Modulating Treg stability to improve cancer immunotherapy.

cancer immunotherapy immunotolerance lineage instability regulatory T cells tumor microenvironment

Journal

Trends in cancer
ISSN: 2405-8025
Titre abrégé: Trends Cancer
Pays: United States
ID NLM: 101665956

Informations de publication

Date de publication:
11 2023
Historique:
received: 03 05 2023
revised: 23 07 2023
accepted: 25 07 2023
medline: 23 10 2023
pubmed: 20 8 2023
entrez: 19 8 2023
Statut: ppublish

Résumé

Immunosuppressive regulatory T cells (Tregs) provide a main mechanism of tumor immune evasion. Targeting Tregs, especially in the tumor microenvironment (TME), continues to be investigated to improve cancer immunotherapy. Recent studies have unveiled intratumoral Treg heterogeneity and plasticity, furthering the complexity of the role of Tregs in tumor immunity and immunotherapy response. The phenotypic and functional diversity of intratumoral Tregs can impact their response to therapy and may offer new targets to modulate specific Treg subsets. In this review we provide a unifying framework of critical factors contributing to Treg heterogeneity and plasticity in the TME, and we discuss how this information can guide the development of more specific Treg-targeting therapies for cancer immunotherapy.

Identifiants

pubmed: 37598003
pii: S2405-8033(23)00142-5
doi: 10.1016/j.trecan.2023.07.015
pii:
doi:

Types de publication

Journal Article Review Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

911-927

Informations de copyright

Copyright © 2023 Elsevier Inc. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of interests R.Z. is inventor on patent applications related to work on GITR, PD-1, and CTLA-4. R.Z. is scientific advisory board member of iTEOS Therapeutics, has consulted for Leap Therapeutics, and receives grant support from AstraZeneca and Bristol Myers Squibb.

Auteurs

Jee Hye Kang (JH)

Weill Cornell Medicine, Weill Cornell Medical College of Cornell University, New York, NY, USA; Immunology and Microbial Pathogenesis Program, Weill Cornell Graduate School, New York, NY, USA.

Roberta Zappasodi (R)

Weill Cornell Medicine, Weill Cornell Medical College of Cornell University, New York, NY, USA; Immunology and Microbial Pathogenesis Program, Weill Cornell Graduate School, New York, NY, USA. Electronic address: roz4002@med.cornell.edu.

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Classifications MeSH