A case of carcinoid syndrome probably exacerbated by hemodialysis in which prochlorperazine maleate was effective.

Carcinoid syndrome Hemodialysis Portal systemic encephalopathy

Journal

CEN case reports
ISSN: 2192-4449
Titre abrégé: CEN Case Rep
Pays: Japan
ID NLM: 101636244

Informations de publication

Date de publication:
22 Aug 2023
Historique:
received: 21 10 2022
accepted: 31 07 2023
medline: 22 8 2023
pubmed: 22 8 2023
entrez: 22 8 2023
Statut: aheadofprint

Résumé

Carcinoid syndrome is caused by the release of serotonin and other substances, which commonly occurs due to liver metastasis of neuroendocrine tumors. It rarely occurs due to liver metastasis of neuroendocrine carcinoma. We report the case of a patient with liver metastasis of neuroendocrine carcinoma who suffered from acute abdominal pain and diarrhea triggered by hemodialysis. Various differential diagnoses were considered, but we concluded these symptoms to be probably caused by exacerbation of carcinoid syndrome, as the serum 5HIAA level was markedly elevated, and a drug with anti-serotonin activity was effective. Prochlorperazine maleate, which has anti-serotonin activity, was effective for these symptoms, and the patient was able to continue maintenance hemodialysis, which contributed to his quality of life and prognosis. We speculated the mechanism of carcinoid exacerbation was that substances such as serotonin had entered the systemic circulation via the increased extrahepatic shunt of the portal venous blood flow, entering the inferior vena cava and that this condition had been triggered by hemodialysis via the same mechanism as portal systemic encephalopathy.

Identifiants

pubmed: 37606883
doi: 10.1007/s13730-023-00814-6
pii: 10.1007/s13730-023-00814-6
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

© 2023. The Author(s), under exclusive licence to Japanese Society of Nephrology.

Références

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Auteurs

Keiko Oda (K)

Department of Cardiology and Nephrology, Mie University Graduate School of Medicine, 2-174 Edobashi, Tsu, Mie, 514-8507, Japan.

Tomohiro Murata (T)

Department of Cardiology and Nephrology, Mie University Graduate School of Medicine, 2-174 Edobashi, Tsu, Mie, 514-8507, Japan. tmhr0421@clin.medic.mie-u.ac.jp.

Kayo Tsujimoto (K)

Department of Cardiology and Nephrology, Mie University Graduate School of Medicine, 2-174 Edobashi, Tsu, Mie, 514-8507, Japan.

Fumika Tanaka (F)

Department of Cardiology and Nephrology, Mie University Graduate School of Medicine, 2-174 Edobashi, Tsu, Mie, 514-8507, Japan.

Daisuke Takahashi (D)

Department of Cardiology and Nephrology, Mie University Graduate School of Medicine, 2-174 Edobashi, Tsu, Mie, 514-8507, Japan.

Ryosuke Saiki (R)

Department of Cardiology and Nephrology, Mie University Graduate School of Medicine, 2-174 Edobashi, Tsu, Mie, 514-8507, Japan.

Yosuke Hirabayashi (Y)

Department of Cardiology and Nephrology, Mie University Graduate School of Medicine, 2-174 Edobashi, Tsu, Mie, 514-8507, Japan.

Akira Tsunoda (A)

Department of Hematology and Oncology, Mie University Graduate School of Medicine, Tsu, Japan.

Kanako Saito (K)

Department of Hematology and Oncology, Mie University Graduate School of Medicine, Tsu, Japan.

Hiroto Yuasa (H)

Department of Pathology, Mie University Graduate School of Medicine, Tsu, Japan.

Hiroshi Imai (H)

Department of Pathology, Mie University Graduate School of Medicine, Tsu, Japan.

Kan Katayama (K)

Department of Cardiology and Nephrology, Mie University Graduate School of Medicine, 2-174 Edobashi, Tsu, Mie, 514-8507, Japan.

Kaoru Dohi (K)

Department of Cardiology and Nephrology, Mie University Graduate School of Medicine, 2-174 Edobashi, Tsu, Mie, 514-8507, Japan.

Classifications MeSH