First-in-Human Study of the Safety, Pharmacokinetics, and Pharmacodynamics of MHV370, a Dual Inhibitor of Toll-Like Receptors 7 and 8, in Healthy Adults.


Journal

European journal of drug metabolism and pharmacokinetics
ISSN: 2107-0180
Titre abrégé: Eur J Drug Metab Pharmacokinet
Pays: France
ID NLM: 7608491

Informations de publication

Date de publication:
Sep 2023
Historique:
accepted: 16 07 2023
medline: 6 9 2023
pubmed: 3 8 2023
entrez: 2 8 2023
Statut: ppublish

Résumé

MHV370, a dual antagonist of human Toll-like receptors (TLR) 7 and 8, suppresses cytokines and interferon-stimulated genes in vitro and in vivo, and  has demonstrated efficacy in murine models of lupus. This first-in-human study aimed to evaluate the safety, tolerability, pharmacokinetics and pharmacodynamics of single and multiple doses of MHV370 in healthy adults, as well as the effects of food consumption on a single dose of MHV370. This was a phase 1, randomised, placebo-controlled study conducted in three parts. In part A, participants received (3:1) a single ascending dose (SAD) of 1, 3, 10, 20, 40, 80, 160, 320, 640 and 1000 mg MHV370 or placebo. In part B, participants received (3:1) multiple ascending doses (MAD) of 25, 50, 100, 200 and 400 mg MHV370 twice daily (b.i.d) or placebo for 14 days. In part C, participants received an open-label single dose of 200 mg MHV370 under fasted or fed conditions. Safety, pharmacokinetic and pharmacodynamic parameters were evaluated. MHV370 was well tolerated, and no safety signal was observed in the study. No dose-limiting adverse events occurred across the dose range evaluated. Plasma concentrations of MHV370 increased with dose (mean [SD] maximum plasma concentrations ranged from 0.97 [0.48] to 1670 [861.0] ng/mL for SAD of 3-1000 mg, 29.5 [7.98] to 759 [325.0] ng/mL for MAD of 25-400 mg b.i.d. on day 1). The intake of food did not have a relevant impact on the pharmacokinetics of MHV370. Pharmacodynamic data indicated time- and dose-dependent inhibition of TLR7-mediated CD69 expression on B cells (100% inhibition at 24 h post-dose starting from SAD 160 mg and MAD 50 mg b.i.d.) and TLR8-mediated TNF release after ex vivo stimulation (>90% inhibition at 24 h post-dose starting from SAD 320 mg and MAD 100 mg b.i.d.). The safety, pharmacokinetic and pharmacodynamic data support the further development of MHV370 in systemic autoimmune diseases driven by the overactivation of TLR7 and TLR8.

Sections du résumé

BACKGROUND AND OBJECTIVE OBJECTIVE
MHV370, a dual antagonist of human Toll-like receptors (TLR) 7 and 8, suppresses cytokines and interferon-stimulated genes in vitro and in vivo, and  has demonstrated efficacy in murine models of lupus. This first-in-human study aimed to evaluate the safety, tolerability, pharmacokinetics and pharmacodynamics of single and multiple doses of MHV370 in healthy adults, as well as the effects of food consumption on a single dose of MHV370.
METHODS METHODS
This was a phase 1, randomised, placebo-controlled study conducted in three parts. In part A, participants received (3:1) a single ascending dose (SAD) of 1, 3, 10, 20, 40, 80, 160, 320, 640 and 1000 mg MHV370 or placebo. In part B, participants received (3:1) multiple ascending doses (MAD) of 25, 50, 100, 200 and 400 mg MHV370 twice daily (b.i.d) or placebo for 14 days. In part C, participants received an open-label single dose of 200 mg MHV370 under fasted or fed conditions. Safety, pharmacokinetic and pharmacodynamic parameters were evaluated.
RESULTS RESULTS
MHV370 was well tolerated, and no safety signal was observed in the study. No dose-limiting adverse events occurred across the dose range evaluated. Plasma concentrations of MHV370 increased with dose (mean [SD] maximum plasma concentrations ranged from 0.97 [0.48] to 1670 [861.0] ng/mL for SAD of 3-1000 mg, 29.5 [7.98] to 759 [325.0] ng/mL for MAD of 25-400 mg b.i.d. on day 1). The intake of food did not have a relevant impact on the pharmacokinetics of MHV370. Pharmacodynamic data indicated time- and dose-dependent inhibition of TLR7-mediated CD69 expression on B cells (100% inhibition at 24 h post-dose starting from SAD 160 mg and MAD 50 mg b.i.d.) and TLR8-mediated TNF release after ex vivo stimulation (>90% inhibition at 24 h post-dose starting from SAD 320 mg and MAD 100 mg b.i.d.).
CONCLUSION CONCLUSIONS
The safety, pharmacokinetic and pharmacodynamic data support the further development of MHV370 in systemic autoimmune diseases driven by the overactivation of TLR7 and TLR8.

Identifiants

pubmed: 37532923
doi: 10.1007/s13318-023-00847-3
pii: 10.1007/s13318-023-00847-3
pmc: PMC10480294
doi:

Substances chimiques

Toll-Like Receptor 7 0
Toll-Like Receptor 8 0

Types de publication

Randomized Controlled Trial Clinical Trial, Phase I Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

553-566

Informations de copyright

© 2023. The Author(s).

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Auteurs

Tamas Shisha (T)

Novartis Pharma AG, Novartis Institutes for BioMedical Research, 4002, Basel, Switzerland. tamas.shisha@novartis.com.

Maximilian G Posch (MG)

Charité Research Organisation GmbH, Berlin, Germany.

Jeanette Lehmann (J)

Charité Research Organisation GmbH, Berlin, Germany.

Roland Feifel (R)

Novartis Pharma AG, Novartis Institutes for BioMedical Research, 4002, Basel, Switzerland.

Tobias Junt (T)

Novartis Pharma AG, Novartis Institutes for BioMedical Research, 4002, Basel, Switzerland.

Stuart Hawtin (S)

Novartis Pharma AG, Novartis Institutes for BioMedical Research, 4002, Basel, Switzerland.

Jens Schuemann (J)

Novartis Pharma AG, Novartis Institutes for BioMedical Research, 4002, Basel, Switzerland.

Alexandre Avrameas (A)

Novartis Pharma AG, Novartis Institutes for BioMedical Research, 4002, Basel, Switzerland.

Rambabu Danekula (R)

Novartis Pharma AG, Novartis Institutes for BioMedical Research, 4002, Basel, Switzerland.

Patrycja Misiolek (P)

Novartis Pharma AG, Novartis Institutes for BioMedical Research, 4002, Basel, Switzerland.

Richard Siegel (R)

Novartis Pharma AG, Novartis Institutes for BioMedical Research, 4002, Basel, Switzerland.

Peter Gergely (P)

Novartis Pharma AG, Novartis Institutes for BioMedical Research, 4002, Basel, Switzerland.

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Classifications MeSH