Ocular Vascular Diseases: From Retinal Immune Privilege to Inflammation.
age-related macular degeneration
diabetic retinopathy
immune privilege
inflammation
retina
retinopathy of prematurity
Journal
International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791
Informations de publication
Date de publication:
28 Jul 2023
28 Jul 2023
Historique:
received:
22
06
2023
revised:
21
07
2023
accepted:
26
07
2023
medline:
16
8
2023
pubmed:
12
8
2023
entrez:
12
8
2023
Statut:
epublish
Résumé
The eye is an immune privileged tissue that insulates the visual system from local and systemic immune provocation to preserve homeostatic functions of highly specialized retinal neural cells. If immune privilege is breached, immune stimuli will invade the eye and subsequently trigger acute inflammatory responses. Local resident microglia become active and release numerous immunological factors to protect the integrity of retinal neural cells. Although acute inflammatory responses are necessary to control and eradicate insults to the eye, chronic inflammation can cause retinal tissue damage and cell dysfunction, leading to ocular disease and vision loss. In this review, we summarized features of immune privilege in the retina and the key inflammatory responses, factors, and intracellular pathways activated when retinal immune privilege fails, as well as a highlight of the recent clinical and research advances in ocular immunity and ocular vascular diseases including retinopathy of prematurity, age-related macular degeneration, and diabetic retinopathy.
Identifiants
pubmed: 37569464
pii: ijms241512090
doi: 10.3390/ijms241512090
pmc: PMC10418793
pii:
doi:
Types de publication
Journal Article
Review
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : NEI NIH HHS
ID : R01 EY029238
Pays : United States
Organisme : NEI NIH HHS
ID : R01 EY030140
Pays : United States
Organisme : NIH HHS
ID : R01EY030140, R01EY029238
Pays : United States
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