Combining clinical and molecular characterization of CDH1: a multidisciplinary approach to reclassification of a splicing variant.

CDH1 Hereditary diffuse gastric cancer Lobular breast cancer RNA sequencing

Journal

Familial cancer
ISSN: 1573-7292
Titre abrégé: Fam Cancer
Pays: Netherlands
ID NLM: 100898211

Informations de publication

Date de publication:
Oct 2023
Historique:
received: 02 05 2023
accepted: 25 07 2023
pubmed: 4 8 2023
medline: 4 8 2023
entrez: 4 8 2023
Statut: ppublish

Résumé

Pathogenic germline variants (PGVs) in the CDH1 gene are associated with diffuse gastric and lobular breast cancer syndrome (DGLBC) and can increase the lifetime risk for both diffuse gastric cancer and lobular breast cancer. Given the risk for diffuse gastric cancer among individuals with CDH1 PGVs is up to 30-40%, prophylactic total gastrectomy is often recommended to affected individuals. Therefore, accurate interpretation of CDH1 variants is of the utmost importance for proper clinical decision-making. Herein we present a 45-year-old female, with lobular breast cancer and a father with gastric cancer of unknown pathology at age 48, who was identified to have an intronic variant of uncertain significance in the CDH1 gene, specifically c.833-9 C > G. Although the proband did not meet the International Gastric Cancer Linkage Consortium (IGCLC) criteria for gastric surveillance, she elected to pursue an upper endoscopy where non-targeted gastric biopsies identified a focus of signet ring cell carcinoma (SRCC). The proband then underwent a total gastrectomy, revealing numerous SRCC foci, but no invasive diffuse gastric cancer. Simultaneously, a genetic testing laboratory performed RNA sequencing to further analyze the CDH1 intronic variant, identifying an abnormal transcript from a novel acceptor splice site. The RNA analysis in conjunction with the patient's gastric foci of SRCC and family history was sufficient evidence for reclassification of the variant from uncertain significance to likely pathogenic. In conclusion, we report the first case of the CDH1 c.833-9 C > G intronic variant being associated with DGLBC and illustrate how collaboration among clinicians, laboratory personnel, and patients is crucial for variant resolution.

Identifiants

pubmed: 37540482
doi: 10.1007/s10689-023-00346-z
pii: 10.1007/s10689-023-00346-z
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

521-526

Subventions

Organisme : DeGregorio Family Foundation
ID : N/A

Informations de copyright

© 2023. The Author(s), under exclusive licence to Springer Nature B.V.

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Auteurs

Corrine Fillman (C)

Cancer Risk and Genetics Program, St. Luke's University Health Network, Bethlehem, PA, USA.

Arravinth Anantharajah (A)

University of Pennsylvania Perelman School of Medicine, Philadelphia, PA, USA.

Briana Marmelstein (B)

Cancer Risk and Genetics Program, St. Luke's University Health Network, Bethlehem, PA, USA.

Monica Dillon (M)

Cancer Risk and Genetics Program, St. Luke's University Health Network, Bethlehem, PA, USA.

Carolyn Horton (C)

Ambry Genetics, Aliso Viejo, CA, USA.

Candace Peterson (C)

Ambry Genetics, Aliso Viejo, CA, USA.

Joseph Lopez (J)

Cancer Risk and Genetics Program, St. Luke's University Health Network, Bethlehem, PA, USA.

Rashmi Tondon (R)

University of Pennsylvania Perelman School of Medicine, Philadelphia, PA, USA.

Terra Brannan (T)

Ambry Genetics, Aliso Viejo, CA, USA.

Bryson W Katona (BW)

University of Pennsylvania Perelman School of Medicine, Philadelphia, PA, USA. bryson.katona@pennmedicine.upenn.edu.
Division of Gastroenterology and Hepatology, University of Pennsylvania Perelman School of Medicine, 3400 Civic Center Blvd. 751 South Pavilion, Philadelphia, PA, 19104, USA. bryson.katona@pennmedicine.upenn.edu.

Classifications MeSH