Endometrial/Endometrioid Stromal Tumors With Extensive Whorling and CTNNB1 Translocation : A Report of 3 Cases.


Journal

The American journal of surgical pathology
ISSN: 1532-0979
Titre abrégé: Am J Surg Pathol
Pays: United States
ID NLM: 7707904

Informations de publication

Date de publication:
01 11 2023
Historique:
medline: 6 11 2023
pubmed: 16 8 2023
entrez: 16 8 2023
Statut: ppublish

Résumé

Endometrial/endometrioid stromal tumors are rare and morphologically heterogenous, and their diagnosis may be challenging. We identified 3 endometrial/endometrioid stromal tumors with identical and previously undescribed histologic features and herein report their morphologic, immunohistochemical, and molecular profiles. Patients were 53, 62, and 79 years. Tumors were well-circumscribed, tan-yellow solid masses measuring 10.0, 11.0, and 18.7 cm, and were intramyometrial (n=2) or in the broad ligament (n=1). All showed small, tight whorls of epithelioid to slightly spindled tumor cells with minimal cytoplasm and negligible mitoses, multifocally associated with hyalinization and myxoid change set in a loose fibroblastic background with small, delicate vessels. This morphology was seen throughout in 1 tumor and in ∼20% and 70% of the 2 others with the remaining areas showing sex cord-like differentiation. Tumor cells expressed CD10 (3/3, 1 focal), calretinin (3/3 diffuse), WT1 (3/3 diffuse), estrogen receptor (1/1, diffuse). RNA-sequencing was successful in 1 tumor and revealed a GREB1-CTNNB1 in-frame fusion. All 3 tumors harbored a CTNNB1 translocation by fluorescence in situ hybridization correlating with nuclear β-catenin expression. Whole-genome DNA methylation analysis classified all 3 tumors within the low-grade endometrial stromal sarcoma reference class with flat copy number profiles. One patient (79-y-old) died of unrelated causes 2 months after surgery and the other 2 were alive without disease after 13 and 75 months. We have described a rare subset of endometrial/endometrioid stromal tumors with extensive whorling and a CTNNB1 translocation, expanding the morphologic and molecular spectrum of these neoplasms.

Identifiants

pubmed: 37584555
doi: 10.1097/PAS.0000000000002094
pii: 00000478-990000000-00218
doi:

Substances chimiques

beta Catenin 0
Biomarkers, Tumor 0
CTNNB1 protein, human 0

Types de publication

Case Reports Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1285-1290

Subventions

Organisme : NCI NIH HHS
ID : P30 CA008748
Pays : United States

Informations de copyright

Copyright © 2023 Wolters Kluwer Health, Inc. All rights reserved.

Déclaration de conflit d'intérêts

Conflicts of Interest and Source of Funding: B.W. reports research funding from Repare Therapeutics, outside the scope of this study. For the remaining authors none were declared.

Références

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Auteurs

Baris Boyraz (B)

James Homer Wright Pathology Laboratories, Department of Pathology, Massachusetts General Hospital, Harvard Medical School, Boston, MA.

Arnaud da Cruz Paula (A)

Departments of Surgery.

Kelly A Deveraux (KA)

Department of Pathology, New York University, Langone Medical Center, New York, NY.

Ivy Tran (I)

Department of Pathology, New York University, Langone Medical Center, New York, NY.

Edaise M da Silva (EM)

Pathology and Laboratory Medicine, Memorial Sloan Kettering Cancer Center.

Robert H Young (RH)

James Homer Wright Pathology Laboratories, Department of Pathology, Massachusetts General Hospital, Harvard Medical School, Boston, MA.

Matija Snuderl (M)

Department of Pathology, New York University, Langone Medical Center, New York, NY.

Britta Weigelt (B)

Pathology and Laboratory Medicine, Memorial Sloan Kettering Cancer Center.

Esther Oliva (E)

James Homer Wright Pathology Laboratories, Department of Pathology, Massachusetts General Hospital, Harvard Medical School, Boston, MA.

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