from baseline at each time point or median time to achieve HIV-1 RNA <20 copies/mL in BP and SP and <20 copies/swab in RF. HIV-1 RNA decay patterns were compared in individuals receiving DTG + 3TC. Despite significantly higher percentages for changes from baseline in BP, median (IQR) times to HIV-1 RNA suppression were shorter in SP (7 days; 0-8.75) and RF (10.5 days; 3-17.5) than in BP (28 days; 14-84; P < .001). Comparable HIV-1 RNA decay in BP, SP, and RF was observed between DTG + 3TC and BIC/FTC/TAF. As shown with triple-drug integrase inhibitor-based regimens, rapid HIV-1 RNA suppression in SP and RF is achieved with DTG + 3TC, despite decay patterns differing from those of BP. EudraCT 2019-004109-28.

Sections du résumé

BACKGROUND
Decay of HIV in seminal plasma (SP) and rectal fluid (RF) has not yet been described for the antiretroviral combination of dolutegravir (DTG) + lamivudine (3TC).
METHODS
In this randomized multicenter pilot trial, males who were antiretroviral naive were randomized (2:1) to DTG + 3TC or bictegravir/emtricitabine/tenofovir alafenamide (BIC/FTC/TAF). HIV-1 RNA was measured in blood plasma (BP), SP, and RF at baseline; days 3, 7, 14, and 28; and weeks 12 and 24.
RESULTS
Of 25 individuals enrolled, 24 completed the study (DTG + 3TC, n = 16; BIC/FTC/TAF, n = 8). No significant differences were observed between groups for median decline in HIV-1 RNA from baseline at each time point or median time to achieve HIV-1 RNA <20 copies/mL in BP and SP and <20 copies/swab in RF. HIV-1 RNA decay patterns were compared in individuals receiving DTG + 3TC. Despite significantly higher percentages for changes from baseline in BP, median (IQR) times to HIV-1 RNA suppression were shorter in SP (7 days; 0-8.75) and RF (10.5 days; 3-17.5) than in BP (28 days; 14-84; P < .001).
CONCLUSIONS
Comparable HIV-1 RNA decay in BP, SP, and RF was observed between DTG + 3TC and BIC/FTC/TAF. As shown with triple-drug integrase inhibitor-based regimens, rapid HIV-1 RNA suppression in SP and RF is achieved with DTG + 3TC, despite decay patterns differing from those of BP.
CLINICAL TRIALS REGISTRATION
EudraCT 2019-004109-28.

Identifiants

pubmed: 37526315
pii: 7234537
doi: 10.1093/infdis/jiad304
doi:

Substances chimiques

tenofovir alafenamide EL9943AG5J
Lamivudine 2T8Q726O95
bictegravir 8GB79LOJ07
dolutegravir DKO1W9H7M1
Heterocyclic Compounds, 3-Ring 0
Pyridones 0
Anti-Retroviral Agents 0
Drug Combinations 0
RNA, Viral 0
Emtricitabine G70B4ETF4S
Anti-HIV Agents 0

Banques de données

EudraCT
['2019-004109-28']

Types de publication

Randomized Controlled Trial Multicenter Study Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

919-925

Informations de copyright

© The Author(s) 2023. Published by Oxford University Press on behalf of Infectious Diseases Society of America. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com.

Déclaration de conflit d'intérêts

Potential conflicts of interest. S. S. has received financial compensation for educational activities, as well as training courses, funds for research, travel grants, and nonfinancial support from Gilead Sciences, Janssen-Cilag, Merck Sharp & Dohme, and ViiV Healthcare. P. D. has received financial compensation for lectures, consultancy work, and educational activities, from Gilead Sciences, GSK, Ferrer International, Janssen-Cilag, Merck Sharp & Dohme, Roche, Thera Technologies, and ViiV Healthcare. A. C. has received financial compensation for lectures, consultancy work, and educational activities, as well as funding for research, travel grants, and nonfinancial support from Gilead Sciences, Janssen-Cilag, Merck Sharp & Dohme, Thera Technologies, and ViiV Healthcare. J. T. has received financial compensation for lectures, consultancies, and educational activities as well as research funding from Gilead Sciences, Janssen-Cilag, Merck Sharp & Dohme, and ViiV Healthcare. D. P. has received research grants and/or honoraria for advisory boards and/or conferences from Gilead Sciences, Janssen-Cilag, Merck Sharp & Dohme, and ViiV Healthcare. A. I. has received financial compensation for lectures, consultancy work, and educational activities, as well as funding for research, travel grants, and nonfinancial support from Gilead Sciences, Janssen-Cilag, Merck Sharp & Dohme, Thera Technologies, and ViiV Healthcare. All other authors report no potential conflicts.

Auteurs

Sofía Scévola (S)

HIV and STI Unit, Department of Infectious Diseases, Bellvitge University Hospital, Bellvitge Biomedical Research Institute (IDIBELL), University of Barcelona, L'Hospitalet de Llobregat, Barcelona, Spain.

Jordi Niubó (J)

Department of Microbiology, Bellvitge University Hospital, Bellvitge Biomedical Research Institute (IDIBELL), University of Barcelona, L'Hospitalet de Llobregat, Barcelona, Spain.

Pere Domingo (P)

HIV Unit, Santa Creu i Sant Pau Hospital, Barcelona, Spain.

Guillermo Verdejo (G)

Department of Internal Medicine, Sant Camil Hospital, Sant Pere de Ribes, Barcelona, Spain.

Adrian Curran (A)

Department of Infectious Diseases, Vall d'Hebron University Hospital, Vall d'Hebron Research Institute, Barcelona, Spain.

Vicens Diaz-Brito (V)

Department of Infectious Diseases, Parc Sanitari Sant Joan de Déu, Sant Boi de Llobregat, Barcelona, Spain.

Judith Peñafiel (J)

Biostatistics Unit, Bellvitge Biomedical Research Institute (IDIBELL), L'Hospitalet del Llobregat, Barcelona, Spain.

Juan Tiraboschi (J)

HIV and STI Unit, Department of Infectious Diseases, Bellvitge University Hospital, Bellvitge Biomedical Research Institute (IDIBELL), University of Barcelona, L'Hospitalet de Llobregat, Barcelona, Spain.

Sandra Morenilla (S)

HIV and STI Unit, Department of Infectious Diseases, Bellvitge University Hospital, Bellvitge Biomedical Research Institute (IDIBELL), University of Barcelona, L'Hospitalet de Llobregat, Barcelona, Spain.

Benito Garcia (B)

HIV and STI Unit, Department of Infectious Diseases, Bellvitge University Hospital, Bellvitge Biomedical Research Institute (IDIBELL), University of Barcelona, L'Hospitalet de Llobregat, Barcelona, Spain.

Irene Soriano (I)

HIV and STI Unit, Department of Infectious Diseases, Bellvitge University Hospital, Bellvitge Biomedical Research Institute (IDIBELL), University of Barcelona, L'Hospitalet de Llobregat, Barcelona, Spain.

Daniel Podzamczer (D)

HIV and STI Unit, Department of Infectious Diseases, Bellvitge University Hospital, Bellvitge Biomedical Research Institute (IDIBELL), University of Barcelona, L'Hospitalet de Llobregat, Barcelona, Spain.

Arkaitz Imaz (A)

HIV and STI Unit, Department of Infectious Diseases, Bellvitge University Hospital, Bellvitge Biomedical Research Institute (IDIBELL), University of Barcelona, L'Hospitalet de Llobregat, Barcelona, Spain.

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Classifications MeSH