Prenatal exposure to selenium, mercury, and manganese during pregnancy and allergic diseases in early childhood: The Japan Environment and Children's study.


Journal

Environment international
ISSN: 1873-6750
Titre abrégé: Environ Int
Pays: Netherlands
ID NLM: 7807270

Informations de publication

Date de publication:
09 2023
Historique:
received: 21 03 2023
revised: 23 07 2023
accepted: 28 07 2023
medline: 18 9 2023
pubmed: 19 8 2023
entrez: 18 8 2023
Statut: ppublish

Résumé

Prenatal exposure to metallic elements may adversely affect early childhood health. However, more evidence is needed as population-based cohort studies are currently limited. We aimed to examine the associations between prenatal metallic (mercury, selenium, and manganese) exposure and the risk of allergic diseases in early childhood until three years of age. The data from 94,794 mother-infant pairs, who participated in the Japan Environment and Children's study, were used in this study. Prenatal metallic element exposure was measured in maternal blood collected during mid-pregnancy. The incidence of atopic dermatitis, food allergies, asthma, and allergic rhinitis during the first three years of life was prospectively investigated using self-reports of physician-diagnosed allergies. A multivariable modified Poisson regression model was used to estimate the cumulative incidence ratio and their 95% confidence intervals of allergic diseases associated with prenatal exposure to mercury, selenium, and manganese. We further evaluated the interaction between mercury and selenium exposures in this association. We confirmed 26,238 cases of childhood allergic diseases: atopic dermatitis, food allergies, asthma, and allergic rhinitis in 9,715 (10.3%), 10,897 (11.5%), and 9,857 (10.4%), 4,630 (4.9%), respectively. No association was found between prenatal mercury or manganese exposure and the risk of allergic diseases. Prenatal selenium exposure was inversely associated with atopic dermatitis, food allergies, allergic rhinitis, and any allergic diseases, but not with asthma. These inverse associations were more pronounced for lower mercury exposures than for higher exposures. Our findings suggest that prenatal exposure to selenium may be beneficial for reducing the risk of atopic dermatitis, food allergies, allergic rhinitis, and any allergic diseases in early childhood, especially with lower prenatal mercury exposure.

Sections du résumé

BACKGROUND
Prenatal exposure to metallic elements may adversely affect early childhood health. However, more evidence is needed as population-based cohort studies are currently limited.
OBJECTIVES
We aimed to examine the associations between prenatal metallic (mercury, selenium, and manganese) exposure and the risk of allergic diseases in early childhood until three years of age.
METHODS
The data from 94,794 mother-infant pairs, who participated in the Japan Environment and Children's study, were used in this study. Prenatal metallic element exposure was measured in maternal blood collected during mid-pregnancy. The incidence of atopic dermatitis, food allergies, asthma, and allergic rhinitis during the first three years of life was prospectively investigated using self-reports of physician-diagnosed allergies. A multivariable modified Poisson regression model was used to estimate the cumulative incidence ratio and their 95% confidence intervals of allergic diseases associated with prenatal exposure to mercury, selenium, and manganese. We further evaluated the interaction between mercury and selenium exposures in this association.
RESULTS
We confirmed 26,238 cases of childhood allergic diseases: atopic dermatitis, food allergies, asthma, and allergic rhinitis in 9,715 (10.3%), 10,897 (11.5%), and 9,857 (10.4%), 4,630 (4.9%), respectively. No association was found between prenatal mercury or manganese exposure and the risk of allergic diseases. Prenatal selenium exposure was inversely associated with atopic dermatitis, food allergies, allergic rhinitis, and any allergic diseases, but not with asthma. These inverse associations were more pronounced for lower mercury exposures than for higher exposures.
CONCLUSIONS
Our findings suggest that prenatal exposure to selenium may be beneficial for reducing the risk of atopic dermatitis, food allergies, allergic rhinitis, and any allergic diseases in early childhood, especially with lower prenatal mercury exposure.

Identifiants

pubmed: 37595534
pii: S0160-4120(23)00396-3
doi: 10.1016/j.envint.2023.108123
pii:
doi:

Substances chimiques

Selenium H6241UJ22B
Manganese 42Z2K6ZL8P
Vitamins 0
Mercury FXS1BY2PGL

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

108123

Informations de copyright

Copyright © 2023 The Authors. Published by Elsevier Ltd.. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper.

Auteurs

Junji Miyazaki (J)

Division of Environmental Medicine and Population Sciences, Department of Social Medicine, Osaka University Graduate School of Medicine, 2-2, Yamadaoka, Suita-shi, Osaka 565-0871, Japan; Osaka Regional Center for Japan Environment and Children's Study (JECS), Osaka University, 1-3, Yamadaoka, Suita-shi, Osaka 565-0871, Japan; Department of Preventive Medicine and Public Health, School of Medicine, Keio University, 35, Shinano-cho, Shinjuku-ku, Tokyo 160-8582, Japan.

Satoyo Ikehara (S)

Division of Environmental Medicine and Population Sciences, Department of Social Medicine, Osaka University Graduate School of Medicine, 2-2, Yamadaoka, Suita-shi, Osaka 565-0871, Japan; Osaka Regional Center for Japan Environment and Children's Study (JECS), Osaka University, 1-3, Yamadaoka, Suita-shi, Osaka 565-0871, Japan.

Kanami Tanigawa (K)

Division of Environmental Medicine and Population Sciences, Department of Social Medicine, Osaka University Graduate School of Medicine, 2-2, Yamadaoka, Suita-shi, Osaka 565-0871, Japan; Osaka Regional Center for Japan Environment and Children's Study (JECS), Osaka University, 1-3, Yamadaoka, Suita-shi, Osaka 565-0871, Japan; Osaka Maternal and Child Health Information Center, Osaka Women's and Children's Hospital, 840, Murodo-cho, Izumi-shi, Osaka 594-1101, Japan.

Takashi Kimura (T)

Department of Public Health, Faculty of Medicine, Hokkaido University, North 15, West 7, Kita-ku, Sapporo-shi, Hokkaido 060-8638, Japan.

Kimiko Ueda (K)

Division of Environmental Medicine and Population Sciences, Department of Social Medicine, Osaka University Graduate School of Medicine, 2-2, Yamadaoka, Suita-shi, Osaka 565-0871, Japan; Osaka Regional Center for Japan Environment and Children's Study (JECS), Osaka University, 1-3, Yamadaoka, Suita-shi, Osaka 565-0871, Japan; Faculty of Health and Well-being, Kansai University, 1-11-1 Kaorigaoka-cho, Sakai-ku, Sakai, Osaka, 590-8515, Japan.

Keiichi Ozono (K)

Department of Pediatrics, Osaka University Graduate School of Medicine, 2-2, Yamadaoka, Suita-shi, Osaka 565-0871, Japan.

Tadashi Kimura (T)

Department of Obstetrics and Gynecology, Osaka University Graduate School of Medicine, 2-2, Yamadaoka, Suita-shi, Osaka 565-0871, Japan.

Yayoi Kobayashi (Y)

Japan Environment and Children's Study Programme Office, National Institute for Environmental Studies, 16-2, Onogawa, Tsukuba-shi, Ibaraki 305-8506, Japan.

Shin Yamazaki (S)

Japan Environment and Children's Study Programme Office, National Institute for Environmental Studies, 16-2, Onogawa, Tsukuba-shi, Ibaraki 305-8506, Japan.

Michihiro Kamijima (M)

Department of Occupational and Environmental Health, Graduate School of Medical Sciences, Nagoya City University, 1, Kawasumi, Mizuho-Cho, Mizuho-ku, Nagoya 467-8601, Japan.

Tomotaka Sobue (T)

Division of Environmental Medicine and Population Sciences, Department of Social Medicine, Osaka University Graduate School of Medicine, 2-2, Yamadaoka, Suita-shi, Osaka 565-0871, Japan; Osaka Regional Center for Japan Environment and Children's Study (JECS), Osaka University, 1-3, Yamadaoka, Suita-shi, Osaka 565-0871, Japan.

Hiroyasu Iso (H)

Osaka Regional Center for Japan Environment and Children's Study (JECS), Osaka University, 1-3, Yamadaoka, Suita-shi, Osaka 565-0871, Japan; Institute for Global Health Policy Research, Bureau of International Health Cooperation, National Center for Global Health and Medicine, 1-21-1, Toyama, Shinjuku-ku, Tokyo 162-8655, Japan. Electronic address: iso@pbhel.med.osaka-u.ac.jp.

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