Utilization of donor-derived Cell-Free DNA in pediatric kidney transplant recipients: A single center study.

acute rejection anti-HLA antibody antibody mediated rejection pediatric kidney transplantation

Journal

Pediatric transplantation
ISSN: 1399-3046
Titre abrégé: Pediatr Transplant
Pays: Denmark
ID NLM: 9802574

Informations de publication

Date de publication:
07 Aug 2023
Historique:
revised: 02 06 2023
received: 07 04 2023
accepted: 07 07 2023
medline: 8 8 2023
pubmed: 8 8 2023
entrez: 7 8 2023
Statut: aheadofprint

Résumé

High donor-derived cell-free DNA (dd-cfDNA) levels indicate transplant allograft injury and can identify graft rejection in kidney transplant recipients. Here, we evaluated the use of dd-cfDNA in pediatric kidney transplant rejection monitoring and treatment. Forty-two pediatric kidney transplant patients were enrolled between February 2020 and August 2021. Dd-cfDNA was tested before and after biopsy/rejection treatment. There was a total of 61 allograft biopsies (44 for-cause, 17 surveillance). Graft rejection was found in 35/61 biopsies. Rejection was more common in basiliximab induction compared to rATG (77.1% vs. 22.9%, p = .0121). Median dd-cfDNA was higher in those with rejection (1.2% [0.34-3.12] vs. 0.24% [0.08-0.78], p < .0001). Dd-cfDNA was highest in biopsies with AMR and mixed AMR/TCMR. In addition, dd-cfDNA in basiliximab induction was higher compared to rATG (0.92% [0.27-1.8] vs. 0.26% [0.08-2], p = .0437). Median change in dd-cfDNA after rejection treatment was -0.57% (-1.67 to 0.05). Median time to dd-cfDNA <1% post-rejection treatment was 8.5 days (3.0-19.5). Dd-cfDNA in AMR was higher compared to TCMR or mixed rejection, and levels remained higher in AMR after treatment. In surveillance biopsies, 4/17 had rejection. Median dd-cfDNA was not different in those with versus without rejection (0.48% vs. 0.28%, p = .2342). Those without rejection all had dd-cfDNA <1%. In those with rejection, only one patient had dd-cfDNA >1%, and all had TCMR. Our findings support dd-cfDNA as a useful indicator of graft rejection and response to treatment. Additional studies are needed to determine the role of dd-cfDNA in graft health surveillance.

Identifiants

pubmed: 37550268
doi: 10.1111/petr.14582
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

e14582

Informations de copyright

© 2023 Wiley Periodicals LLC.

Références

Clayton PA, McDonald SP, Russ GR, Chadban SJ. Long-term outcomes after acute rejection in kidney transplant recipients: an ANZDATA analysis. J Am Soc Nephrol. 2019;30(9):1697-1707. doi:10.1681/ASN.2018111101
Redfield RR, Scalea JR, Zens TJ, et al. The mode of sensitization and its influence on allograft outcomes in highly sensitized kidney transplant recipients. Nephrol Dial Transplant. 2016;31(10):1746-1753. doi:10.1093/ndt/gfw099
Josephson MA. Monitoring and managing graft health in the kidney transplant recipient. Clin J Am Soc Nephrol. 2011;6(7):1774-1780. doi:10.2215/CJN.01230211
Kasiske BL, Zeier MG, Chapman JR, et al. KDIGO clinical practice guideline for the care of kidney transplant recipients: a summary. Kidney Int. 2010;77(4):299-311. doi:10.1038/ki.2009.377
Filippone EJ, Farber JL. The monitoring of donor-derived cell-free DNA in kidney transplantation. Transplantation. 2021;105(3):509-516. doi:10.1097/TP.0000000000003393
Puliyanda DP, Swinford R, Pizzo H, Garrison J, De Golovine AM, Jordan SC. Donor-derived cell-free DNA (dd-cfDNA) for detection of allograft rejection in pediatric kidney transplants. Pediatr Transplant. 2021;25(2):e13850. doi:10.1111/petr.13850
Bloom RD, Bromberg JS, Poggio ED, et al. Cell-free DNA and active rejection in kidney allografts. J Am Soc Nephrol. 2017;28(7):2221-2232. doi:10.1681/ASN.2016091034
Stites E, Kumar D, Olaitan O, et al. High levels of dd-cfDNA identify patients with TCMR 1A and borderline allograft rejection at elevated risk of graft injury. Am J Transplant. 2020;20(9):2491-2498. doi:10.1111/ajt.15822
Lamarche C, Côté JM, Sénécal L, Cardinal H. Efficacy of acute cellular rejection treatment according to Banff score in kidney transplant recipients: a systematic review. Transplant Direct. 2016;2(12):e115. doi:10.1097/TXD.0000000000000626
Bouatou Y, Viglietti D, Pievani D, et al. Response to treatment and long-term outcomes in kidney transplant recipients with acute T cell-mediated rejection. Am J Transplant. 2019;19(7):1972-1988. doi:10.1111/ajt.15299
Hinojosa RJ, Chaffin K, Gillespie M, Villarreal VH. Donor-derived cell-free DNA may confirm real-time response to treatment of acute rejection in renal transplant recipients. Transplantation. 2019;103(4):e61. doi:10.1097/TP.0000000000002579
Steggerda JA, Pizzo H, Garrison J, et al. Use of a donor-derived cell-free DNA assay to monitor treatment response in pediatric renal transplant recipients with allograft rejection. Pediatr Transplant. 2022;26(4):e14258. doi:10.1111/petr.14258
Haas M, Loupy A, Lefaucheur C, et al. The Banff 2017 kidney meeting report: revised diagnostic criteria for chronic active T cell-mediated rejection, antibody-mediated rejection, and prospects for integrative endpoints for next-generation clinical trials. Am J Transplant. 2018;18(2):293-307. doi:10.1111/ajt.14625
Dandamudi R, Gu H, Goss CW, Walther L, Dharnidharka VR. Longitudinal evaluation of donor-derived Cellfree DNA in pediatric kidney transplantation. Clin J Am Soc Nephrol. 2022;17(11):1646-1655. doi:10.2215/CJN.03840322
Bu L, Gupta G, Pai A, et al. Clinical outcomes from the assessing donor-derived cell-free DNA monitoring insights of kidney allografts with longitudinal surveillance (ADMIRAL) study. Kidney Int. 2022;101(4):793-803. doi:10.1016/j.kint.2021.11.034
Chang J, Alvarado Verduzco H, Toma K, Sritharan S, Mohan S, Husain SA. Donor-derived cell-free DNA and renal allograft rejection in surveillance biopsies and indication biopsies. Clin Transpl. 2022;36(4):e14561. doi:10.1111/ctr.14561
Gupta G, Moinuddin I, Kamal L, et al. Correlation of donor-derived cell-free DNA with histology and molecular diagnoses of kidney transplant biopsies. Transplantation. 2022;106(5):1061-1070. doi:10.1097/TP.0000000000003838
Kataria A, Kumar D, Gupta G. Donor-derived cell-free DNA in solid-organ transplant diagnostics: indications, limitations, and future directions. Transplantation. 2021;105(6):1203-1211. doi:10.1097/TP.0000000000003651
Oellerich M, Budde K, Osmanodja B, et al. Donor-derived cell-free DNA for personalized immunosuppression in renal transplantation. Ther Drug Monit. 2023;45(1):20-25. doi:10.1097/FTD.0000000000001023
Oellerich M, Sherwood K, Keown P, et al. Liquid biopsies: donor-derived cell-free DNA for the detection of kidney allograft injury. Nat Rev Nephrol. 2021;17(9):591-603. doi:10.1038/s41581-021-00428-0
Wijtvliet VPWM, Plaeke P, Abrams S, et al. Donor-derived cell-free DNA as a biomarker for rejection after kidney transplantation: a systematic review and meta-analysis. Transpl Int. 2020;33(12):1626-1642. doi:10.1111/tri.13753
Anand S, Lopez-Verdugo F, Sanchez-Garcia J, et al. Longitudinal variance of donor-derived cell-free DNA (dd-cfDNA) in stable kidney transplant (KTx) patients are influenced by donor/recipient variables. Clin Transpl. 2021;35(9):e14395. doi:10.1111/ctr.14395
Mehta SG, Chang JH, Alhamad T, et al. Repeat kidney transplant recipients with active rejection have elevated donor-derived cell-free DNA. Am J Transplant. 2019;19(5):1597-1598. doi:10.1111/ajt.15192
Sureshkumar KK, Lyons S, Chopra B. Impact of kidney transplant type and previous transplant on baseline donor-derived cell free DNA. Transpl Int. 2020;33(10):1324-1325. doi:10.1111/tri.13673
Wolf-Doty TK, Mannon RB, Poggio ED, et al. Dynamic response of donor-derived cell-free DNA following treatment of acute rejection in kidney allografts. Kidney360. 2021;2(4):729-736. doi:10.34067/KID.0000042021
Brennan DC, Daller JA, Lake KD, Cibrik D, Del Castillo D; Thymoglobulin Induction Study Group. Rabbit antithymocyte globulin versus basiliximab in renal transplantation. N Engl J Med. 2006;355(19):1967-1977. PMID: 17093248. doi:10.1056/NEJMoa060068

Auteurs

Daniel Ranch (D)

Department of Pediatrics, UT Health San Antonio, San Antonio, Texas, USA.

Mingwei Fei (M)

Biostatistics Department, University of North Carolina at Chapel Hill, Chapel Hill, North Carolina, USA.

Elisabeth Kincade (E)

University Health Transplant Institute, San Antonio, Texas, USA.
Pharmacotherapy Division, College of Pharmacy, The University of Texas at Austin, Austin, Texas, USA.

Kim Piburn (K)

Department of Pediatrics, UT Health San Antonio, San Antonio, Texas, USA.

Kelley Hitchman (K)

Department of Pathology and Laboratory Medicine, UT Health San Antonio, San Antonio, Texas, USA.

Kelsey Klein (K)

University Health Transplant Institute, San Antonio, Texas, USA.
Pharmacotherapy Division, College of Pharmacy, The University of Texas at Austin, Austin, Texas, USA.

Classifications MeSH