Circular RNA circ_0000119 promotes cervical cancer cell growth and migration via miR-433-3p/PAK2 axis.


Journal

Journal of applied genetics
ISSN: 2190-3883
Titre abrégé: J Appl Genet
Pays: England
ID NLM: 9514582

Informations de publication

Date de publication:
Sep 2023
Historique:
received: 02 02 2023
accepted: 19 07 2023
revised: 17 07 2023
medline: 28 8 2023
pubmed: 4 8 2023
entrez: 4 8 2023
Statut: ppublish

Résumé

The purpose of this study was to investigate the role of circ_0000119 on CC progression and its molecular mechanism. The expression levels of circ_0000119, miR-433-3p, and p21-activated kinase 2 (PAK2) in CC tissues and cell lines were measured by quantitative real-time polymerase chain reaction (qRT-PCR). Cell proliferation was assessed using 3-(4,5)-dimethylthiahiazo (-z-y1)-3,5-di-phenytetrazoliumromide (MTT) assay, 5-Ethynyl-2'-deoxyuridine (EdU) assay and colony formation assay. Cell cycle and apoptosis were assessed by flow cytometry. Cell migration and invasive ability were examined by Transwell assays. Downstream binding targets of circ_0000119 were predicted by online bioinformatics tools and confirmed by dual luciferase reporter gene assay, RNA immunoprecipitation (RIP) assay, and RNA pull-down assay. The role of circ_0000119/miR-433-3p/PAK2 axis in regulating the CC process was explored by rescue experiments. A xenograft model was constructed to further determine the effect of circ_0000119 on CC tumor growth in vivo. Immunohistochemistry (IHC) assay was conducted for Ki67 expression. Circ_0000119 was aberrantly upregulated in CC tissues and cell lines. Knockdown of circ_0000119 inhibited CC cell proliferation, cell cycle progress, migration, invasion, and promoted apoptosis of CC cells. MiR-433-3p was a binding target of circ_0000119, and PAK2 was a downstream gene of miR-433-3p. MiR-433-3p inhibition reversed the inhibitory effect of silencing circ_0000119 on CC progression. In addition, PAK2 overexpression reversed the effect of miR-433-3p on CC progression. PAK2 expression was regulated by circ_0000119 and miR-433-3p. Moreover, circ_0000119 knockdown reduced tumor growth of CC in vivo. Circ_0000119 was upregulated in CC, and circ_0000119 knockdown suppressed CC malignant development through the miR-433-3p/PAK2 axis.

Identifiants

pubmed: 37540462
doi: 10.1007/s13353-023-00772-w
pii: 10.1007/s13353-023-00772-w
doi:

Substances chimiques

RNA, Circular 0
p21-Activated Kinases EC 2.7.11.1
MicroRNAs 0
PAK2 protein, human EC 2.7.11.1
MIRN433 microRNA, human 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

531-543

Informations de copyright

© 2023. The Author(s), under exclusive licence to Institute of Plant Genetics Polish Academy of Sciences.

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Auteurs

Junxiao Zhang (J)

Third Department of Gynecology, Northwest Women's and Children's Hospital, No. 1616, Yanxiang Road, Yanta District, Xi'an, 710061, People's Republic of China. fakcdyx@163.com.

Guanghua Chu (G)

Second Department of Gynecology, Northwest Women's and Children's Hospital, Xi'an , 710061, China.

Lihua Zheng (L)

Third Department of Gynecology, Northwest Women's and Children's Hospital, No. 1616, Yanxiang Road, Yanta District, Xi'an, 710061, People's Republic of China.

Juandi Liu (J)

Third Department of Gynecology, Northwest Women's and Children's Hospital, No. 1616, Yanxiang Road, Yanta District, Xi'an, 710061, People's Republic of China.

Juxian He (J)

Third Department of Gynecology, Northwest Women's and Children's Hospital, No. 1616, Yanxiang Road, Yanta District, Xi'an, 710061, People's Republic of China.

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Classifications MeSH